XSD200中文资料
SD215中文资料
High-Speed Analo N-Channel DMOS FETs SD211 / SD213 / SD215FEATURES•High Input to Output Isolation. . . . . . . . . . . . . . . . 120dB •Low On Resistance. . . . . . . . . . . . . . . . . . . . . . . . 30 Ohm •Low Feedthrough and Feedback Transients•Low Capacitance:–Input (Gate). . . . . . . . . . . . . . . . . . . . . . . . . . . 2.4pF typ.–Output. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 1.3pF typ.–Feedback. . . . . . . . . . . . . . . . . . . . . . . . . . . . 0.3pF typ.•Built-in Protection Diode from Gate to SubstrateAPPLICATIONSSD211:•Analog Switch DriverSD213 and SD215:•Analog Switches•High-Speed Digital Switches•Multiplexers• A to D Converters• D to A Converters•Choppers•Sample & Hold DESCRIPTIONThe Calogic SD211 is a 30V analog switch driver with built-in protection diode from gate to substrate The SD211 is used with SD213 and SD215 DMOS analog switches. ORDERING INFORMATIONPart Package Temperature Range SD211E Hermetic TO-72 Package-55C to +125C XSS211Sorted Chips in Carriers-55o C to +125o C SD213DE Hermetic TO-72 Package-55o C to +125o C XSD213Sorted Chips in Carriers-55o C to +125o C SD215DE Hermetic TO-72 Package-55o C to +125o C XSD215Sorted Chips in Carriers-55o C to +125oCABSOLUTE MAXIMUM RATINGSDrain Current. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 50mA Total Device Dissipation at 25o C Case Temperature . . . 1.2W Storage T emperatue Range . . . . . . . . . . . . . -65o C to +200o C Lead Temperature (1/16" from case for 10 sec.). . . . . . 300o C Operating T emperature Range . . . . . . . . . . . -55o C to +125o CDC CHARACTERISTICS (T A = 25oC, unless otherwise specified)AC ELECTRICAL CHARACTERISTICSInformation furnished by Calogic is believed to be accurate and reliable. However, no responsibility is assumed for its use: nor for any infringement of patents or other rights of third parties which may result from its use. No license is granted by implication or otherwise under any patent rights of Calogic.PARAMETER SD211SD212SD215UNIT V DS Drain-to-Source +30+10+20V dc V SD Source-to-Drain +10+10+20V dc V DB Drain-to-Body +30+15+25V dc V SB Source-to-Body +15+15+25V dc V GS Gate-to-Source -15+25-15+25-25+30V dc V GB Gate-to-Body -0.3+25-0.3+25-0.3+30V dc V GDGate-to-Drain-30+25-15+25-25+30V dc。
中文DEO200概说书_CH
DEO200-E -006Industrial - DEO工程师入门Copyright, Notices and TrademarksPrinted in Japan - © Copyright 2003 by Yamatake CorporationWhile this information is presented in good faith and believed to be accurate, Yamatake Corporation disclaims the implied warranties of merchantability and fitness for a particular purpose and makes no express warranties except as may be stated in its written agreement with and for its customer.In no event is Yamatake Industrial Systems Corporation liable to anyone for any indirect, special or consequential damages. The information and specifications in this document are subject tochange without notice.目录1 概述 (1)1.1 创建JOB 文件 (2)1.2 制作图形画面 (3)1.3 设置历史功能 (4)1.4 设置区域操作环境 (5)1.5 设置显示工具条 (6)1.6 设置顺序出错状态显示功能 (7)1.7 制作报表 (8)1.8 设置自动登陆 (8)1.9 设置自动启动 (8)2 设置文件 (9)2.1 区域设置文件 (9)2.1.1 组画面设置文件(Group.csv) (11)2.1.2 图形画面设置文件（Graphic.csv） (12)2.1.3 远程趋势画面设置文件（Trend.csv） (13)2.1.5 远程趋势服务器设置文件(TrendSvr.csv) (16)2.1.6 单元分配设置文件(Uassign.csv) (17)2.1.7 相关画面设置文件(Assoc.csv) (18)2.1.8 在线手册设置文件(Manual.csv) (18)2.2 按钮设置文件(C:\Ucspkg\mmi\Button.txt) (20)2.3 顺序出错状态显示设置文件GlobalJamState.txt） (23)2.4 初始设置文件（.ini） (24)2.4.1 Tuning.ini 文件 (25)2.4.2Journal.ini 文件 (26)2.4.3 Hardcopy.ini 文件 (29)2.4.4 Udpman.ini 文件 (32)2.4.5 Hdse.ini / Hdse_w.ini文件 (33)2.5 自动登录设置工具(Autolog.exe) (57)练习: 启用自动登录 (58)练习: 取消自动登录 (58)2.6 自动启动 (59)练习: 启用自动启动 (59)3 设置另一个节点 (61)1 概述本章梗概介绍了工程环境，在操作本系统时，需要完成若干工作环境的设置，设置如下：■创建JOB文件(适用于所有DEO工作站)■完成DEO-SE 程序的初始文件设置. (由DEO工作站完成)■制作图形画面. (仅在DOSS内完成)■组态历史功能(仅在DOHS/DOSS_H/DOSS_OH内完成)■通过配置文件设置区域操作环境. (仅在DOSS内完成)■配置显示工具条. (仅在DOSS内完成)■设置一个顺序阻塞状态显示功能的设置文件. (仅在DOSS内完成)■制作报表. (仅在DOSS or 客户机内完成)■如有需要，设置自动登陆功能.. (仅在. DOSS内完成)■如有需要，设置本地趋势. (仅在DOSS内完成)■其他1.1 创建JOB 文件所有DEO工作站需要通过用户的Job文件来了解你的DEO系统的结构和点. 用户Job文件可由RTC编辑器创建 .同样，DEO-SE的操作环境也可以由其初始设置文件来设置，如：hdse.ini and hdse_w.ini 文件用于工作站的用户Job文件的文件路径为：C:¥Jobdir¥user_job_name¥it¥xxx.csv用于box(控制器等)的用户Job文件的文件路径为：C:¥Jobdir¥user_job_name¥idb¥HDBXnnn.idbHDPTnnn.idbDEO-SE初始设置文件的路径：C:¥Winnt¥hdse.ini¥hdse_w.ini详细信息参考如下手册.-RTC用户指南(MS2-DEO200-2002)-本手册第二章l1.2 制作图形画面DEO系统利用“Intouch”制作和显示DEO画面，如组画面，趋势图，图形画面等.所有DEO的画面都放在“MMIPKG”目录中，并且通过“Intouch”调用.Mmipkg的目录路径：C:¥Sysdir¥Mmipkg详细信息参考如下手册-Intouch Window-Maker (DEO200-E-002)1.3 设置历史功能DEO 系统有一个历史站如DOHS, DOSS_H and DOSS_OH.DOSS提供历史客户应用软件 --“历史管理器”，以通过DOSS远程配置和管理历史功能。
xsditz常用日语200句(附中文发音)
-+懒惰是很奇怪的东西，它使你以为那是安逸，是休息，是福气；但实际上它所给你的是无聊，是倦怠，是消沉;它剥夺你对前途的希望，割断你和别人之间的友情，使你心胸日渐狭窄，对人生也越来越怀疑。
—罗兰第一部分01、はじめまして。
初次见面。
ha ji me ma xi te02、どうぞよろしく。
请多关照。
do......jio lu xi ku03、そうですか。
是吗？so......de si ka04、はい。
是的。
hai05、そうです。
是那样的（是的）。
so......de si06、いいえ。
不对（不是）。
i i e07、おはよございます。
早上好！o ha yo go za i ma si08、こんにちは。
你好！kon ni qi wa09、こんばんは。
晚上好！kon ban wa10、おやすみなさい。
晚安（您休息吧）！o ya si mi ta qi......11、ご饭（はん）ですよ。
吃饭了！go han te si yo12、ただいま。
我回来了。
ta dai ma13、あしたまた。
明天见。
a xi ta ma ta第二部分中文意思: 早上好！汉语拼音发音：ou ha you中文意思: 晚上好！汉语拼音发音：kong ba wa中文意思: 晚安汉语拼音发音：ou ya si ni中文意思: 你好吗？汉语拼音发音：kong ni ji wa中文意思: 我回来了！汉语拼音发音：ta da yi ma中文意思: 等一下！汉语拼音发音：ma dai中文意思: 老头子！汉语拼音发音：ou ji sang中文意思: 父亲汉语拼音发音：(ou) dao sang中文意思: 儿子汉语拼音发音：mu si gao中文意思: 真的！？汉语拼音发音：hong dou ni中文意思: 我明白了！汉语拼音发音：wa ka da wa中文意思: 对不起！汉语拼音发音：gu min na sa yi中文意思: 没关系！？不要紧！？汉语拼音发音：dai zou bu中文意思: 可爱、可爱的。
sdpp串行数据包协议技术手册Ver200(中性)
SDPP串行数据包协议技术手册Version2.00目录一、概述 (3)二、物理层描述 (3)三、传送层 (4)1、数据包格式 (4)2、数据包的重发标志和“转义插入” (4)四、控制层 (5)1、控制概述 (5)2、从机地址 (5)2.1 从机地址分配 (5)2.2 发卡器从机地址设臵 (5)3、通用命令和应答 (5)3.1 通用主机发送命令 (5)3.2 通用从机应答命令 (6)4、发卡器（TCD）命令和应答 (7)4.1 主机发送命令 (7)4.2 发卡器（TCD）应答命令 (8)4.3 发卡器（TCD）命令操作 (9)5、收卡器（TCR）命令和应答 (11)5.1 主机—TCR命令 (11)5.2 收卡器（TCR）应答命令 (12)5.3 收卡器（TCR）命令操作 (13)五、附录 (14)1、CRC校验算法程序 (14)2、版本更改记录 (16)一、概述SDPP（Serial Data Packet Protocol）是一个简单可靠的串行数据包通讯协议。
该协议以数据包方式传送数据，在每一个传送的数据包中都采用16位CRC校验来保证数据的完整性、正确性和可靠性。
SDPP主要特点:∙使用通用串行口（UART）即可进行控制∙四总线系统：两根数据线和两根电源线（+24V,GND,TXD,RXD）∙RS232C驱动方式，接口更加容易∙符合一个主机多个从机的半双工通讯要求∙9600 波特高速传输∙16 位 CRC 错误校验使用SDPP的好处：∙控制接口简单容易、价格低廉，占用硬件资源极少（仅用一个串口）∙由于采用总线型控制，方便系统扩展和升级∙对系统支付设备（如发卡器、收卡器、识币器等）控制可靠性高∙开放的协议标准，轻松地集成到你的系统之中二、物理层描述通讯采用标准的8位异步数据传送格式。
数据传送格式如下：编码：NRZ波特率：9600双工：半双工起始位：1位数据位：8位较验位：无停止位: 2位外围设备的电源可以与串行总线一起传送，也可以分开传送。
NOKIAMSCDX200基础知识
目录NOKIA DX200（MSC）平台 ```硬件一，DX200的硬件概况二，DX200各单元的PIU三，DX200各单元的功能NOKIA MSC DX200平台软件一，软件装载方式二，软件包基本知识三，软件包结构四，软件包的管理五，软件包的状态迁移NOKIA DX200（MSC ） 平台硬件一， DX200的硬件概况1， DX200平台概况A ） D X200平台的特性全分散的处理结构：具有较差的可靠性，易于管理：属于分散控制方式硬件模块化：灵活，可根据需要来做不同的配置：可根据情况增加单元所有的单元具有共同的插件：各单元的基本插板PIU 相同软件模块化：方便引入新的功能B ） 平台结构基本图C ） N OKIA DX200 MSC 各单元连接结构图2，DX200单元分类按功能分类A，信令单元CCSU，BSU，CCMU，CASU等B，交换有关的单元GSW，M，DTMFG，TGFP，CNFCC，与数据库和统计有关的单元D，与外部接口和数据有关的单元E，其他单元按结构A，交换单元GSW，M等B，计算机单元信令单元，CCMU，CMU等C，其他单元CLS，ET，ECU3，DX200单元A）D X200公共单元ET，M，GSW，MB，OMU，CM B）其余的单元CCMU，CMU，STU，CHU，VLRU，BDCU，CLSU，CCSU，BSU，IWCU，ECU，DASU，LSU，CASU，CNFC，VANG二，DX200各单元的PIU1，OMU单元WTI_I:P:OMU,1IDENTIFY PLUG-IN UNIT TYPE:MCU_S 0 人工控制单元，可人工控制选MASS_S 0 预处理器，时钟控制WDD 0 硬盘MBIF_U 1 MB接口板MBIF_U 0CP4E64 0 CPU插板SCSIF 0 连接I/O设备（FDU，CTU）的接口板AAL_S 1 外部告警单元AAL_S 0SERO 1 LAN连接接口SERO 02，M单元WTI_I:P:M,1IDENTIFY PLUG-IN UNIT TYPE:TG 0 信号音发生器SWCOP 2 交换控制部分MBIF_U 1MBIF_U 0SWCOP 1SWCOP 0CP4HX 0VANG_S 0 录音通知板DTMFG 0 双音多频发生器CLB 2 时钟BUFFER，给其他的单元提供时钟CLB 1CLB 03，GSW单元WTI_I:P:GSW,0,,/* IDENTIFY PLUG-IN UNIT TYPE:SWCSM 8 交换控制和交换MEMORYSWCSM 10PSC1 0 电源提供SWSPS 14 串/并、并/串变换 SWSPS 12SWSPS 10SWSPS 9SWSPS 8SWCSM 9SWCSM 1SWCSM 2PSC1 0SWSPS 6SWSPS 4SWSPS 2SWSPS 1SWSPS 0SWCSM 0SWCSM 16SWSPS 18PSC1 0SWSPS 16SWCSM 18SWCSM 174，ET单元WTI_I:P:ET,234/* IDENTIFY PLUG-IN UNIT TYPE:ET1E 0 ET接口板5，CM单元WTI_I:P:CM,1IDENTIFY PLUG-IN UNIT TYPE:CP4HX 0MBIF_U 1MBIF_U 06，STU单元WTI_I:P:STU,1IDENTIFY PLUG-IN UNIT TYPE:WDD 0MBIF_U 1MBIF_U 0CP4HX 07，CCSU单元WTI_I:P:CCSU,1/* IDENTIFY PLUG-IN UNIT TYPE:AFS_S 0 预处理器，用于收集ET告警信息MBIF_U 1MBIF_U 0AS7_U 1 处理CCS7，4条LINK/块，最多2块/CCSUAS7_U 0CP4HX 0注：AS7-S 1条LINK/块，最多8块/CCSUAS7-V 16条LINK/块，最多1块/CCSU（在M9后CCSU可处理16条LINK）8，CHU单元WTI_I:P:CHU,1/* IDENTIFY PLUG-IN UNIT TYPE:WDD 0MBIF_U 1MBIF_U 0SCSIF 0CP4HX 09，CCM单元WTI_I:P:CCM,1/* IDENTIFY PLUG-IN UNIT TYPE:CP4HX 0MBIF_U 1MBIF_U 010，BSU单元WTI_I:P:BSU,1;__[J/* IDENTIFY PLUG-IN UNIT TYPE:AFS_S 0MBIF_U 0AS7_U 1AS7_U 0CP4HX 011，IWCU单元WTI_I:P:IWCU,1/* IDENTIFY PLUG-IN UNIT TYPE:AS7_U 0 可提供CCS7 LINK，X.25,LAPD MBIF_U 1MBIF_U 0CP4HX 0AS7_U 5AS7_U 4AS7_U 3AS7_U 2AS7_U 112，CMU单元WTI_I:P:CMU,1/* IDENTIFY PLUG-IN UNIT TYPE:CP4HX 0MBIF_U 1MBIF_U 013，VLRU单元WTI_I:P:VLRU,1,1：/* IDENTIFY PLUG-IN UNIT TYPE:CP4HX 0MBIF_U 1MBIF_U 014，BDCU单元WTI_I:P:BDCU,1/* IDENTIFY PLUG-IN UNIT TYPE:CP4HX 0AC25_S 4 用于模拟X.25COCEN 3AS7_U 2 用于数字的X.25AC25_S 1AC25_S 0MBIF_U 1MBIF_U 0注：在BDCU中可能有COCEN板，此板用于LAN网的连接15，CLS单元WTI_I:P:CLS,1/* IDENTIFY PLUG-IN UNIT TYPE:CLPM 0 时钟相位CLOS 0 晶源CLCP 0 时钟控制处理器16，ECU单元WTI_I:P:ECU,234/* IDENTIFY PLUG-IN UNIT TYPE:EC1P 017，CNFC单元WTI_I:P:CNFC,0/* IDENTIFY PLUG-IN UNIT TYPE:CNFC 018，MPC单元WTI_I:P:MPC,0,,/* IDENTIFY PLUG-IN UNIT TYPE:COCA 0DM2 0VF10 2VF10 1VF10 0PSC1 0DASA 7DASA 6DASA 5DASA 4DASA 3DASA 2DASA 1DASA 0ECMFA 0FAXMO 9FAXMO 8FAXMO 7ECMFA 6ECMFA 5ECMFA 4ECMFA 3ECMFA 2ECMFA 1FAXMO 10ECMFA 19ECMFA 18ECMFA 17FAXMO 16FAXMO 15FAXMO 14FAXMO 11FAXMO 12FAXMO 13三，DX200各单元的功能1，OMU ... OPERATION AND MAINTENANCE UNIT包括检测，ALARM和恢复功能，人机命令接口。
