ER23

合集下载

BF6910(BF6911)ASXX规格书

BF6910(BF6911)ASXX规格书
8.1 上电、掉电复位 ...................................................................................................................................... 20 8.2 FLASH 编程复位 ....................................................................................................................................... 20 8.3 软件复位 .................................................................................................................................................. 21 8.4 看门狗定时器溢出复位 .......................................................................................................................... 21 8.5 PC 指针溢出复位 ..................................................................................................................................... 21
3.1 引脚图 ....................................................................................................................................................... 6 3.2 引脚描述 ................................................................................................................................................... 6

部编版小学一年级语文上册汉语拼音总复习+知识点汇总

部编版小学一年级语文上册汉语拼音总复习+知识点汇总

部编版小学一年级语文上册汉语拼音总复习+知识点汇总人教部编版一年级语文上册汉语拼音总复习资料一、熟练背诵下面三个表:1、声母(23个)b p m f d t n lg k h j q xzh ch sh r z c sy w注意:zh ch sh r是翘舌音z c s是平舌音2、韵母(24个)单韵母6个a o e i uü复韵母9个ai ei ui ao ou iu ie üe er前鼻韵母5个an en in un ün后鼻韵母4个ang eng ing ong3、整体认读音节(不能拼读,只能直呼)(16个)zhi chi shi ri zi ci siyi wu yu ye yue yuanyin yun ying四、给韵母加声调的儿歌。

有a找a,没a找o e,i u并排,标在后。

拼音部分:音节词拼读内容一年级拼音练习(一)y wyǔ yīwū yālǐ yúmǎ yǐyù mǐyā zi bù wá wɑyá chǐjī wōā yízhī bùxǐ yī fuwū zi yī fu yǐ zi dà wùxià yǔyā líwà zi zhuō zizhuō yǐyā zhe mù wū。

1、nà shì yì bǎ yǐ zi。

2、ā yí在xǐ wà zimǐlì。

3、小鸟在zhuó yùbà bɑmā mɑbó bo pó po pá pōpù bùdà fómā bùdà mǐmù fámù mǎbǐ mòmó mòfā bù山pō。