Expression of MiR200a, miR93, Metastasis-related Gene RECK and MMP2_MMP9 in Human Cervical C
Asian Pacific Journal of Cancer Prevention, Vol 14, 20132113DOI:/10.7314/APJCP .2013.14.3.2113MiR200a, miR93, RECK and MMP2/MMP9 Expression in Human Cervical CarcinomaAsian Pacific J Cancer Prev, 14 (3), 2113-2118IntroductionIn the worldwide, cervical cancer remains the third most common cancer in women globally after breast and colorectal cancer. However, 86% of all deaths caused by cervical cancer occur in developing countries (Arbyn et al., 2011). Data from the IARC GLOBOCAN 2008 database (http://globocan.iarc.fr/fact sheets/cancers/cervix.asp) estimate that there are 529, 512 new cases of cervical cancer diagnosed per year globally, corresponding to an age standardized incidence rate (ASIR) of 15.2/100,000 and 274,967 deaths. There is a striking difference in incidence of and mortality from cervical cancer in different regions of the world (Denny, 2012) So well-characteristic biomarkers are necessary for early diagnosis, to predict metastatic progression.Metastatic disease, rather than the primary tumor itself, is responsible for the death in most solid tumors, including cervical carcinoma (Lee et al., 1997; Welch et al., 2000; Yang et al., 2004). Degradation of basal membranes and the extracellular matrix (ECM) is essential for angiogenesis, invasion metastasis, and matrixDepartment of Obstetrics and Gynecology, the Second Affiliated Hospital of Jilin University, Changchun, Jilin, China *For correspondence: zanghu@AbstractAim and Background: Cervical cancer remains the third most common cancer in women globally after breast and colorectal cancer. Well-characterized biomarkers are necessary for early diagnosis and to predict metastatic progression and effective therapy. MiRNAs can regulate gene expression, cell growth, differentiation and apoptosis by targeting mRNAs for translational repression or degradation in tumor cells. The present study was conducted to assess expression of miR93, miR200a, RECK, MMP2, MMP9 in invasive cervical carcinoma, and analyze their clinical significance. Method: A total of 116 patients with invasive cervical carcinoma and 100 patients undergoing hysterectomy for benign lesions were retrospectively examined. Quantitative real-time PCR was performed to determine expression of miR93 and miR200a while RECK, MMP2, MMP9 and MVD were assessed by immunohistochemical staining. Results: Cervical carcinoma patients demonstrated up-regulation of miR-93, miR-200a, MMP2 and MMP9, with down-regulation of RECK as compared to benign lesion tissues. RECK was significantly inversely related to invasion and lymphatic metastasis. The 5-year survival rate for patients with strong RECK expression was significantly higher than that with weakly expressing tumors. Conclusion: MiR-93 and miR-200a are associated with metastasis and invasion of cervical carcinoma. Thus together with RECK they are potential prognostic markers for cervical carcinoma. RECK cooperating with MMP2, MMP9 expression is a significant prognostic factor correlated with long-term survival for patients with invasive cervical carcinoma.Keywords: miR-93 - miR-200a - RECK - MMP2 - MMP9 - MVD RESEARCH ARTICLEExpression of MiR200a, miR93, Metastasis-related Gene RECK and MMP2/MMP9 in Human Cervical Carcinoma - Relationship with PrognosisLing Wang, Qiang Wang, He-Lian Li, Li-Ying Han*metalloproteinases (MMPs) are potent enzymes that play a key role in these processes (Sabrina et al., 2012).Matrix Metalloproteinase 2 (MMP-2) (gelatinase A, 72 kDa) and Matrix Metalloproteinase 9 (MMP-9) (gelatinase B, 92 kDa) cleave type IV collagen and gelatin, which are the main structural components of the basal membrane (Toi et al., 1998). Expression of MMP-9 and MMP-2 has been implicated in the development and progression of many tumors, such as prostate, colorectal, breast cancer and cervical cancer (Liabakk et al., 1996; Kodate et al., 1997; Eissa et al., 2007; Rita et al., 2009).Several miRNAs are reported be associated with cervical carcinoma, Up-regulation of miR-200a and miR-93 promotes metastasis and tumor invasion. According to computational methodology current predictions-MicroCosm MMP2 is target gene to miR93, and TIMP1 is target gene to miR200a, while TIMP3 is target gene to miR93.MicroRNAs (miRNAs) is a novel class of small non-coding RNA molecules, 20-25 nucleotides in length, were shown to have important posttranscriptional gene regulatory functions. While miRNAs seed regionLing Wang et alAsian Pacific Journal of Cancer Prevention, Vol 14, 20132114which comprised of 2-8 nucleotides at 5’ end, target to special mRNA at 3’ untranslated region (UTR) (Bartel et al., 2009; Kim et al., 2009). If the complementarity of the miRNA-mRNA complex is perfect, miRNAs can exert translational repression function. However, if the complementarity is not perfect, the translation of the target mRNA is suppressed. To date, more than 1900 human mature miRNAs have been identified (http://www.miRbase. org/index. shtml), which are supposed to regulate more than 10% of protein coding genes (Wu et al., 2008), approximately one-third of expressed human genes contain miRNA regulatory target sites. Thus, this suggests that different clusters of miRNAs can regulate the cassette of specific genes which involve in one specific kind of cellular function together (Yang et al., 2003). It has been reported that RECK over-expression decreases the amount of active MMP-2 and MMP-9 and inhibits metastatic activity in vitro (Oh et al., 2001) and in vivo (Chang et al., 2008). RECK is a membrane-anchored glycoprotein of approximately 110 kDa containing multiple epidermal growth factor-like repeats and serine protease inhibitor-like domains. Down-regulation of RECK in several tumor cell lines and oncogene-transformed fibroblasts identified RECK as a common negative target for oncogenic signals. RECK low-expression, a hallmark of cancer, has been demonstrated to create a hypoxic tumor microenvironment.The aim of our study was to test the expression of miR200a and miR93 in cervical carcinoma, we propose the induction of the correlation between the expression of miR200a, miR93 and MMP2/9,RECK genes and to investigate whether miR200a, miR93 and MMP-2, MMP2/9 and RECK are expressed in a related pattern respectively in cervical carcinoma. Furthermore, we evaluate important prognostic parameters, analyzed the expression of RECK with 5-year survival rate, to conclude whether RECK is an independent factor to evaluate prognosis.Materials and MethodsTissue specimensCervical carcinoma specimens were obtained from patients undergone primary hysterectomy at the Department of Gynecology from Jan 2005 to Sep 2007, while control group were obtained from patients undergone hysterectomy for benign lesion. The specimens were frozen in liquid nitrogen at -80 ℃ within 30 minutes after isolated, and all cases were obtained from archives of the Department of Pathology in the Second Affiliated Hospital of Jilin University. The H&E stained slides of the cases were reviewed by gynecological pathologist. Morphology and protein expression were evaluated in consecutive sections. All protocols were reviewed and approved by the Ethical Committee of Second Affiliated Hospital of Jilin University. Written consent was obtained from all participating patients.Follow-upPatients were followed regularly for 5 years at the Second Affiliated Hospital of Jilin University. All patientswere followed until death or the study closing date (September 30, 2012). Disease-free survival (DFS) rate, which measured the first recurrence at any site, and overall survival (OS), measuring death from any case, were the two assessments used for prognostic analyses. Patients were re-examined (history, ultrasound examination, cervical screening test) once every 3 months during the first year, once every 6 months from the second year to the third year, and once every year after that. During the follow-up period, 6 patients were loss of follow up. 26 patients had disease recurrence and 36 patients died. miRNA isolationMiRNA was extracted from the tissue using the mirVana miRNA Isolation Kit (AM1561, Ambion) for hysterectomy specimen according to the protocols. The quantity and quality of the miRNA was verified with the NanoDrop spectrophotometer (Thermo Fisher Scientific Incorporated, Wilmington DE, USA) according to the manufacturer’s instructions.Quantitative real-time PCR (QPCR)miRNAs was reverse transcribed in a 20 μl reaction using the one step primescript miRNA