er302二极管参数代换

er302二极管参数代换

er302二极管参数代换1.引言1.1 概述概述ER302二极管是一种常见的电子元器件,广泛应用于电子电路中。

二极管参数代换是一种常用的方法,用于简化二极管的具体参数,并将其代换为等效的理想二极管模型。

这个方法的目的是简化电路分析和计算,提高电路设计的效率和准确性。

本文将会详细介绍二极管ER302的参数,包括它的结构、特性和性能指标。

然后,我们将深入解析二极管参数代换的原理和方法。

通过了解二极管的特性和原理,我们能够更好地理解二极管参数代换的必要性和应用场景。

在正文部分,我们将会介绍二极管ER302的具体参数,包括正向导通电压、反向截止电压、最大正向电流等。

我们还会深入探讨这些参数对电路行为的影响和作用。

随后,我们将详细解析二极管参数代换的原理。

通过代换等效模型,我们能够简化电路的计算和分析。

我们还会介绍常用的二极管等效模型,如常见的理想二极管模型。

结论部分,我们将阐述二极管参数代换的应用场景。

这种方法能够简化电路设计和分析的工作量,提高设计的效率和准确性。

我们还会总结二极管参数代换的优缺点,并对未来的发展进行展望。

通过本文的阅读,读者将能够全面了解二极管ER302的参数和二极管参数代换的原理。

同时,读者也能够掌握该方法的应用技巧,提高电路设计和分析的能力。

希望本文能为读者在电子领域的学习和实践中提供有益的指导和帮助。

1.2文章结构文章结构:本文分为引言、正文和结论三个部分。

引言部分包括概述、文章结构和目的三个小节。

概述部分将介绍本文所要讨论的主题——er302二极管参数代换,并简要说明该主题的重要性。

文章结构部分将对本文的组织结构进行说明,让读者清晰了解全文的内容和安排。

目的部分将明确本文的目标,即通过对er302二极管参数代换的研究,深入探讨其应用和意义。

正文部分包括二极管ER302的参数和二极管参数代换的原理两个小节。

在二极管ER302的参数部分,将详细介绍er302二极管的主要参数,如电压、电流和频率等,并解释这些参数的物理意义和测量方法。

小麦抗白粉病基因

小麦抗白粉病基因

西北植物学报2003,23(5):822—829Acta Bot.Boreal.-Occident.Sin.文章编号:1000-4025(2003)05-0822-08小麦抗白粉病基因解超杰,杨作民,孙其信*(中国农业大学农学与生物技术学院,北京100094)摘 要:到目前为止,小麦中已经鉴定出31个主效抗白粉病基因位点(Pm1-P m31),对这些小麦抗白粉病基因位点的来源、染色体定位、遗传特点以及载体品种等方面进行了概括性综述.关键词:小麦;白粉病;抗病基因;染色体定位中图分类号:Q945.8;S512.1 文献标识码:AResistance genes to powdery mildew in wheatXIE Chao-jie,YANG Zuo-m in,SU N Qi-x in*(College of Agronomy and Bio-technology,China Agricultural U nivers ity,Beijing100094,C hina)Abstract:Pow dery mildew is o ne o f the m ost serious w heat diseases in China.Breeding fo r resistant culti-vars has pro ved the effective and an env ir onm entally safe w ay to co ntrol this disease.Up to now,31m ajor w heat pow dery m ildew r esistance g enes(P m1—Pm31)had been repor ted.This paper briefly review ed the so urces,chrom oso mal location,g enetics,and the carr ying cultivars o f these P m genes.Key words:wheat;pow dery mildew;resistance g enes;chromo som al locatio n 小麦白粉病是由小麦白粉菌Blumeria grami-nis f.sp.tritici(=Ery sip he gr aminis f.sp.tritici)引起的一种气传病害,是威胁我国小麦生产的重要常见病害之一[1].培育抗病品种是防治小麦白粉病的一项既安全又经济有效的措施,而抗病育种的基础是多样化的抗源.深入研究抗病基因的抗性表现及遗传特点,将有助于对这些抗源进行有效利用.目前小麦白粉病抗性基因的研究中有关主效基因的研究较多,其正式命名的基因符号为Pm(po w-dery m ildew),未正式命名的临时符号或早期符号为Ml.曾有相关文献对小麦抗白粉病基因研究进行综述报道[2~8].近年来研究工作进展很快,陆续有新基因鉴定出来,其所在的染色体位置也基本明确(表1).P m1:该基因最早在澳大利亚春小麦品种T hew、法国品种Nor mandie以及加拿大品种Ax minster中发现.Sears和Briggle[9]利用中国春端体系将Pm1基因定位于7A染色体长臂上. Hsam等[10]发现该基因位点包括4个复等位基因(P m1a,Pm1b,P m1c,Pm1d),其载体品种分别为Ax minster/8*Cc、含有一粒小麦(T riticum mono-coccum,2n=2x=14,AA)抗白粉病基因的抗病衍生系M ocZlatka、德国抗病新品系Weihenstephan Stamm M1N(M1N)(原定名为P m18)和来自罗马尼亚的斯卑尔脱小麦(T.sp elta v ar.d uhamelianum(TSD),2n=6x=42,AABBDD)抗病系T RI2258.收稿日期:2002-04-12;修改稿收到日期:2002-07-04基金项目:中以农业研究基金(SIARF)项目、国家自然科学基金项目(30200174)、北京市科委项目(951501800)作者简介:解超杰(1969-),男(汉族),副教授,博士,主要从事小麦抗病育种研究.*通讯联系人.C or res pon dence to:SUN Qi-xin.表1 小麦抗白粉病基因T able1 Resistance g enes to wheat po wdery mildew基因Gene 位点Location来源Sour ce代表品系Cultivar sPm1a7AL普通小麦Axm inster/8*CcPm1b7AL一粒小麦M ocZlatkaPm1c7AL一粒小麦M1NPm1d7AL斯卑尔脱小麦T.sp elta var d uhamelianum T RI2258 Pm25DS未确定Ulka/8*CcPm3a1AS普通小麦As os anPm3b1AS普通小麦Ch ulPm3c1AS普通小麦S on or aPm3d1AS普通小麦Kolibr iPm3e1AS普通小麦AUS6449Pm3f1AS普通小麦M ichigan Amber/8*CcPm3g1AS普通小麦Aris tidePm3h1AS普通小麦Abess iPm3I1AS普通小麦N324Pm3j1AS普通小麦GUS122Pm4a2AL二粒小麦Khapli/8*CcPm4b2AL波斯小麦ArmadaPm5a7BL二粒小麦HopePm5b7BL未确定IbisPm5c7BL印度园粒小麦KolandiPm5d7BL普通小麦IGV1-455Pm5e7BL普通小麦复壮30Pm6T2B/2G提莫菲维小麦Coker747Pm7T4BS.4BL-2R#1L黑麦(Ros en)T ran secPm8T1BL/1RS黑麦(Petku s)Amb as sadorPm97AL普通小麦Normandie(Pm1,P m2)Pm101D普通小麦Norin4Pm116BS普通小麦Ch ines e Sprin gPm12T6BS-6SS.6S L拟斯卑尔脱山羊草Line#31(W embley*6/Ae.sp eltoid es) Pm13T3BL.3BS-3S l#1S高大山羊草R1AT3DL.3DS-3Sl#1S R1DPm146B普通小麦Norin10(P m15)Pm157DS普通小麦Norin10(P m14)Pm164A野生二粒小麦T.d icoccoides CL1060025Pm17T1AL/1R#2S黑麦(Insave F.A.)AmigoT1BL/1R#2S T AM107Pm197D粗山羊草XX186Pm20T6BS/6RL黑麦(Prolific)PI583795Pm21T6AL/6VS簇毛麦R137,R55等Pm221D普通小麦VirestPm235A普通小麦Line81-7241Pm241DS普通小麦Ch iyacaoPm251A野生一粒小麦NC96BGTA5Pm262BS野生二粒小麦T TD140Pm27T6B/6G提莫菲维小麦146-155-TPm281B未确定M eriPm297DL卵穗山羊草PovaPm305BS野生二粒小麦87-1/C20//2*8866Pm31(M lG)6AL野生二粒小麦G-305-M/781//Jing411*3 Pm2:Pug sley和Carter[11]发现前苏联地方品种Ulka中带有不同于P m1的抗病基因,后来U lka 中的抗病基因被定名为P m2[12].M cIntosh和Bak-er[13]将P m2定位于5D染色体短臂上.Nyquist[14]发现在提莫菲维小麦(T.timo p hee-vii,2n=4x=28,AAGG)的六倍体小麦衍生系 CI12633 中至少有两个抗白粉病基因,其中一个显性基因为Mlx.Brig gle[15]根据U lka和CI12632 (CI12632为CI12633的姊妹系)测交分析及Ulka/ 8*Cc和CI12632/8*Cc的抗性比较结果,认为P m2和Mlx是等位基因,而其中另一个基因后来证明是P m6.关于P m2的来源有不同的报道.Bennett[7]分析了CI12632和CI12633的系谱(T.timo p heevii//8235期解超杰,等:小麦抗白粉病基因Illino is No.1/Chinese Spring),认为P m2的来源未知.因为Illinois No.1和Chinese Spring不含有Pm2,而提莫菲维小麦无D组染色体,其中的抗病基因不可能位于5D染色体上.谢皓,陈孝[4]和贾继增[2]认为P m2来自于CI12632和CI12633系谱中的提莫菲维小麦,经易位到染色体5DS上.齐莉莉等[16]根据与已知白粉病抗性基因比较的抗谱分析表明,阿拉拉特小麦(T. ar aratum,2n=4x=28,AAGG)携有主效抗病基因Pm2.阿拉拉特小麦和提莫菲维小麦具有相同染色体组,该结论似乎与谢皓,陈孝[4]和贾继增[2]的推论相符.然而,Lutz等[17]通过抗谱分析、染色体定位以及等位性测验证明合成六倍体小麦 XX194 (T. durum'M oro ccos183'/A e.squarrosa'AE457/78')含有Pm2基因,抗病基因来自于方穗山羊草(A e. squar rosa,2n=2x=14,DD).Lutz等[17]还介绍Pugsley[18]发现澳大利亚小麦选系 Javelin-325 带有Mlx(即P m2)基因,该基因来自 Sears 合成六倍体小麦(T.dur um cv'Ium illo'/A e.squar rosa)中的方穗山羊草,因为Ium illo是感病品种.这些结果表明P m2很有可能是来自方穗山羊草.尽管如此,仍不能排除P m2可能是自发突变产生的[17].Pm3:Br ig gle[12]在对23个小麦抗病品种(系)的研究中发现并命名了该基因.以后Br ig gle[15]又进一步证明该基因位点具有3个等位基因,即P m3a, Pm3b,P m3c,载体品种分别为Asosan,Chul和Sonora.单体及端体分析结果表明,这些抗病基因位于1AS上[19].Heun和Fischbeck[20]发现小麦品种Ko libri中含有抗病基因Mlk,Zeller等[21]将其定位到1A,并证明是P m3的复等位基因,定名为Pm3d.目前Pm3位点已经发现了10个等位基因[8],其载体品种分别是澳大利亚品种A US6449(P m3e)、美国品种Michig an Amber/8*Cc(P m3f)、法国品种Aris-tide(P m3g,即M lar)、埃塞俄比亚硬粒小麦和德国春小麦品种Amor杂交组合的六倍体小麦衍生系Abessi(P m3h),来自尼泊尔的品系N324(Pm3i)和来自俄罗斯的品系GUS122(Pm3j).Pm4:该位点目前有2个等位基因(P m4a和Pm4b).据Bennett[7]介绍,Briggle[22]首先发现在印度广泛用作育种亲本的二粒小麦(T.dicoccum,2n =4x=28,AABB)品系Khapli的抗病性.后来Br ig gle[23]将Khapli中的抗病基因转移到普通小麦品种Chancellor中,命名为P m4,以后发现了另一个等位基因,改名为P m4a[24].P m4b(原名Mle)来源于四倍体波斯小麦(T. carthlicum,2n=4x=28,AABB),带有Pm4b的品种有ELS,T P229,Arm ada等.The T T等[24]发现Pm4a和Mle(即Pm4b)都位于2AL上,表现出等位基因或紧密连锁的遗传特性.遗传研究表明Khapli带有3个抗病基因,而Khapli/8*Cc只含有P m4a,说明从Khapli只转过来一个抗病基因[7].这种现象在从近缘物种向小麦中导入抗病基因的过程中很普遍.P m5:Lebsock和Brigg le[25]将小麦品种H ope 具有的一个隐性抗白粉基因命名为P m5,该基因苗期不能充分表达,4~5叶期接种则抗性表现明显.因为Hope是感病普通小麦品种Mar quis和二粒小麦Yaroslav的杂交后代,因此推断P m5来源于二粒小麦Yaroslav[7].Hope的抗白粉病基因定位在染色体7BL上[26].Ho pe染色体7BL上除有Pm5以外,还有一个隐性抗秆锈基因Sr17和一个不完全显性抗叶锈基因Lr14a.Hope曾在小麦育种中广泛应用.抗白粉病基因M li最初在法国冬小麦品种Flanders等中发现[7].后来Heun和Fischbeck[27]证明Mli抗性方式与Pm5相似,并将Mli定名为P m5.但是M cIntosh[28]指出P m5是由二粒小麦转移到普通小麦Hope,而M li却没有这种明显的起源关系.Hsam等[29]证明P m5为一复等位基因位点,包括Hope中的Pm5a、Ibis等中的P m5b(即M li)、印度圆粒小麦(T.sp haerococcum v ar. R otund atum)品系Kolandi中的P m5c和普通小麦品系IGV1-455中的P m5d,而P m5d的来源是中国的普通小麦.最近,Huang等[30]在中国小麦品系复壮30中又鉴定出Pm5e.P m6:来源于提莫菲维小麦,曾被认为是最成功和广泛应用的抗白粉病基因[7],由Jorg ensen和Jensen[31]正式命名.据Bennett[7]介绍,Nyquist[14]发现在CI12633中除含有Mlx(即P m2)外,还有一个独立的基因,与M lx共同控制成株抗性,并与抗秆锈病基因S r36(原定名为Sr9c)紧密连锁,Jor-g ensen和Jensen[32]证明这两个基因分别是Pm2和P m6.P m6与S r36紧密连锁,而S r36定位于2B 上,因此P m6也位于2B上.Friebe等[33]根据C-分带证明P m6位于易位染色体T2B/2G上,易位部分824西 北 植 物 学 报23卷涉及2B的两个臂.陶文静等[34]也通过分子标记证明了这一结论.Pm7:该基因是通过辐射易位由黑麦(Secale cer eale,2n=2x=14,RR)品种Rosen导入普通小麦T ransec中[35],其中黑麦染色体2RL的一段易位到小麦染色体4BL上,形成T4BS.4BL-2RL易位系[33].该易位片段还带有抗叶锈基因L r25,这两个基因表现为紧密连锁.Pm7并没有得到广泛应用.Pm8:来源于黑麦,位于黑麦品种Petkus染色体1RS上.通过1R(1B)代换系(如洛夫林13等)或1BL/1R#1S易位系(如洛夫林10等)转入普通小麦中.在这个黑麦染色体片段上除有P m8外,还有抗条锈基因Yr9,抗叶锈基因L r26,和抗秆锈基因Sr31[28,33].转入小麦的黑麦Pm基因中,P m8的应用最为广泛.Pm8最初推广时表现高抗,但随着与之相对应的致病性小种频率迅速上升,其抗性很快丧失[7]. Pm8在我国曾经广泛地被应用,现在也已丧失抗性[1].Pm8基因的表达受到其它基因的抑制,包括位于小麦1A S染色体上的抑制基因Su P m8[36]和位于小麦品种Caribo染色体7D上的Pm8与Pm17的抑制基因[37].Pm9:法国品种No rmandie中除含有Pm1和Pm2外,还有P m9.Schneider[38]对Pm9与P m1和Pm2相对遗传关系的研究发现,Pm9呈单基因隐性遗传,与P m2互相独立,而与Pm1连锁,遗传距离为8.5cM.已知Pm1位于7AL,因此判断P m9也在7AL上.含有该基因的品种还有Mephisto,An-field,Pom pe和Ring.Pm10和Pm11:To sa等[39,40]利用小麦白粉菌(E.graminis f.sp.tritici)和冰草白粉菌(E. graminis f.sp.agr o p y ri)的杂交后代,在No rin4, Norin26等品种中检测出P m10基因,在中国春中检测出P m11,并分别定位到染色体1D和6BS上.Pm12:来源于拟斯卑尔脱山羊草(Ae.sp el-toides,2n=2x=14,SS),通过易位转移到小麦品种Wembley中,获得抗病材料品系31号(line# 31).P m12最初定位于小麦染色体6A,但Jia等[41]利用分子标记将该基因重新定位到易位染色体T6BS-6SS.6SL上.Pm13:来源于高大山羊草(Ae.longissim a(= T.longissimum),2n=2x=14,SlSl).Ceoloni等[42]通过诱导部分同源染色体配对重组方法将高大山羊草3Sl短臂携带的显性抗白粉病基因Pm13转移到中国春中,获得了R1A,R1D等抗病易位系.单体分析和C分带研究表明,R1A等易位系中Pm13位于易位染色体T3BL.3BS-3S l#1S上,而R1D等易位系中该基因位于易位染色体T3DL.3DS-3 S l#1S上[33].P m14和Pm15:T osa等[43]用小麦白粉菌和冰草白粉菌的杂交种,发现在Akabozu,Norin10等品种中含有P m14和P m15基因,分别定位于6B和7D染色体上.P m10,Pm11,P m14和Pm15只对冰草白粉菌表现抗性,不抗小麦白粉菌,在小麦抗病育种中无实际利用价值.P m16:Reader和M iller[44]将野生二粒小麦(T.d icoccoides,2n=4x=28,AABB)材料CL1060025中的抗白粉病基因Pm16转入普通小麦Norman 中,并将该基因定位于4A染色体上.P m17:来源于黑麦品种Insave F.A..经用X 射线处理普通小麦与八倍体小黑麦杂交种,获得了抗麦二叉蚜的品种Amigo[45].在Am igo中,黑麦染色体1RS易位到1A上,形成T1AL.1R#2S易位系.在这段1RS片段上不仅含有抗麦二叉蚜基因G b2,还含有一个与Pm8不同的抗白粉病基因,命名为P m17[46,47].后来Hsam等[48]通过 Am ig o/Helios(T1BL. 1RS) 杂交组合将P m17转移到易位系T1BL.1RS 上,并证明P m17和P m8之间是等位基因关系[49].近来发现小麦品种Car ibo的7D染色体上带有P m8与P m17的抑制基因[37].P m18:据Hsam等[10]介绍,德国普通小麦品系W eihenstephan Stam m M1(M1)携带有P m4b基因,而在1986年得到一个M1种子样品,经鉴定这个样品的抗谱与原M1不同,为一新抗病系,取名为W eihenstephan Stamm M1N(M1N),其中含有的抗病基因被定名为Pm18[50].Hsam等[10]通过单体分析发现该基因位于7A染色体上,并与Ax minis-ter/8*Cc中的P m1a基因等位,据此将其重新命名为Pm1c.Hsam等[10]认为Pm1c可能来自于一粒小麦.P m19:Lutz等[17]根据对小麦白粉菌不同生理小种的抗性反应,在合成六倍体小麦XX186中发现了一个显性抗病基因,并利用单体分析证明该基因位于7D染色体上.由于在7D上尚未有已知P m基因,因此命名该基因为P m19.XX186的组合为8255期解超杰,等:小麦抗白粉病基因T.durum v ar Santa M arta'/A e.squarrosa BGRC 1458',由于硬粒小麦'Santa M ar ta'是感病品种,所以判断P m19来自方穗山羊草.Pm20:来自黑麦品种Prolific.Friebe等[51]用小麦-黑麦6RL(6D)单体代换系M S6RL(6D)与小麦-黑麦T6BS.6R#2L纯合易位系品种T AM104杂交,通过同源重组将抗白粉病基因Pm20转移到易位染色体T6BS.6R#2L上,所获得的抗病材料命名为 KS93WGRC28 [52].据Fr iebe等[33]报道,在 KS93WGRC28 中的易位染色体形成T6BS.6R# 2L-6R#3L.C分带结果表明,Pm20位于Pro lific的6R#3染色体长臂的端粒附近.Pm21:这是中国学者发现的第一个小麦抗白粉病基因[53].该基因位于簇毛麦(H ay naldia villosa, 2n=2x=14,VV)6V染色体短臂,通过染色体添加、代换和易位转入普通小麦中[53,54].