cDNA Synthesis Kit (Takara, D350A). Forward primer sequences miR93: CAAAGTGCTGTTCGTGCAGGTAG, miR200a: GTAA CACTGTCTGGTAACGATGQPCR was performed on a BioMad Real-Time PCR System (ABI) using Power- SYBR Green PCR Master Mix (Takara, DRR081) in a 20 μl reaction and U6 as an endogenous control, miRlet-7 as positive control, result was determined using the 2-ΔΔCT . The QPCR experiments were run triplely within each experiment run, relative expression values were normalized to standard deviations from the mean.Immunohistochemical stainTo determine the expression of RECK, MMP2, MMP9 and MVD, immunohistochemical staining was carried out using the two-step plus poly-HRP method as described previously. After blocking with 3% hydrogen peroxide, the slides were incubated with primary anti-RECK antibody, anti-MMP2 antibody, anti-MMP9 antibody and CD34 antibody (1:50 goat mAb respectively; Santa Cruz Biotechnology, Santa Cruz, CA, USA). Afterwards, the slides were stained with the two-step plus poly-HRP antigoat IgG detection system (ZSGB-Bio, Beijing, China). For negative controls, the primary antibody was substituted with PBS in order to confirm the specificity of the primary antibody.Evaluation of immunohistochemical stainingTwo experienced investigators, who provided a consensus opinion of stain patterns by light microscopy, evaluated sections. RECK, MMP2 and MMP9 expression was estimated from the staining intensity and graded as follows: Grade 0, no staining (-); Grade 1, faint staining (+); Grade 2, moderate staining (++); and Grade 3, strong staining (+++). The positively stained area (distribution) was expressed as the percentage of the whole area under evaluation and scored as follows: 0, no staining; 1, 1~25% positive cells; 2, 26~50% positive cells; 3, 51~75%Asian Pacific Journal of Cancer Prevention, Vol 14, 20132115P0.0002 0.0193 0.0062Table 2. Correlation Between RECK Expression andVarious Clinicopathological Features in Cervical Cancer PatientsN RECK positive χ2 Pnegative positive (%)Stage I 64 33 31 48.44 6.198 0.1024 II 33 21 12 36.36 III 17 12 5 29.41 IV 2 2 0 0 Grade I 23 13 10 43.48 3.6204 0.1636 II 63 42 21 33.33 III 30 15 15 50 Invasive depth T2 66 43 23 34.85 0.0184 T3-T4 50 36 24 48 Lymph node status N1-N3 15 14 1 6.67 0.0237 N0 101 58 53 52.48 Squamous carcinoma 99 60 39 39.4 1 adenocarcinoma 17 11 6 35.29in Cervical Carcinoma in Relation to Normal TissueA BFigure 2. Expression of RECK, MMP2, MMP9. (A)expression of RECK in control group; (B) expression of RECK in cervical carcinoma; (C) expression of MMP2 in cervical carcinoma; (D) expression of MMP9 in cervical carcinoma; (E) MVD expression in control group; (F) expression of MVD in cervical carcinomapositive cells; and 4, 76~100% positive cells. Overall expression was then graded as low expression (score 0~2), intermediate expression (score 3~5), and high expression (score 6~7).Statistical analysisAnalysis were carried out by BioMad CFX system and statistical software SPSS 14.0 (SPSS, Chicago, IL, USA). The correlation of RECK, MMP2/9 expression with patients’ clinicopathological factors was analyzed by the Fisher’s exact test. The Kaplan-Meier method was used to estimate OS. Survival differences according to RECK expression were analyzed by the log-rank test. The risk ratio and its 95% confidence interval were recorded for each marker. P -values< 0.05 were considered statistically significant in all of the analysis.ResultsMean age of the total 116 patients was 49.3±2.39 years (24~77 years). 36 patients (31.03%) died, and 74 patients (63.79%) were alive at the end of research. Results of qPCR showed that miR93 and miR200a expression was higher in cervical carcinoma tissues (Figure 1A, Figure 1B). RECK was detected in the cytoplasm of normal cells (Figure 2A, Figure 2B), and of cervical carcinoma specimen its expression was much lower than that in control group, MMP9, MMP2 was detected in the cytoplasm of cells (Figure 2C, Figure 2D). MMP2 and MMP9 expression was significantly higher in cervical carcinoma than that in control group (Table 1). Accordingto Cox regression analysis result, the expression of MMP2 was positively related to expression of miR93 (P =0.0027) and miR200a (P =0.0016). However, higher expression of RECK related to lower expression of MMP2, MMP9. We also examined positive RECK staining in different clinopathological factors such as stage, grade, invasion depth and lymph node metastasis (Table 2). These data indicated that the frequency of RECK expression in high-grade was much higher than in low-grade cervical carcinoma. RECK expression, however, was significantly associated with lymph node metastasis (P =0.0237)Ling Wang et alAsian Pacific Journal of Cancer Prevention, Vol 14, 20132116and invasive depth (P =0.0184). Lymph node negative patients had higher RECK expression (53/101, 52.48%) than lymph node positive patients (1/15, 6.67%). Deeper invasive patients had lower RECK expression (24/50, 48%) than lower invasive patients (23/66, 34.85%). We found that histopathological grade, pathological TNM stage have no significance as prognostic predicators (Table 2). Multivariate analysis was carried out on the same set of patients for RECK expression and pathological predictors using the Cox regression model. The results indicated that RECK status (risk ratio, 3.312; P <0.05) was independent prognostic factor.Microscopic observation of MVD staining showed that in cervical carcinoma group, micro-vascular arranged disorderly, size and shape were irregular, thickness of vascular wall was nonuniform (Figure 2F), while in control group, clearer expression of micro-vascular endothelial cells were round or oval and in regular shape (Figure 2E). According to the Cox regression analysis, RECK expression in cervical carcinoma was negatively associated with MVD value (r=-0397, p =0.0495) (Table 3).To determine the relation between RECK expression and prognosis, Patients were divided into two groups on the basis of their prognosis. Our results indicated that patients with a poor prognosis(recurrence or metastasis) had low levels of RECK expression (P <0.05). Kaplan-Meier survival analysis showed that RECK positive patients also had significantly higher OS rates (P <0.05, log-rank test; Figure 3A).RECK positive patients had higher DFS rates compared with RECK negative patients (P =0.0387, log-rank test; Figure 3B).DiscussionAccumulating reports demonstrated that miRNAs have been observed in a variety of human cancers, and miRNA signatures accurately reflect the developmental lineagesand differential expression states of tumors by microarray profiling studies (Lu et al., 2005; Rosenfeld et al., 2008) Furthermore, they certified miRNAs involved in tumor cells invasion, apoptosis, angiogenesis and metastasis through regulation to target genes of corresponding signal pathways (MA et al., 2012). In previous research on human cervical cancer, expression of miR-15a, miR-20b, miR-21 and miR-224 is obviously increased in tissue and let-7c, miR-143, miR-199a-5p, miR-203 and miR-145 is reduced (Pereira et al., 2010; Wang et al., 2008). In our study, miRNAs expressions were quantified by using quantitative real-time PCR in which miR-93, miR-200a were up-regulated in cervical carcinoma tissue. Their expressions were accompanied by over-expression in MMP-2, MMP-9 and suppression in RECK gene.The miR-93 gene is located on chromosome 7q22.1, it can suppress proliferation and differentiation of cancer stem cells, while promoting tumor growth and malignant cells survival (Fang et al., 2011; Yu et al., 2011; Suling et al., 2012). In the present study, mir-93 expression was 5.29 fold higher compared to normal tissue. Our data are consistent with other reports indicating that mir-93 expression increased with cervical carcinoma (Lui et al., 2007). By microcosm predictor system, MMP2 has the target gene in 3’ UTR to miR-93, also TIMP3 is the proposal target gene of miR-93 ,whose sequence GAUGGACGUGCUUGUCGUGAAA was relatively complemented with CTTTCTATGTGCAAGGCACTTT in TIMP3 (/enright-srv /microcosm/htdocs/targets/v5). Endogenous angiogenesis inhibitors TIMPs are necessary to block the mitogenic stimuli in the vascular endothelium (Curran et al., 2000). TIMP3 is inhibitor of MMP2 and associated with actin and serve to stabilize microfilaments, so it act as tumor suppressor gene (Perry, 2001). miR93 was indentified up-regulated expression of MMP2 in cervical carcinoma, for the up-regulated mir-93, the inhibiting function of tumor suppressor genes TIMP3 maybe suppressed. But this hypothesis has not been certified. The miR-200a gene is located on chromosome 1P36.33, and can enhance invasion and growth of malignant cells. In this study, miR-200a was over-expressed by 3.65 folds in cervical carcinoma compared to normal tissues respectively. Similar studies indicated the miR-200a was up regulated which may supporting the concept that miR-200a functions as oncogene (Cong et al., 2013; Rasheed et al., 2013; Yu et al., 2013). In our study, the over-expression of miR-200a was associated with the over-expression of MMP2, MMP9. According to Microcosm target gene predictor system, TIMP1 is the corresponding target gene of miR-200a. miR-200a has been supposed to involve in down-regulating of TIMP1, whose inhibitor function to MMP2 and MMP9 is weakened and led to over-expression of MMP2 and MMP9. Similar research instructed that miR-200b is overexpressed in endometrial adenocarcinomas and enhances MMP2 activity by down regulating TIMP2 in human endometrial cancer cell Line HEC-1A cells (Dai et al., 2013). But the accurate mechanism in cervical carcinoma still need to certified in vitro until now.In this research, RECK expression is suppressedTable 3. Expression of MVD in Cervical Carcinoma and Control Tissue (n)P n MVD P RECK r (n/*400) (positive) [n(%)] Cervical carcinoma 116 19.4615±3.0718 0.000 48(40.31) -0.397Control group 100 12.0000±2.6629 0.0495 86(86.0)Figure 3. Kaplan–eier Analysis for Disease-free Survival (DFS) and Overall Survival (OS) Based on RECK Expression in Cervical Carcinoma Patients.