目前应用的多为T6AL.6VS易位系,李辉等[54]还获得了T6DL.6VS易位系.Pm22:Peusha等[55]通过抗谱分析发现小麦品种Virest带有新的抗白粉病基因,单体分析表明该基因为一显性主效基因,位于1D染色体上,命名为Pm22.含有该基因的还有意大利品种Elia,Est M ottin,Ov est和T udest等.Pm23:四川农业大学杨足君(YANG Z J)和任正隆(REN ZH L)报道在小麦品系81-7241中发现新的抗白粉病基因,并定位于染色体5A上,命名为Pm23[28].Pm24:Huang等[56]发现中国小麦地方品种Chiy acao中含有一个与已知P m基因抗谱不同的新基因,通过单体分析将Chiyacao中的这个显性主效抗病基因定位于6D上,并命名为P m24.但是后来通过分子标记证明该基因应该是位于1DS[57].Pm25:Shi等[58]从野生一粒小麦(T.m onococ-cum var.boeoticum,2n=2x=14,AA)向普通小麦转移了一个显性抗白粉病基因,命名为P m25.遗传分析表明该基因与Pm3a连锁,据此将该基因定位于1A上.Pm26:来自野生二粒小麦.Rong等[59]将以色列野生二粒小麦T TD140中的一个抗白粉病基因转移到普通小麦Bethlehem中.该基因为隐性,命名为P m26,通过对一系列染色体臂代换系(chro mo-some-arm substitution lines CASLs)进行RFLP标记被定位于2BS上.P m27:Ja¨r ve等[60]通过杂交和回交从提莫菲维小麦材料K-38555向普通小麦146-155中转移了一个显性抗白粉病基因,获得纯合抗病系146-155-T.单体分析和分子标记研究表明该基因来自6G并易位到6B染色体上,命名为P m27.P m28:Peusha等[61]根据单体分析结果证明芬兰-爱沙尼亚春小麦品种M eri带有一个显性抗白粉基因,定位于1B.该基因与原来1B上来自黑麦的P m8和P m17的抗性表现不同,命名为P m28.目前尚未确定P m28的来源.P m29:Zeller等[62]从普通小麦Poros的卵穗山羊草(A e.ov ata,2n=4x=28,UU MM)二体异附加系POS的后代中,选出了具有42条染色体的抗白粉病品系Po va,遗传分析表明Pova带有一个显性抗白粉病基因.该基因位于小麦染色体7D上,但与7D上的P m19基因独立分离,进一步的分子标记分析将其定位于7D长臂,正式命名为P m29.P m30:中国农业大学小麦组通过杂交和回交将以色列野生二粒小麦材料C20的抗白粉病基因导入普通小麦遗传背景中,并通过分子标记将该基因定位在染色体5BS上,现在该基因被正式命名为P m30[63].这是由中国学者发现和定名的第3个小麦抗白粉病基因.P m31(MlG):来源于以色列野生二粒小麦材料G-305-M.这是中国农业大学小麦组得到的第二个来自以色列野生二粒小麦的抗白粉病基因,微卫星分子标记表明该基因位于小麦染色体6A长臂,临时命名为M lG[64],现被定名为P m31.抗白粉病基因在小麦染色体上广泛分布,目前,除2D,3A,4D,6D染色体外,其余染色体上都有抗白粉病基因位点存在.这些抗白粉病基因大多表现显性遗传,少数为隐性(如Pm5,P m9,P m26等).许多Pm基因来源于小麦近缘种属,因此利用外源抗病基因可以极大地丰富普通小麦抗病基因资源,同时也有助于扩大小麦育种的遗传基础.参考文献:[1] YANG Z M(杨作民),T ANG B R(唐伯让),SHEN K Q(沈克全),XIA X C(夏先春).A strategic problem in w heat resis tance b reeding-building and utilization of sources of second-line res istance against rus ts and mildew in China[J].A cta A gr onomica S inica(作物学报), 826西 北 植 物 学 报23卷1994,20(4):385-394(in C hinese ).[2] JIA J Z(贾继增).Resis tance gen es to pow dery m ildew and s ources in w heat[J].J ournal of Triticeae C rop s (麦类作物学报),1990,10:18-19(in Chinese).[3] LIU J Y(刘金元),LIU D J(刘大均).Progress of the study on w heat pow dery m ildew res istance genes [J].A cta Phyto p atholog ica S inica(植物病理学报),2000,30(11):289-295(in Chin es e).[4] XIE H(谢 皓),C HEN X(陈 孝).Progress of th e s tu dy on location of w heat pow dery mildew genes and r esis tance evaluation[J ].Jour -nal of B eij ing A g ricu ltu ral College (北京农学院学报),1998,13(3):104-112(in C hines e ).[5] ZHAN G D Y (张德玉),ZHONG S B (钟少斌),YAO J X (姚景侠).Pr ogr ess of th e study on location of w h eat pow dery mildew genes andtheir m olecular m ark ers [J].Journal of T ritic eae Cr ops (麦类作物学报),1994,3:44-47(in Ch ines e).[6] ZHANG R(张 瑞),ZHANG J Y(张继益).Progres s of th e study on location of w heat pow der y m ildew genes and m olecular marker s[J].Journal of T ritic eae Cr ops (麦类作物学报),1999,19(3):11-14(in Ch ines e).[7] BENNET T F G A.Resis tance to pow dery mild ew in w heat:a review of its us e in agriculture and b reeding program s[J].Plant P athol ,1984,33:279-300.[8] ZE LLER F J ,HSAM S L K .Progress in breeding for resistance to p ow dery m ildew in common wh eat (T riticum aestiv um L .)[A ].In :Slinkard AE (ed )Proc 9th Int Wheat Genet S ymp [C ].Sas katoon ,Sask .,Canada ,U nivers ity Ex tention Pr ess ,1998,Vol .1:178-180.[9] SEARS E R,BRIGGL E L W.M apping the gene P m 1for res istance to Erysiph e gram inis f.sp.tritici on ch romosome 7A of w heat[J].Crop Sc i ,1969,9:96-97.[10] HSAM S L K,HU ANG X Q,ERNST F,HARTL L,ZELL ER F J.Chromos om al location of genes for res istance to pow der y mildewin common w heat (Tr iticum ae stiv um L.em.Th ell.)5.Alleles at the P m 1locus [J].T heor Ap pl Genet ,1998,96:1129-1134.[11] PULGSLEY A T ,CART ER M V.Resis tance of tw elve varieties of T riticum v ulgar e to Erys iphe gr amin is tritici[J ].A ust J of B iolS ci ,1953,6:335-346.[12] BRIGGL E L W .T hree loci in w heat involving res istan ce to E rysip he graminis f .sp .tritici [J ].Crop Sc i ,1966,6:461-465.[13] M CINT OS H R A ,BAKER E P .Cytogenetic studies in w heat IV .Ch romosomal location and linkage studies involving the P m 2locu sfor pow dery mildew res istance[J].Euphytica ,1970,19:71-77.[14] NYQUIST W E.Inh eritance of powd ery m ildew res istan ce in hyb rids in volving a comm on w heat s train d erived from T ritiv um timop hee -v i [J].Crop Sc i ,1963,3:40-43.[15] BRIGGL E L W.Near-isogenic lines of w heat w ith genes for res istance to Erysip he g raminis f.s p.tr itici [J ].Crop S ci ,1969,9:70-72.[16] QI L L (齐莉莉),ZHOU B(周 波),ZHANG S Z(张守忠),CH EN P D(陈佩度),LIU D J(刘大均),SHENG B Q(盛宝钦),XIANG Q J(向齐君),DU N X Y (段霞瑜),ZHOU Y L (周益林).Trans fer of a gene to powd ery mildew resis tance from T ritic um ararticum J ak ubz to T riticum aestiv um L .I .Studies on cytogenetics and identification of powdery m ildew res istance in hybrid progenies of T .aestiv um and T .arar atic um [J].A cta Genetica S inica (遗传学报),1998,25(1):59-66(in Chinese).