(A) Kaplan–Meier analysis for OS based on RECK expression in patients with cervical carcinoma (P =0.0399, log–rank test); (B) Kaplan–Meier analysis for DFS based on RECK expression in patients with cervical carcinoma (P = 0.0387, log–rank test). RECK(+): RECK-positive patients (n =48 ); RECK(-): RECK-negative patients (n = 68)Asian Pacific Journal of Cancer Prevention, Vol 14, 20132117DOI:/10.7314/APJCP .2013.14.3.2113MiR200a, miR93, RECK and MMP2/MMP9 Expression in Human Cervical Carcinomain cervical carcinoma tissue when compared to benign lesion tissue. There are almost certainly pathways by which RECK is down-regulated in cancer. Hypoxia induces RECK down-regulation through the recruitment of HDAC1 and HIF-1α to the rHRE2 site in the promoter and the inhibition of hypoxic RECK silencing would be a therapeutic and preventive target for early tumorigenesis (Zhang et al., 2012). However, the CpG island promoter hypermethylation is associated silencing of tumor suppressor genes, which is the most recognized epigenetic disruption in human tumors (Rodriguez et al., 2011). Low RECK expression is closely correlated with high MMP2, MMP9 expression. In addition, increased expression of MMP2, MMP9 with decreased expression of RECK in invasive cervical carcinoma irrespective of histological grading supports the fact that RECK has a negative effect on the invasiveness of cervical cancer. Mori had found (Mori T et al., 2007) that MMP-2 activity, but not its mRNA expression, was significantly down-regulated in HT1080 cells after they were transferred into the RECK plasmid (Bin Zhang et al., 2009). Similarly, our results showed a negative correlation between RECK and MMP-2 protein expression. In HUVECs, specific inhibition of MMP-2 significantly antagonized the effect of RECK depletion on β1-integrin signaling, cell proliferation, and tube elongation (Namwat et al., 2011). Moreover, RECK-mediated suppression of MMP-9 promoter activity requires 12-O- tetradecanoylphorbol-13-acetate-responsive element (TRE) and KB sites. Moreover, the binding ability of Fra-1 and c-Jun to TRE within the MMP-9 promoter region was suppressed by RECK (Satoshi et al., 2007). In this research, MVD CD34 for tumors with lower-expression RECK is obviously increased, which indicates that RECK can inhibit angiogenesis. Targeting RECK specifically in tumor-associated vascular endothelial cells resulted in tumor regression (Takao et al., 2010).RECK positive patients showed higher 5-year survival rates and DFS rates. Furthermore, we found that RECK expression was significantly associated with lymph node metastasis and deeper invasion. HER-2/neu oncogene inhibits the expression of RECK to promote cell invasion (Tsung-Te et al., 2012). Hypermethylation of RECK promoter is also a common event in human ESCC, which occurs concurrently in tumor-adjacent normal mucosa and is correlated with poor prognosis in ESCC patients (Long NK et al., 2008). RECK displays as a metastasis suppressor and up-regulation of RECK expression could provide a potential therapy to improve the prognosis (Namwat et al., 2011).In conclusion, MiR-93, miR-200a is associated with metastasis and invasion of cervical carcinoma, thus MiR-93, miR-200a, RECK expression is a potentially prognostic marker for cervical carcinoma. RECK cooperating with MMP2, MMP9 expression is a significant prognostic factor correlated with long-term survival for patients with invasive cervical carcinoma.ReferencesArbyn M, Andersson K, Bergeron C, et al (2011). Cervicalcytology biobanks as a resource for molecular epidemiology. Methods Mol Biol , 675, 279-98.Bartel DP (2009). MicroRNAs, target recognition and regulatoryfunctions. Cell , 136, 215-33.Chang CK, Hung WC, Chang HC (2008). The Kazal motifs ofRECK protein inhibit MMP-9 secretion and activity and reduce metastasis of lung cancer cells in vitro and in vivo. J Cell Mol Med , 12, 12-6.Cong N, Du P , Zhang A, et al (2013). Downregulated microRNA-200a promotes EMT and tumor growth through the wnt/β-catenin pathway by targeting the E-cadherin repressors ZEB1/ZEB2 in gastric adenocarcinoma. Oncol Rep , 10, 3892-7.Curran S, Murray GI (2000). Matrix metalloproteinases,molecular aspects of their roles in tumour invasion and metastasis. Eur J Cancer , 36, 1621-30Dai Y, Xia W, Song T, et al (2013). MicroRNA-200b isoverexpressed in endometrial adenocarcinomas and enhances MMP2 activity by downregulating TIMP2 in human endometrial cancer cell line HEC-1A cells. Nucleic Acid Ther , 23, 29-34.Denny Lynette (2012). Cervical cancer, prevention andtreatment. Discov Med , 14, 125-11.Eissa S, Ali-Labib R, Swellam M, et al (2007). Noninvasivediagnosis of bladder cancer by detection of matrix metalloproteinases (MMP-2 and MMP-9) and their inhibitor (TIMP-2) in urine. Eur Urol , 52, 1388-96.Fang L, Deng Z, Shatseva T, et al (2011). MicroRNA miR-93promotes tumor growth and angiogenesis by targeting integrin-β8. Oncogene , 30, 806-21.Figueira RC, Gomes LR, Neto JS, et al (2009). Correlationbetween MMPs and their inhibitors in breast cancer tumor tissue specimens and in cell lines with different metastatic potential. BMC Cancer , 9, 20-5.Liu S, Patel SH, Ginestier C, et al (2012). MicroRNA93 regulatesproliferation and differentiation of normal and malignant breast stem cells. PLoS Genet , 8, e1002751.Kim VN, Han J, Siomi MC (2009). Biogenesis of small RNAsin animals. Nat Rev Mol Cell Biol , 10, 126-39.Kodate M, Kasai T, Hashimoto H, et al (1997). Expression ofmatrix metalloproteinase (gelatinase) in T1 adenocarcinoma of the lung. Pathol Int , 47, 461-9.Lee JH, Welch DR (1997). Suppression of metastasis in humanbreast carcinoma MDA-MB-435 cells after transfection with the metastasis suppressor gene, KiSS-1. Cancer Res , 57, 2384-7.Liabakk NB, Talbot I, Smith RA, et al (1996). Matrix metallo-proteinase 2 (MMP-2) and matrix metalloproteinase 9 (MMP-9) type IV collagenase in colorectal cancer. Cancer Res , 56, 190-6.Lim LP, Lau NC, Garrett-Engele P, et al (2005). Microarrayanalysis shows that some microRNAs downregulate large numbers of target mRNAs. Nature , 433, 769-73.Long NK, Kato K, Yamashita T, et al (2008). Hypermethylationof the RECK gene predicts poor prognosis in oral squamous cell carcinomas. Oral Oncol , 44, 1052-8.Lu J, Getz G, Miska EA, et al (2005). MicroRNA expressionprofiles classify human cancers. Nature , 435, 834-8.Lui WO, Pourmand N, Patterson BK, Fire A (2007). Patternsof known and novel small RNAs in human cervical cancer. Cancer Res, 67, 6031-43.Ma D, Zhang YY , Guo YL, Li ZJ, Geng L (2012). Profiling ofmicroRNA-mRNA reveals roles of microRNAs in cervical cancer. Chin Med J , 125, 4270-76.Mori T, Moriuchi R, Okazaki E, et al (2007). Tgat oncoproteinfunctions as a inhibitor of RECK by association of the unique C-terminal region. Biochem Biophys Res Commun ,Ling Wang et alAsian Pacific Journal of Cancer Prevention, Vol 14, 20132118355, 937-43.Namwat N, Puetkasichonpasutha J, Loilome W, et al (2011).Downregulation of reversion-inducing-cysteine-rich protein with Kazal motifs (RECK) is associated with enhanced expression of matrix metalloproteinases and cholangiocarcinoma metastases. J Gastroenterol, 46, 664-75.Oh J, Takahashi R, Kondo S, et al (2001). The membraneanchored MMP inhibitor RECK is a key regulator of extracellular matrix integrity and angiogenesis. Cell , 107, 789-800.Pereira PM, Marques JP, Soares AR, Carreto L, Santos MA(2010). MicroRNA expression variability in human cervical tissues. PLoS One , 5, e11780.Perry SV (2001). V ertebrate tropomyosin, distribution, propertiesand function. J Muscle Res Cell Motil , 22, 5-49.Rasheed SA, Teo CR, Beillard EJ, V oorhoeve M, Casey PJ(2013). MicroRNA-182 and microRNA-200a control G-protein subunit alpha-13 (GNA13) expression and cell invasion synergistically in prostate cancer. Cells J Biol Chem , 10, 1074-7.Rodriguez-Paredes M, Esteller M (2011). Cancer epigeneticsreaches mainstream oncology. Nat Med , 17, 330-9.Rosenfeld N, Aharonov R, Meiri E, Rosenwald S, et al (2008).MicroRNAs accurately identify cancer tissue origin. Nat Biotechnol, 26, 462-9.Reis ST, Leite KR, Piovesan LF, et al (2012). Increasedexpression of MMP-9 and IL-8 are correlated with poor prognosis of Bladder Cancer. BMC Urol , 12, 18-23.Takagi S, Simizu S, Osada H (2009). RECK negatively regulatesmatrix metalloproteinase-9 transcription. Cancer Res , 69, 1502-8.Miki T, Shamma A, Kitajima S, et al (2010). TThe ß1-integrin-dependent function of RECK in physiologic and tumor angiogenesis. Mol Cancer Res , 8, 665-76.TChung TT, Yeh CB, Li YC, et al (2012). Effect of RECK genepolymorphisms on hepatocellular carcinoma susceptibility and clinicopathologic features. Plos one , 7, e33517.Toi M, Ishigaki S, Tominaga T(1998). Metalloproteinases andtissue inhibitors of metallo-proteinases. Breast Cancer Res Treat , 52, 113-24.Wang X, Tang S, Le SY , et al(2008). Aberrant expression ofoncogenic and tumor-suppressive microRNAs in cervical cancer is required for cancer cell growth. PLoS One, 3, e2557.Welch DR, Steeg PS, Rinker-Schaeffer CW (2000). Molecularbiology of breast cancer metastasis. Genetic regulation of human breast carcinoma metastasis. Breast Cancer Res , 2, 408-16.Wu L, Belasco JG (2008). Let me count the ways, mechanisms ofgene regulation by miRNAs and siRNAs. Mol Cell, 29, 1-7.Yang J, Mani SA, Donaher JL, et al (2004). Twist, a masterregulator of morphogenesis, plays an essential role in tumor metastasis. Cell , 117, 927-39.Yang L, Parkin DM, Li L, Chen Y (2003). Time trends in cancermortality in China, 1987-1999. Int J Cancer , 106, 771-83.Yu SJ, Hu JY, Kuang XY, et al (2013). MicroRNA-200aPromotes Anoikis Resistance and Metastasis by Targeting YAP1 in Human Breast Cancer. Clin Cancer Res , 10, 1158-63.Yu XF, Zou J, Bao ZJ, Dong J (2011). miR-93 suppressesproliferation and colony formation of human colon cancer stem cells. World J Gastroenterol , 17, 4711-7.Zhang B, Zhang J, Xu ZY , Xie HL (2009). Expression of RECKand matrix metalloproteinase-2 in ameloblastoma. BMC Cancer , 9, 427-35.Zhang C, Ling Y , Zhang C, et al (2012). The silencing of RECKgene is associated with promoter hypermethylation and poorsurvival in hepatocellular carcinoma. Int J Biol Sci , 8, 451-8.。
XSD402中文资料
PIN CONFIGURATION
SCHEMATIC DIAGRAM
DRAIN (2)
TO-92
GAT1-1
D S G
7-74
元器件交易网
SD400 / SD402
CORPORATION
ABSOLUTE MAXIMUM RATINGS (TC = +25oC unless otherwise noted) VDS VGS VDG Drain-Source Voltage SD400 . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . +30V SD402 . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . +15V Gate-Source Voltage . . . . . . . . . . . . . . . . . . . . . -0.3 V +20 V Gate-Drain Voltage . . . . . . . . . . . . . . . . . . . . . . -0.3 V +20 V VSD ID Source-Drain Voltage . . . . . . . . . . . . . . . . . . . . -0.3 V Continuous Drain Current . . . . . . . . . . . . . . . . . 50 mA Power Dissipation (at or below TC = +25oC) . . 300mW Linear Derating Factor. . . . . . . . . . . . . . . . 3.0 mW/ oC Operating Storage and Junction Temperature Range . . . . . . -55oC to +125oC
sdz200说明书-2
错误！链接无效。
4.2 通信模件本模件主要完成与I/O模件的通信、与GPS校时、与PLC通信、与其它装置通信的功能。
由于通信任务比较多，模件扩展了一路通信口。
各功能模块说明如下：●通信模块：通信模块提供了RS232接口和RS422/485接口，可视具体情况灵活配置。
该通信模块主要完成与其他装置的通信功能，以便将采集到的数据传输给监控系统，数据传输波特率由软件设定。
●GPS对时模块：对时模块主要用来校时，用GPS装置接收并传输来的同步时钟来及时更新本装置上的硬件时钟，以保持时钟准确。
其原理简图如图3-3：错误！链接无效。
●硬件时钟模块：装置本身有一硬件时钟，主要用来提供记录所需的时标。
该单元所用芯片内部带有电池，即使装置掉电，其内部时间也不丢失或停止，并且可保持最长达十年之久，从而保证了时间的连续性、可靠性。
●CAN总线接口模块：装置在通信板上留有CAN总线接口，用作与所有I/O板之间数据传输，以及与其他装置的数据交换。
装置所有模件之间采用CAN总线进行数据交换。
装置后背板CAN总线接口，可与其它装置就地联网。
●监控模块：系统能够自动恢复过来。
●报警模块：装置设有报警模块，主要用来监视通信、设置通知等，以便于装置调试。
4．3I/O模件I/O模件主要用来完成对被监控信号的监视、记录时标以及按需要控制输出等。
该模件主要有信号输入模块、显示输出模块、控制输出模块、CAN总线接口模块、监控模块等。
各模块说明如下：●信号输入模块：输入信号可以是有源的，也可为无源的。
由于现场的信号大多比较杂，而且干扰比较多，为了最大限度的减小信号带来的干扰，所有信号都经过光隔才引入系统处理。
●显示输出模块显示输出模块主要是用来显示板号、信号输入状态、信号输出状态以及调试用。
采用的是矩阵式二极管作为显示元件，共32个二极管，分为8行4列排列。
其中24个二极管用来显示24点输入信号的状态，另外8个则用来显示8路输出信号的状态。
信号的显示与输入信号、输出信号是一一对应的，若该路信号的当前状态为“1”，则对应通道的二极管点亮，对输入信号而言则表示该通道上的开关量输入为“1”，对输出信号而言则表示该通道有输出。
1-ZXMP S200 V2.0设备总体介绍
四光口 100Ω8 T1S200 系统主 板： SMBxF 100E0
四光口 100Ω2 1T1S20 0系统 主板： SMBxF 100F0
双光口S200型系统主板
四光口S200型系统主板
系统主板SMB
中兴通讯版权所有
ZXMP S200设备构架
E1/T1电支路板与PCB的对应关系
E1/T1电支路板：ET1
平台化设计：做到系列化产品分步实现，包含V1.00/V2.00/V2.10/V2.20四个版
本，V1.00开发一块系统主板，后续各个版本主要是增加功能插板，并完善SMB软件 功能。 兼容性强：传输产品整体需要，可与传输其它产品对接、互通。
中兴通讯版权所有
内容提纲
ZXMP S200设备市场定位
平台化设计
ZXMP S200 V2.00 ZXMP S200 V2.10 ZXMP S200 V2.20
市场化进程
ZXMP S200 V3.00
中兴通讯版权所有
市场化进程
内容提纲
ZXMP S200设备市场定位
内部公开▲
ZXMP S200设备整体特点
ZXMP S200命名和版本说明 ZXMP S200设备构架 ZXMP S200设备功能和性能 ZXMP S200设备应用环境说明
主板
中兴通讯版权所有
ZXMP S200设备构架
ZXMP S200 V2.00设备物理参数
造型图
内部公开▲
外形尺寸：与ZXMP S200 V1.00相同
构成：比ZXMP S200 V1.00多一个功能插板槽位 重量：5.5kg（视功能插板类型有差异） 典型配置功耗：28W （视功能插板类型有差异）
西门子 SIMATIC ET 200SP 数字量输出模块 DQ 8x24VDC 0.5A ST (6
SIMATICET 200SP数字量输出模块DQ 8x24VDC/0.5A ST (6ES7132-6BF01-0BA0) 设备手册Siemens AG Digital Industries Postfach 48 4890026 NÜRNBERG A5E03574579-AFⓅ 04/2019 本公司保留更改的权利Copyright © Siemens AG 2012 - 2019. 保留所有权利法律资讯警告提示系统为了您的人身安全以及避免财产损失，必须注意本手册中的提示。
人身安全的提示用一个警告三角表示，仅与财产损失有关的提示不带警告三角。
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SD214中文资料
High-Speed Analog N-Channel DMOS FETs SD210 / SD212 / SD214FEATURES•High Input to Output Isolation. . . . . . . . . . . . . . . . 120dB •Low On Resistance. . . . . . . . . . . . . . . . . . . . . . . . 30 Ohm •Low Feedthrough and Feedback Transients•Low Capacitance:–Input (Gate). . . . . . . . . . . . . . . . . . . . . . . . . . . 2.4pF typ.–Output. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 1.3pF typ.–Feedback. . . . . . . . . . . . . . . . . . . . . . . . . . . . 0.3pF typ.•No protection Diode from Gate to Substrate for Very High Impedance Applications•Maximum Gate Voltage. . . . . . . . . . . . . . . . . . . . . . . ±40VAPPLICATIONSSD210:•Analog Switch DriverSD212 and SD214:•Analog Switches•High-Speed Digital Switches•Multiplexers• A to D Converters• D to A Converters•Choppers•Sample & Hold DESCRIPTIONThe Calogic SD210 is a 30V analog switch driver without a built-in protection diode from gate to substrate for use with SD212 and SD214 DMOS analog switches.ORDERING INFORMATIONPart Package Temperature Range SD210E Hermetic TO-72 Package-55C to +125C XSS210Sorted Chips in Carriers-55o C to +125o C SD212DE Hermetic TO-72 Package-55o C to +125o C XSD212Sorted Chips in Carriers-55o C to +125o C SD214DE Hermetic TO-72 Package-55o C to +125o C XSD214Sorted Chips in Carriers-55o C to +125oCABSOLUTE MAXIMUM RATINGSDrain Current. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 50mA Total Device Dissipation at 25o C Case Temperature . . . 1.2W Storage T emperatue Range . . . . . . . . . . . . . -65o C to +200o C Lead Temperature (1/16" from case for 10 sec.). . . . . . 300o C Operating T emperature Range . . . . . . . . . . . -55o C to +125o CPARAMETER SD210SD212SD214UNIT V DS Drain-to-Source +30+10+20V dc V SD Source-to-Drain +10+10+20V dc V DB Drain-to-Body +30+15+25V dc V SB Source-to-Body +15+15+25V dc V GS Gate-to-Source ±40±40±40V dc V GB Gate-to-Body ±40±40±40V dc V GDGate-to-Drain±40±40±40V dcDC CHARACTERISTICS (T A = 25oC, unless otherwise specified)AC ELECTRICAL CHARACTERISTICSInformation furnished by Calogic is believed to be accurate and reliable. However, no responsibility is assumed for its use: nor for any infringement of patents or other rights of third parties which may result from its use. No license is granted by implication or otherwise under any patent rights of Calogic.。
DX200系统介绍
是MSC的交换网络，由M控制
PIU有SWCSM---Switch Control and Switching Memory
SWSPS---Switch Serial-Parallel-Serial Converter
PSC1------Power Supply
M——Marker(2N备份)
控制和监测GSW，选择空闲时隙，对所有呼叫进行连接和释放。M，GSW，DTMFC， TG是一个切换整体。
PROGRAMBLOCK
Alarm System Program Block (centr.) (ALARMP)
Alarm System Program Block (dist.) (DPALAR)
PROCESS/-FAMILY
ALARMP (OMU) ALARMP (CM)
DPALAR (OMU) DPALAR (CCSU-5) DPALAR (IWCU-0) DPALAR (CMU-1) DPALAR (CM-0) etc
18
2019/12/30
公共单元
OMU——Operation and Maintenance Unit（2N备份） OMU完成所有的监测，告警和恢复功能，也是用户的接口。在OMU的硬盘中是整
个系统的软件，也是所有告警存储的缓存区。 PIU为CPU-------中央处理单元
MBIF------MB接口板 SERO-----SERO-S能接4个异步串行接口。SERO-T能接8个异步串行接口。 速率为75~19200波特。 SCSIF-----包括2个SCSIF总线接口，和每个总线的DMA通道。数据传输速 率大概为3MB/s，速度决定于CPU的时钟。可以提供2个V.24的串行接口。 ASS-------预处理器（时钟控制） AAL------硬件告警单元
xsd讲义
XML Schema 简介XML Schema 是基于 XML 的 DTD 替代者。
XML Schema 可描述 XML 文档的结构。
XML Schema 语言也可作为 XSD（XML Schema Definition）来引用。
您应当具备的基础知识在继续学习之前，您需要对下面的知识有基本的了解： HTML / XHTML XML 以及 XML 命名空间 对 DTD 的基本了解如果您希望首先学习这些项目，请在 首页 访问这些教程。
什么是 XML Schema？XML Schema 的作用是定义 XML 文档的合法构建模块，类似 DTD。
XML Schema: 定义可出现在文档中的元素 定义可出现在文档中的属性 定义哪个元素是子元素 定义子元素的次序 定义子元素的数目 定义元素是否为空，或者是否可包含文本 定义元素和属性的数据类型 定义元素和属性的默认值以及固定值XML Schema 是 DTD 的继任者我们认为 XML Schema 很快会在大部分网络应用程序中取代 DTD。