[17] LUT Z J,HSAM S L K,L IM PERT E,ZELLER F J.Chromos omal location of pow dery mild ew r esis tance gen es in Tr iticum ae stiv umL.(common w heat). 2.Genes P m 2and Pm 19from A eg ilops squar rosa L.[J].H ered ity ,1995,74:152-156.[18] PULGSLEY A T.Additional r esis tance in Tr iticum vu lg are to Ery sip he g raminis tr itici [J ].A ust J B iol Sci ,1961,14:70-75.[19] M CINT OS H R A,BENNET T F G A.Telocentric mapping of genes P m 3a and Hg on chromos ome 1A of hexap loid wh eat[J].CerealRe sear ch Communications ,1978,6:9-14.[20] HEUN M ,FIS CHBECK G .Genes for pow dery mildew res istance in cultivars of sprin g w heat [J ].P lant B reed ,1987,99:282-288.[21] ZELLER F J ,LU TZ J ,ST EPHAN U .Ch romosomal location of genes for res istance to pow dery mildew in common w heat (Tr iticumaestiv um L.).1.M lk and other alleles at the P m 3locus [J ].E up hy tica ,1993,68:223-229.[22] BRIGGL E L W.S ources of resistance to pow dery mildew from tetraploid Tr iticum species [J].A gronomy A bstracts ,1960,65:45.[23] BRIGGLE L W.T ran sfer of res istan ce to Ery sip he graminis f.s p.tr iti ci fr om K h ap li emmer and Yuma du rum to h exaploid w heat[J].Crop Sci ,1966,6:459-461.[24] T HE T T ,M CINTOS H R A ,BENN ETT F G A .Cytogen etic s tudies in w heat .IX .M onosom ic analysis ,telocentric mapping and link-age r elationsh ips of gen es S r 21,P m 4,and M le [J ].A ust J Biol S ci ,1979,32:115-125.[25] LEBSOCK K L ,BRIGGLE L W .Gene Pm 5for res istance to E ry sip he gr aminis f .sp .tritici in Hope w heat [J ].Cr op S ci ,1974,14:561-563.[26] L AW C N,W OLFE M S.Location of genetic factors for mildew r esis tance and ear emergence tim e on chr om osome 7B of w heat[J].Can J Genet A nd Cytol ,1966,8:462-470.[27] HEUN M ,FISCHBEC K G.Identification of w heat pow dery mildew res istan ce gen es by analysing host-pathogen inter actions [J].P lant8275期解超杰,等:小麦抗白粉病基因828西 北 植 物 学 报23卷Br eed,1987,98:124-129.[28] M CINT OS H R A,HA RT G E,DEVOS K M,GALE M D,ROGERS W J.Catalog ue of gene symbols for w heat[A].In:Slin kar d AE(ed)Proc9th In t W heat Genet Symp[C].S as katoon,Sask.,Canada,U nivers ity Extention Press,1998,Vol.5:123-127.[29] HS AM S L K,HU ANG X Q,ZELLE R F J.Chromos om al location of genes for resis tance to powd ery mildew in common w heat(Tr iticum aestiv um L.em.Th ell.) 6.Alleles at the P m5locus[J].Theor App l Genet,2001,02:127-133.[30] HUANG X Q,W ANG L X,XU M X,RO¨DER M S.M icr os atellite mapping of the pow dery mildew res istance gene Pm5e in commonw heat(T riticum aestiv um L.)[J].T heor App l G enet,2003,6:858-865.[31] JORGENS EN J H,JENS EN C J.Gen e Pm6for resis tance to pow dery mildew in wh eat[J].E up hytica,1973,22:423.[32] JORGENSSE N J H,JENS EN C J.Genes for resistance to w heat pow dery mild ew in derivatives of T riticu m timop heev i and T.carth-licum[J].Euphytica,1972,21:121-128.[33] FRIEBE B,JIANG J,RAU PP W J,M CINTOS H R A,GILL B S.Characteriz ation of w h eat-alien trans locations conferrin g resis tanceto dis eases and pests:cur rent statu s[J].Eup hytica,1996,91:59-87.[34] TAO W J(陶文静),LIU J Y(刘金元),LIU D J(刘大均),et al.Precise identification of th e introgres sed s egm ents in pow dery mildew-res istant in trogr ess ion lines of Tr iticum aestiv um-T.timop heev i[J].A cta Botanic a S inica(植物学报),1999,41(9):941-946.[35] DRIS COLL C J,JENSE N N F.Release of a w heat-rye translocation stock in volving leaf rus t and pow dery mildew res istances[J].Crop Sci,1965,5:279-280.[36] REN S X(任树新),W U Y W(吴郁文),M C INT OSH R A,ZHANG C L(张翠兰),LIU C G(刘春光),SHARP P J,THE T T,ZHANG Y(张 炎).A genetic study on th e suppr ess ion of the pow dery mildew res istan ce gene P m8[J].A cta Ge netica S inica(遗传学报),1997,24(4):336-343(in Ch ines e).[37] ZELLER F J,HSAM S L K.Chromosom al location of a gene suppress ing pow dery mildew r esis tance genes Pm8and Pm17in commonw heat(T ritic um aestiv um L.em.T hell.)[J].T heor App l G enet,1996,93:38-40.[38] SC HNEIDER D M,HE UN M,FIS CHBECK G.Inheritan ce of th e pow dery mildew resis tance gene Pm9in relation to Pm1and Pm2ofw heat[J].Plan t Breed,1991,107:161-164.[39] T OSA Y,TS UJIM OT O H,OGU RA H.A gene involved in the resistance of wh eat to wh eatgras s pow dery mild ew fungus[J].Ge nome,1987,29:850-852.[40] T OS A Y,T OKUNAGA H,OGURA H.Identification of a gene for resis tance to w heatgr as s pow dery mildew fungu s in comm on w heatcultivar Chinese S pring[J].Ge nome,1988,30:612-614.[41] JIA J,DEVOS K M,CHAO S,M ILLE R T E,READ S M,GALE M D.RFLP-based maps of the homoeologous group-6chromo-somes of w heat and their application in the tagg ing of Pm12,a pow dery mildew res istance gene transferr ed fr om A eg ilo p s sp eltoides to w heat[J].The or App l Genet,1996,92:559-565.