理由如下： XML Schema 可针对未来的需求进行扩展 XML Schema 更完善，功能更强大 XML Schema 基于 XML 编写 XML Schema 支持数据类型 XML Schema 支持命名空间XML Schema 是 W3C 标准XML Schema 在 2001 年 5 月 2 日成为 W3C 标准。
您可以在我们的《W3C 教程》中获得更多有关 XML Schema 标准的信息。
为什么要使用 XML Schema？XML Schema 比 DTD 更强大。
XML Schema 支持数据类型XML Schema 最重要的能力之一就是对数据类型的支持。
通过对数据类型的支持： 可更容易地描述允许的文档内容 可更容易地验证数据的正确性 可更容易地与来自数据库的数据一并工作 可更容易地定义数据约束（data facets） 可更容易地定义数据模型（或称数据格式） 可更容易地在不同的数据类型间转换数据编者注：数据约束，或称 facets，是 XML Schema 原型中的一个术语，中文可译为“面”，用来约束数 据类型的容许值。
南大傲拓NA200系列可编程序控制器硬件手册说明书
基本说明�感谢您购买了南大傲拓NA200系列可编程序控制器。
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�电话：（+86）02525--68530188�传真：（+86）02525--68530178�网址：目录目录 (3)第1章NA200PLC概述 (5)1.1NA200PLC的硬件组成 (5)1.1.1CPU模块 (5)1.1.2CPU主要特性 (6)1.1.3扩展模块 (7)1.1.4CPU通用技术指标 (9)1.2工作方式介绍 (9)1.3编程软件介绍 (10)1.4快速应用指南 (11)1.4.1物品清点 (11)1.4.2设备安装接线 (12)1.4.3连接电源线 (12)1.4.4建立PC通讯 (12)1.4.5编写控制程序 (13)1.4.6设备投入运行 (13)第2章安装、拆卸与接线原则 (14)2.1指导原则 (14)2.1.1通风散热 (14)2.1.2功率消耗 (15)2.1.3合理布置电缆 (16)2.2电气安全 (16)2.3NA200小型一体化PLC及扩展模块的安装和拆卸 (16)2.3.1安装方式 (16)2.3.1.1板面安装 (17)2.3.1.2导轨安装 (18)2.3.1.3连接扩展电缆 (19)2.4NA200小型一体化PLC及扩展模块的尺寸结构 (20)第3章CPU模块 (22)3.1外观说明 (22)3.2CPU模块功能简述 (22)3.3使用说明 (23)3.3.1选择CPU的工作方式 (23)3.3.2指示灯定义 (23)3.4通讯功能 (24)3.5特殊I/O接线定义 (25)3.5.1I/O接线定义 (25)3.5.2中断号定义 (26)3.6高速计数输入 (27)3.6.1计数模式 (27)3.6.2计数值范围 (28)3.6.3CR寄存器定义 (28)3.714点CPU模块性能参数 (30)3.7.1性能参数 (30)3.7.2CPU201-1401端子定义与接线 (32)3.7.3CPU201-1402端子定义与接线 (33)3.820点CPU模块性能参数 (35)3.8.1性能参数 (35)3.8.2CPU201-2001端子定义与接线 (37)3.8.3CPU201-2002端子定义与接线 (39)3.924点CPU模块性能参数 (40)3.9.1性能参数 (40)3.9.2CPU201-2401端子定义与接线 (42)3.9.3CPU201-2402端子定义与接线 (44)3.1040点CPU模块性能参数 (45)3.10.1性能参数 (45)3.10.2CPU201-4001端子定义与接线 (47)3.10.3CPU201-4002端子定义与接线 (48)第4章I/O扩展模块 (51)4.1数字量扩展模块 (52)4.1.1数字量输入模块 (52)4.1.2数字量输出模块 (55)4.1.3数字量输入/输出模块 (59)4.2模拟量扩展模块 (61)4.2.1模拟量输入模块 (61)4.2.2温度量输入模块 (65)4.2.3模拟量输出模块 (67)4.2.4模拟量输入/输出模块 (72)4.3通讯扩展模块 (74)A附录订货参数 (78)B附录扩展模块功率 (80)第1章NA200PLC概述作为小型一体化PLC产品，NA200PLC无论是独立运行，还是相互连接构成网络，均可以实现强大而复杂的控制功能。
西门子S7-200系列PLC自学手册
西门子S7-200系列PLC自学手册 陈忠平周少华侯玉宝李锐敏编著人民邮电出版社北京图书在版编目（CIP）数据西门子S7-200系列PLC自学手册／陈忠平等编著．—北京：人民邮电出版社，2008.8ISBN 978-7-115-18141-1Ⅰ．西…Ⅱ．陈…Ⅲ．可编程序控制器—技术手册Ⅳ．TP332.3-62中国版本图书馆CIP数据核字（2008）第071204号内容提要本书从实际工程应用出发，以国内广泛使用的德国西门子S7-200系列PLC为对象，讲解PLC的基础与实际应用等方面的内容。
本书分为基础篇和实践篇。
其中基础篇以西门子公司的S7-200系列为例，介绍了PLC的结构配置、工作原理、指令系统、编程方法等内容；并在此基础上介绍了PLC控制系统的设计原则、设计步骤、硬件设计、软件设计等。
实践篇以工程实践为主线，通过实例和相关源程序介绍了PLC在电动机基本控制电路中的应用、利用PLC改造传统机床、PLC小系统的设计和PLC在工程中的设计与应用等内容。
本书语言通俗易懂，实例的实用性和针对性强。
本书既可作为电气控制领域技术人员的自学教材，也可作为高职高专院校、成人高校、本科院校的电气工程、自动化、机电一体化、计算机应用等专业的参考书。
西门子S7-200系列PLC自学手册♦编著陈忠平周少华侯玉宝李锐敏责任编辑张伟♦人民邮电出版社出版发行北京市崇文区夕照寺街14号邮编 100061 电子函件 315@网址 北京鑫正大印刷有限公司印刷♦开本：787×1092 1/16印张：20.75字数：515千字2008年8月第1版印数：1 – 4 000册2008年8月北京第1次印刷ISBN 978-7-115-18141-1/TN定价：36.00元读者服务热线：(010)67129258 印装质量热线：(010)67129223反盗版热线：(010)67171154前言随着科学技术的发展，电气控制技术在各领域，特别是在自动控制领域取得了长足的发展，有了越来越多的应用。
MC200-WEN用户速查手册说明书
MC200-WEN 用户速查手册感谢您使用MC200系列PLC以太网模块。
在使用PLC以太网模块产品前，请您仔细阅读本手册，以便更清楚地掌握产品特性，更安全地应用，充分利用本产品丰富的功能。
本速查手册用于MC200系列PLC以太网模块的设计、安装、连接和维护的快速指引，便于用户现场查阅所需信息,并有相关选配件的简介，常见问题答疑等，便于参考。
本手册适合MC200系列以下成员：MC200–WEN 以太网通讯模块版本号：2.3日期：2019-7-20若需要更详细的产品资料，可参考我公司发行的《MC 系列可编程控制器编程参考手册》MC200-WEN是一款包含以太网功能的通讯模块：◆10M/100M自动检测以太网协议；◆支持的通信协议：MODBUS、MODBUSTCP；◆使用MODBUS协议，可以远程下载用户程序和数据监控；◆使用MODBUSTCP协议，可以与人机界面等进行数据交换；◆最多支持2个客户端同时访问；◆支持虚拟串口工作方式,提供WINDOWS虚拟串口驱动,让用户串口设备无逢升级至以太网通讯方式,无需修改原有串口软件；◆系统块配置下载无需断电即可生效；1.外观以及部件名称2.安装说明采用DIN 槽安装固定在振动不大的环境下，可以采用35mm 宽度的DIN 槽进行安装。
打开模块底部的DIN 卡扣，将模块底部卡在DIN 导轨上。
旋转模块贴近DIN 导轨，合上DIN 卡扣。
仔细检查模块上DIN 卡扣与DIN 导轨是否紧密固定好，如下图：采用螺钉安装固定在振动较大的场合必须使用螺丝来固定，螺丝可选用M3，按照下图所示的尺寸进行定位、钻安装孔；用合适的螺钉将模块固定在背板上。
MC200系列的外形尺寸与安装孔位尺寸如下图所示。
图2-2螺钉安装示意图3.技术参数3.1环境指标◆无腐蚀、易燃易爆气体和液体的场所。
◆坚固无振动的场所。
◆本PLC 设计用于安装环境II 标准、污染等级2的应用场合。
工作温度-5-55℃工作湿度10%-90%RH （不凝结）存储温度-5-55℃存储湿度5%-90%RH3.2功能描述表3-1功能描述4.用户端子与指示灯表4-1MC200-WEN 指示灯功能说明5.接线方式MC200-WEN 有两个RJ45的网口，在侧面有丝印标签1．LAN ，用来进行web 设置，如果使用了系统块的设置，此端口不需使用。
SOM200SX-PC开发套件用户手册说明书
SOM200SX-PC Development KitWith 5S / 4USB/ LAN / 2GPIO128MB DDR2 OnboardUser’s Manual(Revision 1.0A)October, 2012Trademarks AcknowledgmentVortex86SX is the registered trademark of DMP Electronics Inc.Other brand names or product names appearing in this document are the properties and registered trademarks of their respective owners. All names mentioned herewith are reserved for identification purpose only.T a b l e o f C o n t e n t sT a b l e o f C o n t e n t s (iii)C h a p t e r 1 Introduction (1)1.1 Packing List (1)1.2 Product Description (2)1.3 Specifications (3)1.4 Board Dimension (5)C h a p t e r 2 Installation (8)2.1 Board Outline (8)2.2 Connectors & Jumpers Location (11)2.3 Connectors & Jumpers Summary (12)2.4 Pin Assignments & Jumper Settings (13)2.5 System Mapping (23)2.6 Watchdog Timer (28)2.7 GPIO (29)2.8 SPI flash (30)2.9 IDE to SD (31)C h a p t e r 3 Driver Installation (32)Appendix (33)A. TCP/IP library for DOS real mode (33)B. SOM200SX-PC & SOMSX-DEV-PC Schematic (34)C. BIOS Default Setting (35)Warranty (36)This page is blankC h a p t e r 1Introduction 1.1 Packing ListProduct Name PackageSOM200SX-DEV-PCRS232 cable (2.54mm) x 5USB (2.54mm) x 1Product Name PackageSOM200SX-PC1.2 Product DescriptionThe System on Module is a core module with the processor, memory and I/O that would contain the following benefits in the respect of system design.300MHz Vortex86SX System-On-Chip 128 / 256MB DDR2 system memory4 USB Ver. 2.0 (host)5 serial ports16-bit GPIO x2PCI bus2 watchdog timer Enhanced IDE (UltraDMA-100/66/33) JTAG interfaceAMI BIOS2MB SPI flashSingle voltage +5V DCSupport extended operatingtemperature range of -20°C to +70°SOM200SX-PC is suitable for broad range of data-acquisition, Industrial automation, Process control, Automotive controller, AVL, Intelligent Vehicle management device,Medical device, Human machine interface, Robotics and machinery control.SOM200SX-PC measured at only 52mm (L)*52mm (W)*10.5mm (H), is designed particularly as the kernel for the diverse expandable applications. Through 8 rows of25pin connector, SOM200SX-PC is able to provide multiple functions, such as RS-232, IDE, LAN, USB and GPIO.To assist users easily adapt SOM200SX-PC Module into their applications, ICOP offers a complete development board and referential circuit diagram for SOM200SX-PC Module in order to reduce users’ time.Please visit the website below for furtherinformation /tech/vortex86sx/As to the referential circuit drawing, please contact *************.tw1.3 SpecificationsSOM200SX-DEV-PCFeatures SOM200SX-DEV-PC Bus Interface PCI Bus standard compliantConnectors 2.54mm 20-pin header for GPIO x22.54mm 10-pin header for RS-232 x52.0mm 44-pin header for IDE x1External RJ-45 connector for Ethernet x1External 6-pin Mini DIN connector for Keyboard x1SOM200SX-PCFeatures SOM200SX-PCCPU DM&P SoC CPU Vortex86SX- 300MHzReal Time Clock with Lithium Battery BackupCache L1:16K I-Cache, 16K D-CacheBIOS AMI BIOSBus Interface PCI bus standard compliantSystem Memory 128 / 256MB DDR2 onboardWatchdog Timer Software programmable from 30.5 us to 512 seconds x2sets(Watchdog 1 fully compatible with M6117D)LAN Integrated 10/100Mbps EthernetFlash Disk Support Onboard 2MB SPI Flash DiskOnboard SST Flash Disk (512MB/1GB/2GB/4GB areoptional)MSTI EmbedDisk Module (16MB and above)Serial Console Controlled by GP 36/37I /O Interface Enhanced IDE port x1RS-232 port x51.25mm 6-pin Wafer for JTAG x1Power Requirement Single Voltage +5V @ 340mADimension 52 mm (L) x 52 mm (W) x 10.5 mm (H) (With Cover) Weight 15gOperating Temperature -20o C ~ +70o C-40°C ~ +85°C (Optional)1.4 Board DimensionS OM200SX-DEV-PCSOM200SX-PCC h a p t e r 2C h a p t e r 2 Installation2.1 Board OutlineSOM200SX-DEV-PCConsoleRedirectionGPIO 0, 1GPIO 2, 3ATX Power InputPWR -SW PS/2 KB/MouseLPC COM9EIDE InterfaceIDE/SD PowerConnector COM3COM4PCI SlotResetCOM1COM2COM1LAN USB COM2USB 0, 1S OM200SX-DEV-PC with PCI-VGA-Z9sPS/2 Mouse PS/2 KBDCOM1USBCOM2LANPCI-VGA-Z9sBoardDVIVGASOM200SX-PCJTAG2.2 Connectors & Jumpers LocationConnectorsSOM200SX-DEV-PCJ26J11J12J16SW1J7J9J45J13USB1J24J23J20J17J2J38J1J18J10J14J3J37SW22.3 Connectors & Jumpers SummarySOM200SX-DEV-PCNbr Description Type of Connections Pin nbrs.J1 IDE Connector Box Header, 2.0∅, 22x2 44-pinJ2 IDE Connector Box Header, 2.54∅, 20x2 40-pinJ3 USB 2/ USB 3 Box Header, 2.54∅, 5x2 10-pinUSB1A USB 0/ USB 1 USB connector8-pinUSB1B Ethernet LAN RJ45 Connector 8-pinJ7A PS/2 Keyboard Mini-DIN Female6-pinJ7B PS/2 Mouse Mini-DIN Female6-pinJ9 TTL/RS232 Mode Selector Pin Header, 2,54∅, 1x2 2-pinJ10 COM1/P4/PWM Box Header, 2.54∅, 5x2 10-pinJ11 GPIO ( P0 / P1 /PWM) Box Header, 2.54∅,10x220-pinJ12 GPIO (P2/P3/ SPI/2C/PWM) Box Header, 2.54∅, 10x220-pinJ13 TTL/RS232 Mode Selector Pin Header, 2,54∅, 1x2 2-pinJ14 COM2 Box Header, 2.54∅, 5x2 10-pinJ16 ATX Power ATX header 20-pinJ17 Power Connector Terminal Block 5.0∅, 2x1 2-pinJ18 COM3 TTL Mode Box Header, 2.54∅, 5x2 10-pinJ20 COM4 TTL Mode Box Header, 2.54∅, 5x2 10-pinJ21 ISA Slot ISA Slot 98-pinJ23 PCI Slot1 PCI Slot 120-pinJ24 PCI Slot2 PCI Slot 120-pinJ26 Console RedirectionJ35A PC104 Connector – 64 pin Box Header, 2.54∅, 32x2 64-pinJ35B PC104 Connector – 40 pin Box Header, 2.54∅, 20x2 40-pinJ37 LPC Box Header, 2.0∅, 5x2 10-pinJ38 COM9 Box Header, 2.54∅, 5x2 10-pinJ45A COM1 D-Sub Male 9-pinJ45B COM2 D-Sub Male 9-pinSP1 BUZZERSW1 PWR-SWSW2 Reset2.4 Pin Assignments & Jumper Settings SOM200SX-DEV-PCJ1: IDE (44 Pins)Pin # Signal Name Pin # Signal Name1 IDERST2 GND3 IDED74 IDED85 IDED6 6 IDED97 IDED5 8 IDED109 IDED4 10 IDED1111 IDED3 12 IDED1213 IDED2 14 IDED1315 IDED1 16 IDED1417 IDED0 18 IDED1519 GND 20 NC21 IDEREQ 22 GND23 IDEIOW 24 GND25 IDEIOR 26 GND27 ICHRDY 28 GND29 IDEACK 30 GND31 IDEINT 32 NC33 IDESA1 34 IDECBLID35 IDESA0 36 IDESA237 IDECS-0 38 IDECS139 IDELED 40 GND41 VCC 42 VCC43 GND 44 NCJ2: IDE (40 Pins)Pin # Signal Name Pin # Signal Name1 IDERST2 GND3 IDED74 IDED85 IDED6 6 IDED97 IDED5 8 IDED109 IDED4 10 IDED1111 IDED3 12 IDED1213 IDED2 14 IDED1315 IDED1 16 IDED1417 IDED0 18 IDED1519 GND 20 VCC21 IDEREQ 22 GND23 IDEIOW 24 GND25 IDEIOR 26 GND27 ICHRDY 28 GND29 IDEACK 30 GND31 IDEINT 32 NC33 IDESA1 34 IDECBLID35 IDESA0 36 IDESA237 IDECS0 38 IDECS139 IDELED 40 GNDJ3: USBPin # Signal Name Pin # Signal Name1 VCC2 VCC3 LUSBD0-4 LUSBD1-5 LUSBD0+6 LUSBD1+7 GND 8 GND9 GGND 10 GGNDUSB1A: USB0/1Pin # Signal Name Pin # Signal Name1 VCC2 VCC3 -DATA4 -DATA5 +DATA6 +DATA7 GND 8 GNDUSB1B: Ethernet LANPin # Signal Name Pin # Signal Name1 TD+2 TD-3 RO+4 NC5 NC6 RO-7 NC 8 NCJ7A: PS/2 KeyboardPin # Signal Name Pin # Signal Name1 KBDATA2 NC3 GND4 VCC5 KBCLK6 RO-J7B: PS/2 MousePin # Signal Name Pin # Signal Name1 TD+2 TD-3 RO+4 NC5 NC6 RO-J9: TTL/RS232 Mode Selector (Open: On, Close: Off) Pin # Signal Name Pin # Signal Name1 GND2 VCCJ10: COM1/P4/Pin # SignalNamePin # Signal Name1 DCD12 RXD13 TXD14 DTR15 GND6 DSR17 RTS1 8 CTS19 RI1 10 TXDEN1/VCCJ11: GPIO (P0/ P1)Pin # Signal Name Pin # Signal Name1 GND2 VCC3 GP004 GP105 GP016 GP117 GP02 8 GP129 GP03 10 GP1311 GP04 12 GP1413 GP05 14 GP1515 GP06 16 GP1617 GP07 18 GP1719 VCC 20 GNDJ12: GPIO (P2/ P3/ SPI)Pin # Signal Name Pin # Signal Name1 GND2 VCC3 GP204 SPICS5 GP216 SPICLK7 GP22 8 SPIDO9 GP23 10 SPIDI11 GP24 12 GP3413 GP25 14 GP3515 GP26 16 GP3617 GP27 18 GP3719 VCC 20 GNDNote:If you Enable 2M SPI flash Disk on the BIOS setting, you cannot use GP30~GP37 Pins.J13: TTL/RS232 Mode Selector (Open: On, Close: Off)Pin # Signal Name Pin # Signal Name1 GND2 VCCJ14: COM2/P4/PWM Pin # SignalName Pin # Signal Name1 DCD2 2 RXD23 TXD24 DTR25 GND6 DSR27 RTS2 8 CTS29 RI2 10 TXDEN2/VCCJ16: ATX PowerJ17: Power Connector (Terminal Block 5.0mm)Pin # Signal Name1 +5V2 GNDJ18: COM3 TTL Mode Pin # SignalName Pin # SignalName1 DCD32 RXD33 TXD34 DTR35 GND6 DSR37 RTS3 8 CTS39 RI3 10 VCC Pin # Signal Name Pin # Signal Name1 3V32 3V3 3 GND4 VCC5 GND6 VCC7 GND8 NC9 SB5V 10 +12V 11 3V3 12 -12V 13 GND 14 SB5V15 GND 16 GND 17 GND 18 -5V 19 VCC 20 VCCJ20: COM 4 TTL ModePin # SignalNamePin #SignalName1 DCD42 RXD43 TXD4 4 DTR45 GND6 DSR47 RTS4 8 CTS49 RI4 10 VCCJ37: LPCPin # Signal Name Pin # Signal Name1 24MHZ2 LAD03 LFRAME-4 LAD15 GND6 LAD27 LDRQ- 8 LAD39 SERIRQ 10 VCCJ38: COM9Pin # Signal Name Pin # Signal Name1 NC2 RXD93 TXD94 NC5 GND6 NC7 NC 8 NC9 NC 10 VCCJ45A: COM 1Pin # SignalNamePin #SignalName1 DCD12 RXD13 TXD14 DTR15 GND6 DSR17 RTS18 CTS19 RI1J45B: COM 2Pin # SignalNamePin #SignalName1 DCD2 2 RXD23 TXD24 DTR25 GND6 DSR27 RTS28 CTS29 RI2SOM200SX-PCSOM200SX-PCJ1/J2/J3/J4:SOM200SX-PC Signal Assignment2.5 System MappingMemory MappingAddress Description Usage 00000000 - 9000FFFF System RAM *A0000000 - A000FFFF EGA/VGA Video MemoryB0000000 - B0007FFF MDA RAM, Hercules graphics display RAMB0008000 - B000FFFF CGA display RAMC0000000 - C0007FFF EGA/VGA BIOS ROMC0008000 - C000BFFF Boot ROM enableC000C000 - C000FFFF Console Redirection enableD0000000 - D700FFFF Expansion ROM spaceD8000000 - DB00FFFF SPI FLASH Emulation Floppy A EnableDC000000 - DF00FFFF Expansion ROM SpaceE0000000 - E000FFFF USB Legacy SCSI ROM spaceF0000000 - F000FFFF Motherboard BIOS * FEFAD000 - FFFFF000 Standard OpenHCD USB Host Controller * FEFAF800 - FFFFFF00 Standard OpenHCD USB Host Controller * FEFAF400 - FFFFFF00 On board Ethernet Adapter * FEFAE000 - FFFFF000 Standard Enhanced PCI to USB Host Controller * FEFAFC00 - FFFFFF00 Standard Enhanced PCI to USB Host Controller *I/O MappingI/O Address Owner Usage 0000h - 000Fh DMA 8237-1 * 0010h - 0017h COM 90020h - 0021h PIC 8259-1 *0022h - 0023h Indirect Access Registers (6117D configuration port)*002Eh - 002Fh Forward to LPC BUS0040h - 0043h Timer Counter 8254 * 0048h - 004Bh PWM counter 8254 * 004Eh - 004Fh Forward to LPC BUS0060h Keyboard / Mouse data port * 0061h Port B + NMI control port * 0062h - 0063h 8051 download 4k address counter * 0064h Keyboard/ Mouse status/ command port * 0065h WatchDog0 reload counter * 0066h 8051 download 8bit data port * 0067h WatchDog1 reload counter * 0068h - 006Dh WatchDog1 control counter * 0070h - 0071h CMOS RAM port * 0072h - 0075h MTBF control register * 0078h - 007Ch GPIO port 0,1,2,3,4 default setup * 0080h - 008Fh DMA page register * 0092h System control register * 0098h - 009Ch GPIO direction control *00A0h - 00A1h PIC 8259-2 * 00C0h - 00DFh DMA 8237-2 * 00E0h – 00EFh DOS 4G Page access * 0170h – 0177h IDE1(IRQ 15)01F0h – 01F7h IDE0 (IRQ 14) * 0220h – 0227h COM8 Forward to LPC BUS0228h – 022Fh COM7 Forward to LPC BUS0238h – 023Fh COM6 Forward to LPC BUS0278h – 027Fh Printer port (IRQ7, DMA 0) * 02E8h – 02EFh COM4 (IRQ 11) * 02F8h – 02EFh COM2 (IRQ3) * 0338h – 033Fh COM5 Forward to LPC BUS0376h IDE1 ATAPI device control write only register * 03E8h – 03EFh COM3 (IRQ 10) * 03F0h – 03F7h Floppy Disk (IRQ6, DMA2)03F6h IDE0 ATAPI device control write only register * 03F8h – 03FFh COM1 (IRQ 4) * 0480h – 048Fh DMA High page register * 0490h – 0499h Instruction counter register * 04D0h – 04D1h 8259 Edge / level control register * 0CF8h – 0CFFh PCI configuration port * DE00h – DEFFh On board LAN * FC00h – FC05h SPI Flash BIOS control register * FC08h – FC0Dh External SPI BUS control register *IRQ MappingIRQ# Description Usage IRQ0 System Timer * IRQ1 Keyboard Controller * IRQ2 Cascade for IRQ8 – 15IRQ3 Serial Port 2 * IRQ4 Serial Port 1 * IRQ5 USB * IRQ6 USB * IRQ7 Printer Port * IRQ8 Real Time Clock * IRQ9 USB/ Ethernet 10/100M LAN * IRQ10 Serial Port 3 * IRQ11 Serial Port 4 * IRQ12 Mouse * IRQ13 Math Coprocessor * IRQ14 Hard Disk Controller#1 * IRQ15 Hard Disk Controller#2 *DMA MappingDMA# Description Usage DMA0DMA1DMA2 Floppy Disk ControllerDMA3DMA4DMA5DMA6DMA72.6 Watchdog TimerThere are two watchdog timers in Vortex86SX/DX CPU. One is compatible with M6117D watchdog timer and the other is new. The M6117D compatible watchdog timer is called WDT0 and the new one is called WDT1.We also provide DOS, Linux and WinCE example for your reference. For more technical support, please visit: /tech or download the PDF file:/tech/vortex86sx/2.7 GPIO (General Purpose Input / Output)40 GPIO pins are provided by the Vortex86SX/DX for general usage in the system. All GPIO pins are independent and can be configured as inputs or outputs, with or without pull-up/pull-down resistors.We also offer DOS, Linux and WinCE example for your reference. For more technical support, please visit: /tech or download the PDF file:/tech/vortex86sx/2.8 SPI flash (Serial Peripheral Interface)As SPI Flash (Serial Peripheral Interface) offers many benefits including: reduced controller pin count, smaller and simpler PCBs, reduced switching noise, less power consumption, and lower system costMany of users may consider using a formatted SPI flash to boot for the system or emulate SPI flash as Floppy (A: Driver or B: Driver). Then you must know how to set for this condition in CMOS Setup and boot up under DOS 6.22, X-DOS, DR-DOS and Free DOS.For more technical support, please visit: /tech or download the PDF file: /tech/vortex86sx/2.9 IDE to SD (Micro-SD)Vortex86SX SoC also built-in simulation circuit to adapt SD to IDE in order to allow your system to recognize Micro-SD card as C: or D: DriverSD-1917: 44 pins IDE to SD Adapter is an ideal solution for industrial PC or embedded system and 44 pins IDE to SD Adapter can be easily installed on all Vortex86SX CPU boards. You or your customers just do the BIOS setting and use SD-1917 to connect IDE connector of Vortex86SX directly.For further inquiries of SD-1917, please contact ICOP sales team or mail to: *************.tw for your request.