[42] CEOLONI C,SIGNORE G,ERCOLI L,DONINI P.L ocating the alien chromatin segment in common w heat-Aegilops longis simamild ew r esis tant tran sfers[J].H ered itas,1992,116:239-245.[43] T OSA Y,S AKAI K.T he genetics of resistance of hexaploid w h eat to th e w heatgrass pow dery m ildew fungus[J].Genome,1990,33:225-230.[44] RE ADER M,M ILLE R T E.Th e introduction into bread w heat of a m ajor gene for resis tance to pow dery m ildew from w ild emmerw heat[J].Euphytica,1991,53:57-60.[45] SEBES TA E E,WOOD E A,PORT ER D R,WE BS TE R J A,SM IT H E L.Registration of Amigo w h eat germp las m resis tant togreenbu g[J].C rop S ci,1995,35:293.[46] HEUN M,FRIEBE B,BU SHUK W.Ch romosomal location of the pow dery mildew res istance gene of Amigo w heat[J].Phyto p atholo-gy,1990,80:1129-1133.[47] LOWRY J R,S AM M ONS D J,BAENZIGER P S,M OSE M AN J G.Identification and ch aracterization of the gene conditionin g pow-dery mildew res istan ce in'Amigo'w heat[J].Crop S ci,1984,24:129-132.[48] HSAM S L K,CERM ENO M C,FRIEBE B,ZE LLER F J.T rans fer of Amigo w heat pow dery mildew resistance gene P m17fromT1AL.1RS to th e T1BL.1RS w heat-rye tran slocation chrom os ome[J].H e redity,1995,74:497-501.[49] HSAM S L K,ZELLER F J.Eviden ce of allelism betw een genes Pm8and Pm17and chr om osomal location of powdery m ildew and leafrus t resistance g enes in th e common w heat cultivar'Am igo'[J].Plant Br eed,1997,116:119-122.[50] ZELLER F J,ST EPHAN U,LUT Z J.Present statu s of w heat pow dery mild ew resistance genetics[A].In:L i ZS,Xin ZY(eds)Proc8th In t W heat Genet Symp[C].Beijing,C hina Agricultural Scientech Pres s,1993,929-931.[51] FRIEBE B,HEU N M,T ULEE N N,ZEL LER F J,GILL B S.Cytogenetically monitored transfer of pow dery mildew res istan ce fromrye into wh eat [J ].Cr op S ci ,1994,34:621-625.[52] FRIE BE B,GIL L B S ,T ULE EN N A,COX T S.Regis tration of KS 93WGRC28pow dery mildew res istant T 6BS.6RL w heatger mplasm[J ].Cr o p S ci ,1995,35:1237.[53] QI L L (齐莉莉),CHE N P D(陈佩度),LIU D J (刘大均),ZHOU B(周 波),ZHANG S Z(张守中),SHENG B Q(盛宝钦),XIANG Q J(向齐君),DUAN X Y(段霞瑜),ZHOU Y L(周益林).T he gene Pm 21-a n ew s ource for resis tance to w heat powd ery m ildew [J ].Ac ta A gr onomica S inica (作物学报),1995,21(3):257-262(in Ch ines e ).[54] LI H (李 辉),CHEN X (陈 孝),XIN Z Y (辛志勇),M A Y Z (马有志),XU H J (徐惠君).Developm ent an d identification of w heat -Hayn aldia villos a 6DL /6VS trans location lines w ith powdery m ildew res istance[J].S cientia A g ric utura S inica (中国农业科学),1999,32(5):9-15(in C hinese).[55] PEUSHA H,HSAM S L K,ZELLER F J.Chromos omal location of p ow dery mildew res istan ce genes in common w heat (Tr iticum aes -tiv um L.em.T hell.). 3.Gene P m 22in cultivar Virest[J].Eup hytica ,1996,91:149-152.[56] HUANG X Q ,HS AM S L K ,ZELLER F J .Chromosom al location of gene s for resis tance to p ow dery mildew in comm on w heat(Tr iticum aestiv um L .em .Th ell .) 4.Gene Pm 24in Chinese landr ace Chiyacao [J ].T heor Ap pl Genet ,1997,95:950-953.[57] HUANG X Q,HS AM S L K,ZE LLER F J,W ENZEL G,M OHLER V.M olecu lar mapping of the w heat powdery mildew resis tancegen e Pm 24and m ark er validation for molecu lar br eeding[J].The or App l Gene t ,2000,101:407-414.[58] SHI A N,LEAT H S,M U RPHY J P.A major gene for pow dery mildew r esis tance transfer red to comm on wh eat from wild ein kornw heat[J].Phyto p athology ,1998,88:144-147.[59] RONG J K ,M ILLET E ,M ANIST ERS KI J ,FELDM AN M .A new pow dery mildew resis tance gene :Introgress ion from wild emmerinto common w heat and RFLP -based mapp ing [J ].Euphytica ,2000,115:121-126.[60] Ja ¨RVE K,PEUS HA H O,T SYM BALOVA J,TAM M S ,DEVOS K M ,ENNO T M.Chromos om al location of a T riticum timophee-vii-derived p ow dery m ildew resistance gene trans ferred to common w heat[J].Genome ,2000,43:377-381.[61] PEUS HA H,ENNO T ,PRIILINN O.Chromosom al location of pow dery m ildew res istance gen es and cytogen etic analys is of meiosis incomm on w heat cultivar M eri[J].H ered itas ,2000,132:29-34.[62] ZE LLER F J ,KONG L ,HART L L ,M OHLER V ,H SAM S L K .Chromos omal location of g enes for r esis tance to powd ery m ildew incomm on w heat (Tr iticum aestiv um L .em T hell .)7.Gene Pm 29in line Pova [J ].E up hytica ,2002,123:187-194.[63] LIU Z Y,S UN Q X,NI Z F,NE VO E,YANG T M .M olecu lar characterization of a novel pow dery mildew r esis tance gen e Pm 30inw heat originating from wild emmer[J].Eup hytica ,2002,123:21-29.[64] XIE C,SUN Q,NI Z,YANG T ,NEVO E,FAHIM A T.Ch romosomal location of a T riticu m dicoccoides-derived pow dery mildew re-sis tance gene in w heat by using microsatellite mar kers [J].The or App l Genet ,2003,106:341-345.8295期解超杰,等:小麦抗白粉病基因。

简单干净的网名大全

简单干净的网名大全

简单干净的网名简单干净的网名大全简单干净网名篇11、啼红泪。

2、夜色ザ微凉3、冷星魂。

4、夜晟洛。

5、梦冥光。

6、枫无痕。

7、畵凉思。

8、翠烟寒。

9、洛倾颜。

10、静花寒。

11、墨雪卿。

12、陌上花。

13、七秒梦。

14、琴弦上。

15、幕筱晨。

16、花间辞。

17、紫心柔。

18、玻璃心。

19、大天使之剑20、青隐篱。

21、血色骑士。

22、竹青磬。

23、云中歌。

24、乍疏雨。

25、碎流璃。

26、殃樾晨。

27、水空流。

28、陌若惜。

29、画卿颜。

30、花泪兮。

31、梦疾风。

32、残风葬。

33、断秋风。

34、龙吟凤。

35、萧声断。

36、岚风殇。

37、楚碧瑶。

38、黄萝卜魔咒39、陌南尘。

40、桃洛憬。

41、百万亚瑟王42、墨子渊。

43、天之镜。

44、╱方丈大师45、青衫隐。

46、封刀不再战47、箜篌引。

48、烟花落。

49、青丝绕。

50、客梦回。

51、似最初。

52、冷青裳。

53、夜翎绮。

54、梦一场。

55、陌潇潇。

56、花寒弦。

57、冷残影。

59、洗尘衫。

60、安颜丶。

61、不讨囍。

62、冷兮丶。

63、秋雨涩。

64、凝残月。

65、云若依。

66、风晴雪。

67、焚天狂。

68、冷汐夜。

69、汐剑云。

70、幻血凰。

71、末言雨。

72、梦三年。

73、风又吹。

74、蝶恋花。

75、惜醉颜。

76、含笑拥刀锋77、╰妖尛薇╮78、忆逝逝。

79、△菠萝派◎80、弑雪殇。

81、凉景懿。

82、夏唯夜。

83、七星灯。

84、独尊、宠妃85、南鸢°。

86、迟冷熙。

87、花锦瑟。

88、落羽殇。

89、绿亦歌。

90、颜洛殇。

91、等君归。

92、清宵半。

93、夏筱语。

94、坠雨痕。

95、沧古烟。

96、花雨黯。

97、凌兮洛。

98、∑混血嗲妹99、染青城。

100、清幽兰。

101、清泪尽。

简单干净网名篇21、烂人不配情深2、ぁ綯气钸钉ぁ3、我有孤单陪我4、拒絕融化的冰5、依__难以割舍6、輕嘆亽吢多變7、虚情换来假意8、星空下的愿望9、笑的没心没肺10、姐就是棒棒哒11、我笑着说原谅12、怎么开始忘了13、在人群中淹没14、柠檬思密达~15、我们过了太久16、别妄图诱惑我17、重庆个暖男凯18、我是会受伤的`19、始终毫无保留20、追忆流年似水21、泪如雨,美如殇22、国家大秘密。

双数显光纤传感器 ER2-23 使用说明书

双数显光纤传感器 ER2-23 使用说明书

ER2-23双数显光纤传感器使用说明书----------------------------------操作面板说明:注意事项:为了确保您的安全,使用时请务必遵守以下条例:1、本产品仅供目标物检测之用。

请勿将本产品用于保护人体或人体部位等目的。

2、本产品不得作为防爆产品使用。

请勿在危险场所和/或潜在爆炸气体的环境中使用本产品。

3、该产品是DC 电源型传感器。

请勿使用AC 电源。

否则,会导致产品爆炸或着火。

4、请勿沿着电源线或高压线对放大器进行配线,否则传感器会因噪声发生故障或受损。

5、使用商用开关式稳压器时,确保将机框接地端子和接地端子接地。

6、请勿在室外或者外部光线能够直接进入光接收表面的位置使用。

技术规格:型号ER2-23ER2-23P类型NPN 输出PNP输出电源电压12-24VDC±10%,浮动P-P 10%以下消耗电流小于40mA光源红色,4元素发光二极管体检测方式漫反射,对射(由光纤类型确定)检测距离P-1漫反射200mm,对射700mm P-2漫反射280mm,对射1200mm检测输出NPN 集电极开路输出,最大输入电流100mA,外接最大电压30VDC,残余电压小于1V PNP 集电极开路输出,最大输入电流100mA,外接最大电压30VDC,残余电压小于2V输出状态长按(D/L)5S 进行常开/常闭切换延时功能无延时/单次输出延时/拉高延时/拉低延时,四种输出方式可选。

三种延时时间可调:1ms 至9999ms反应时间P--1:小于100uSP--2:小于200uSLED 显示器红色输出指示灯,阀值显示器(4位绿色LED),当前值显示器(4位红色LED)保护电路电源极性反接保护;输出短路或过载保护工作环境亮度白炽灯:最大:20,000lux,日光:最大:30,000lux耐振动性10至55Hz,双重振幅:1.5mm,X,Y,Z 轴分别是2小时环境温度-25至+55℃,无冻结输出电路:ER2-23NPN 型号ER2-23P PNP 型号尺寸图:(mm)正确的安装方法:安装在DIN轨道上1、将主机底部的卡槽与轨道对齐。

23员工关系全方位管理(ER)

23员工关系全方位管理(ER)

• 土壤学说 –公司有很多资源灌溉 土壤 –所有的员工在这片土 地上自然成长,接受 风吹雨打 –能够长高就继续长, 长不高就矮矮的,也 许被拔掉 –员工要与企业同度兴 衰、共图发展。
员工关系管理职能在人力资源部和公司的定位
总公司级: 总公司级:在一个有很多集团的多元化公司里,公司总部 人力资源部中要专人专岗来负责员工关系管理和企业文化 区域级:对于一个下设各大区域或者各分支机构、各代表 区域级: 处的公司来说,在总公司的人力资源部中,也需要单独设 置一个人专门负责企业文化和员工关系管理 分公司级: 分公司级:在一个只有分公司的公司中,因为岗位设置有 限,所以这类公司负责员工关系管理的一般就是人力资源 部的最高负责人,他的主要工作在于员工关系管理和企业 文化,至于招聘培训、考核、福利等内容则有其下属分管 部门级: 部门级:如果公司的规模较小,诸如最多各地有一些代表 处,每个代表处有两三个联络人员这样规模的公司。在这 样的公司中,切忌将员工关系管理置于行政部的工作中。 应该由公司的最高管理层来负责。 **规模越小的公司负责员工关系管理的人越分散,公司的老 规模越小的公司负责员工关系管理的人越分散, 规模越小的公司负责员工关系管理的人越分散 副总、 总、副总、中层经理都可以负责部分员工关系管理的工作
2,员工的参与式管理 如何获得员工的最大承诺? 如何获得员工的最大承诺?
第三 层次 第二 --目标明确 目标明确 我们如何共 --成就感 层次 成就感 同成长? 同成长 第一 --发挥最大潜力 发挥最大潜力 我属于这里吗 层次 --全力以赴 全力以赴 我能贡献什么
承诺
基本问题 我得到了什么? 我得到了什么 --公司对我工作的期望是什么 公司对我工作的期望是什么? 公司对我工作的期望是什么 --我是否拥有做好工作所需要的工具和设备 我是否拥有做好工作所需要的工具和设备