<BIOS setting>Get into the BIOS setup UtilityChoose Primary IDE Pin Select: SD cardPress “F10” to Save configuration changes and exit setupSD-1917SD-1917: /pddetail.aspx?id=125&pid=4C h a p t e r 33C h a p t e rDriver InstallationVGAThe Vortex86SX processor also uses external Display Card ““Volari™ Z9s” which is an ultra low powered graphics chipset with total power consumption at around 1-1.5 W. It is capable in providing VGA display output upto 1600x1200. With DVO interface, developers could easily connect flat Panel to support TFT and LVDS output.Please download the Driver: /sd/sd_download.aspLANThe Vortex86SX processor also integrated 10/100Mbps Ethernet controller that supports both 10/100BASE-T and allows direct connection to your 10/100Mbps Ethernet based Local Area Network for full interaction with local servers, wide area networks such as the Internet.The controller supports: Half / Full-Duplex Ethernet function to double channel bandwidth, auto media detection.Operating system supportThe SOM200SX-PC CPU module provides LAN drivers for DOS 6.22 Windows CE 5.0, CE 6.0, Windows 98, Windows XP Professional, Windows Embedded standard (XPE) and Windows 2000 (SP4).Please get the drivers from the Driver CD which attached with the standard packing ofSOM200SX-PC CPU module or please get it from DMP official website:/tech/vortex86sx/SOM200SX-PC CPU module also supports most of the popular Linux distributions, for more detail information, please visit DMP official website: /tech/vortex86sx/AppendixA. TCP/IP library for DOS real modeDSock is a TCP/IP library for DOS real mode, which is used by RSIP. It provides simple C functions for programmer to write Internet applications. ICOP also provide Internet examples using DSock: BOOTP/DHCP, FTP server, SMTP client/server, HTTP server, TELNET server, Talk client/server, etc.DSock provides a lot of example source code. Programmer can add Internet functions to their project easily and save development time. With a utility "MakeROM”, programmer also can make a ROM image to fit their application, those examples can be seen in the following Application systems: Mity-Mite Serial Server,Web Camera Tiny Server and RSIP Serial Server.DSock is free for All ICOP products using M6117D/Vortex86/Vortex86SX/Vortex86DX CPU and ICOP also provide the business version of DSock for those customers who are using other x86 CPUs.If you would like to use DSock or business version of DSock, Please mail to *************.tw or contact your regional sales.Please download the trial DSock software and Utilities from our website:/tech/dmp-lib/dsock/B. SOM200SX-PC & SOM200SX-DEV-PC SchematicSchematic information can help baseboard designer to optimize exactly how each of these functions implements physically. Designer can place connectors precisely where needed for the application on a baseboard designed to optimally fit a system’s packaging.Please contact or e-mail our regional sales to get SOM200SX-PC CPU module andSOM200SX-DEV-PC Schematic.C. BIOS Default settingIf the system cannot be booted after BIOS changes are made, Please follow below procedures in order to restore the CMOS as default setting.Press “End” Key, when the power onPress <Del> to enter the AMI BIOS setupPress “F9” to Load Optimized DefaultsPress “F10” to Save configuration changes and exit setupWarrantyThis product is warranted to be in good working order for a period of one year from the date of purchase. Should this product fail to be in good working order at any time during this period, we will, at our option, replace or repair it at no additional charge except as set forth in the following terms. This warranty does not apply to products damaged by misuse, modifications, accident or disaster. Vendor assumes no liability for any damages, lost profits, lost savings or any other incidental or consequential damage resulting from the use, misuse of, originality to use this product. Vendor will not be liable for any claim made by any other related party. Return authorization must be obtained from the vendor before returned merchandise will be accepted. Authorization can be obtained by calling or faxing the vendor and requesting a Return Merchandise Authorization (RMA) number. Returned goods should always be accompanied by a clear problem description.。
聚丙烯酰胺细粉再生PLC控制系统改造
聚丙烯酰胺细粉再生PLC控制系统改造摘要：聚丙烯酰胺细粉再生部分，原控制系统为独立的西门子PLC S200，与控制室之间无通讯，不能有效监控，曾多次因设备问题造成堵料、跑料，且控制程序厂家不提供，每次需拆除整个控制盘由厂家进行维护，对生产造成很大影响。
针对此问题，将现场PLC程序进行解析。
关键词：PLC；梯形图；逻辑1、流程简介由生产线来经干燥器干燥后的物料经双层筛Z5000筛出的的细粉进入细粉收集料斗H5050，细粉出料螺杆电机S5050将细粉排出，在废弃风机F6040的作用下在细粉料斗H5020内部形成真空，将由细粉出料电机S5050排出的细粉吸入H5020中，细分下料旋转阀S5020将细粉排入细粉再生料斗。
细粉再生料斗上安装有上下两个阻旋式料位开关，料斗侧壁安装有一台振荡器，下部出口处为螺杆式出料电机。
当由下料旋转阀S5020进入细粉再生料斗的物料达到料斗上部的料位开关处，料位开关产生料位高报报警，此时螺杆式出料电机启动，震荡器启动，注水阀XV501打开向出料螺杆注脱盐水，脱盐水与粉料在螺杆的搅拌下均匀混合后，进入预研磨料仓，达到细粉回收再生的目的，生产流程如图1。
图1 细粉再生流程图2、存在的问题及隐患2.1 现场设备故障本系统投入生产以来，在最初投运时运行良好，但随着运行时间的增长，安全隐患也逐渐显露出来。
由于现场控制器与操作室之间无任何系统通讯，现场也无监控系统，操作人员无法及时了解设备运行情况，使生产无法受控。
现场设备发生故障，会造成PLC程序无法正常运行。
如现场料位高报开关故障，由于PLC接收不到报警信号，程序将进入等待状态，仅需要10分钟左右细粉粉料就会从料斗溢出，造成生产波动，影响产品质量，而工艺操作人员巡检间隔是每两小时一次，在巡检间隔中不能及时发现将造成较大损失。
2.2 PLC本身的问题近几年发生多起PLC程序停止运行情况，维护人员对现场设备进行反复检查没有发现问题，需要对PLC内部程序运行情况进行检查。
SD200驱动器用户手册
5、安装注意事项 安装电气控制柜时，防止粉尘或铁屑进入伺服驱动器内部。
图 2.2 伺服驱动器安装图
SD 200 驱动器使用说明书 第5页
2.3 标准连接
2.3.1 位置控制接线图
三相 AC220V 或单相
特 速度波动率 性
调速比 脉冲频率 控制输入 控制输出
位置控制
速度控制 加减速功能 监视功能
保护功能 适用负载惯量
<±0.03(负载 0～100％)；<±0.02（电源－15～＋10％）（数值对应于额 定速度）
1∶ 5000
≤500kHz
①伺服使能 ②报警清除 ③CCW 驱动禁止 ⑤偏差计数器清零/零速箝位 ⑥指令脉冲禁止 ⑦CCW 转矩限制 ⑧CW 转矩限制
NFB
DC 12～24 伺服使能 报警清除
CCW（逆时针）驱动禁止 CW(顺时针)驱动禁止 偏差计数器清零 指令脉冲禁止
伺服准备好 伺服报警 定位完成
机械制动器释放
模拟速度指令输入
位置指令 PULS
位置指令 SIGN
FG
R S T MC r t
伺服驱动器
U V W FG
14 5V 15 5V 16 5V
SD 200 驱动器使用说明书 第4页
2.2 安装方法
1、安装方向 伺服驱动器应垂直直立安装。
2、安装固定 安装时，上紧伺服驱动器后部的 4 个 M5 固定螺丝。
3、安装间隔 伺服驱动器之间以及与其它设备间的安装间隔距离，请参考图 2.2， 实际中应尽可能地 留有较大的安装间隔，保证散热。
DRIVER SERIES KND-SD200 系列
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BODY (4) SOT-143 SOURCE (1)
SSTSD201 SSTSD203
元器件交易网
SD200 / SD201 / SD202 / ห้องสมุดไป่ตู้D203 / SSTSD201 / SSTSD203
CORPORATION
ABSOLUTE MAXIMUM RATING (TA = +25oC unless otherwise noted) PARAMETER Breakdown Voltages VDS VDB VGS VGB VGD SD200 SD201 SD202 SD203 UNIT ID PT PD Tj Ts Continuous Drain Current . . . . . . . . . . . . . . . . . . 50 mA Power Dissipation (at or below TC = +25oC) . . . . 1.8 W Linear Derating Factor . . . . . . . . . . . . . . . . . 18 mW/ oC Power Dissipation (at or below TA = +25C) . . . 360 mW Linear Derating Factor . . . . . . . . . . . . . . . . . 3.6 mW/ oC Operating Junction Temperature Range . . . . . . . . . . . . . . -55oC to + 125oC Storage Temperature Range . . . . . . . . -65oC to +175oC
APPLICATIONS
• High Gain VHF/UHF Amplifiers • Oscillators • Mixers
PIN CONFIGURATION
(2) DRAIN (4) CASE, BODY
CD10-1 CD10-2
G D CASE, B S
SD201, SD203, zener protected SD202, SD204, non-zener
(3) GATE
(1) SOURCE
GATE (3) DRAIN (2)
PART MARKINGS (SOT-143) P/N MARKING 201 203
BODY INTERNALLY CONNECTED TO CASE. DIODE PROTECTION ON SD201/SD203 ONLY.
ID(OFF) IGBS
µA
(SD202, SD203, SSTSD203) Low Interelectrode Capacitances
DESCRIPTION The SD200 series is manufactured utilizing Calogic’s proprietary DMOS design and processing techniques. The device is designed to operate well through 1 GHz while maintaining excellent frequency response, power gain, and low noise. The DMOS structure is an inherently low capacitance and very high speed design resulting in a device that bridges JFETS and GaAs products in performance characteristics. ORDERING INFORMATION Part SD200DC SD201DC SD202DC SD203DC SSTSD201 SSTSD203 XSD200 XSD201 XSD202 XSD203 Package 4 Lead TO-52 Package 4 Lead TO-52 Package 4 Lead TO-52 Package 4 Lead TO-52 Package Surface Mount SOT-143 Surface Mount SOT-143 Sorted Chips in Carriers Sorted Chips in Carriers Sorted Chips in Carriers Sorted Chips in Carriers SCHEMATIC DIAGRAM Temperature Range -55oC to +125oC -55oC to +125oC -55oC to +125oC -55oC to +125oC -55oC to +125oC -55oC to +125oC -55oC to +125oC -55oC to +125oC -55oC to +125oC -55oC to +125oC
200, 201 SYMBOL PARAMETER MIN STATIC BVDS BVDB Drain-Source Breakdown Voltage Drain-Body Breakdown Voltage Drain-Source OFF Current SD200 Gate-Body Leakage Current SD202 SD201 SD203 VGS(th) rDS(ON) DYNAMIC gfs ciss coss crss Gps NF Pi Common-Source Forward Transcondconductance Common-Source Input Capacitance Common-Source Output Capacitance Common-Source Reverse Transfer Capacitance Common-Source Power Gain Noise Figure Intercept Point 8.0 13 14 2.4 1.0 0.2 10 4.5 29 6.0 3.0 1.2 0.3 8.0 17 20 3.0 1.0 0.2 10 dB 4.0 29 5.0 dBm ∆f = 2 MHz 3.6 1.2 0.3 VDS = 15 V f = 1 GHz ID = 20 mA VSB = 0 pF VGS = 0 mS ID = 20 mA, VDS = 15 V f = 1 KHz, VSB = 0 ID = 20 mA VDS = 15 V f = 1 MHz VSB = 0 Gate Threshold Voltage Drain-Source ON Resistance 0.1 1.0 40 2.0 70 0.1 1.0 35 1.0 1.0 2.0 50 V ohms VDS = VGS, ID = 1µA, VSB = 0 VGS = 5 V, ID = 1 mA, VSB = 0 25 25 1.0 1.0 ±0.1 ±0.1 µA nA 30 20 20 25 V V ID = 1.0µA, VGS = VBS = 0 ID = 1.0µA, VGB = 0 Source OPEN VDS = 25 V VGS = VBS = 0 VDS = 20 V VGV = ±40 V VDB = VSB = 0 VGB = 20 V TYP MAX MIN TYP MAX 202, 203 UNIT TEST CONDITIONS
元器件交易网
CORPORATION
High-Speed Analog N-Channel Enhancement-Mode DMOS FETS
SD200 / SD201 / SD202 / SD203 / SSTSD201 / SSTSD203
FEATURES
• High gain . . . . . . . . . . . . . . . . . . . . . 8.0 dB min @ 1 GHz • Low Noise. . . . . . . . . . . . . . . . . . . . . 5.0 dB max @ 1 GHz
+25 +25 ±40 ±40 ±40
+25 +25 -0.3 +20 -0.3 +20 -0.3 +20
+20 +20 ±40 ±40 ±40
+20 +20 -0.3 +20 -0.3 +20 -0.3 +20
V V V V V V V V
ELECTRICAL CHARACTERISTICS (TA = +25oC unless otherwise noted)