Recombinant autologous Epstein-Barr viral fusion p

Recombinant autologous Epstein-Barr viral fusion p

专利名称:Recombinant autologous Epstein-Barr viral fusion proteins, test kits containing themand methods for the detection of Epstein-Barr virus specific antibodies发明人:HINDERER, WALTER, DR.,VORNHAGEN,ROLF, DR.,LANG, DIETER, DR.,SONNEBORN,HANS-H., DR.申请号:EP96111173.9申请日:19960711公开号:EP0754755B1公开日:20031217专利内容由知识产权出版社提供摘要:New autologous fusion protein (I) comprises: (1) at least part of the p23 protein of Epstein-Barr virus (EBV) and (2) at least part of some other EBV protein. Pref. (1) includes at least the first 100 amino acids (aa), including the N-terminus, of p23, esp. the whole of it. (2) is (part of) a late EBV antigen, serologically defined as 'virus capsid antigen'; esp. those designated p150, p143, gp125, gp85, gp55, gp42, gp25, p40 and/or p18. Most prefd. is at least part, esp. up to 100 C-terminal aa, of p18. The specification includes a 759 aa sequence (and the DNA sequence encoding it) of a prefd. (I).申请人:BIOTEST AG,BIOTEST AG,BIOTEST AG地址:DE国籍:DE代理机构:Keller, Günter, Dr.更多信息请下载全文后查看。

x轴追随误差超过参数2号设定值

x轴追随误差超过参数2号设定值

x轴追随误差超过参数2号设定值下载提示:该文档是本店铺精心编制而成的,希望大家下载后,能够帮助大家解决实际问题。

文档下载后可定制修改,请根据实际需要进行调整和使用,谢谢!本店铺为大家提供各种类型的实用资料,如教育随笔、日记赏析、句子摘抄、古诗大全、经典美文、话题作文、工作总结、词语解析、文案摘录、其他资料等等,想了解不同资料格式和写法,敬请关注!Download tips: This document is carefully compiled by this editor. I hope that after you download it, it can help you solve practical problems. The document can be customized and modified after downloading, please adjust and use it according to actual needs, thank you! In addition, this shop provides you with various types of practical materials, such as educational essays, diary appreciation, sentence excerpts, ancient poems, classic articles, topic composition, work summary, word parsing, copy excerpts, other materials and so on, want to know different data formats and writing methods, please pay attention!X轴追随误差超过参数2号设定值的问题分析与解决方案引言在工业控制系统中,精确的轴追随是确保生产过程平稳运行的关键因素之一。

ERα(sc-542)抗体说明书 santa

ERα(sc-542)抗体说明书 santa

SANTA CRUZ BIOTECHNOLOGY,INC.ER α(MC-20):sc-542Santa Cruz Biotechnology,Inc. 1.800.457.3801831.457.3800fax 831.457.3801Europe +BACKGROUNDEstrogen receptors (ER)are members of the steroid/thyroid hormone receptor superfamily of ligand-activated transcription factors.Estrogen receptors,including ER αand ER β,contain DNA binding and ligand binding domains and are critically involved in regulating the normal function of reproductive tissues.ER αand ER βhave been shown to be differentially activated by vari-ous ligands.Receptor-ligand interactions trigger a cascade of events,including dissociation from heat shock proteins,receptor dimerization,phosphorylation and the association of the hormone activated receptor with specific regulatory elements in target genes.Evidence suggests that ER αand ER βmay be regulated by distinct mechanisms even though they share many functional characteristics.REFERENCES1.Danielian,P .S.,et al.1992.Identification of a conserved region required for hormone dependent transcriptional activation by steroid hormone receptors.EMBO J.11:1025-1033.2.Mosselman,S.,et al.1996.ERb:identification and characterization of a novel human estrogen receptor.FEBS Lett.392:49-53.CHROMOSOMAL LOCATIONGenetic locus:ESR1(human)mapping to 6q25.1;ESR1(mouse)mapping to 10A1.SOURCEER α(MC-20)is an affinity purified rabbit polyclonal antibody raised against a peptide mapping at the C-terminus of ER αof mouse origin.PRODUCTEach vial contains 200µg IgG in 1.0ml of PBS with <0.1%sodium azide and 0.1%gelatin.Blocking peptide available for competition studies,sc-542P ,(100µg peptide in 0.5ml PBS containing <0.1%sodium azide and 0.2%BSA).Available as TransCruz reagent for Gel Supershift and ChIP applications,sc-542X,200µg/0.1ml.Available as fluorescein (sc-542FITC)or rhodamine (sc-542TRITC)conjugates for immunofluorescence,200µg/ml.Available as Alexa Fluor ®405(sc-542AF405),Alexa Fluor ®488(sc-542AF488)or Alexa Fluor ®647(sc-542AF647)conjugates for immunofluores-cence;100µg/2ml.Alexa Fluor ®is a trademark of Molecular Probes,Inc.,Oregon,USASTORAGEStore at 4°C,**DO NOT FREEZE**.Stable for one year from the date of shipment.Non-hazardous.No MSDS required.RESEARCH USEFor research use only,not for use in diagnostic procedures.APPLICATIONSER α(MC-20)is recommended for detection of estrogen receptor αof mouse,rat and human origin by Western Blotting (starting dilution 1:200,dilution range 1:100-1:1000),immunoprecipitation [1-2µg per 100-500µg of total protein (1ml of cell lysate)],immunofluorescence and immunohistochemistry (including paraffin-embedded sections)(starting dilution 1:50,dilution range 1:50-1:500)and solid phase ELISA (starting dilution 1:30,dilution range 1:30-1:3000).Suitable for use as control antibody for ER αsiRNA (h):sc-29305,ER αsiRNA (m):sc-29306,ER αsiRNA (h2):sc-44204,ER αsiRNA (r):sc-45949ER αshRNA Plasmid (h):sc-29305-SH,ER αshRNA Plasmid (m):sc-29306-SH,ER αshRNA Plasmid (h2):sc-44204-SH,ER αshRNA Plasmid (r):sc-45949-SHER αshRNA (h)Lentiviral Particles:sc-29305-V,ER αshRNA (m)Lentiviral Particles:sc-29306-V,ER αshRNA (h2)Lentiviral Particles:sc-44204-V and ER αshRNA (r)Lentiviral Particles:sc-45949-V.ER α(MC-20)X TransCruz antibody is recommended for Gel Supershift and ChIP applications.Molecular Weight of ER α:66kDa.DATASELECT PRODUCT CITATIONS1.Massaad-Massade,L.,et al.2002.HMGA1enhances the transcriptional activity and binding of the estrogen receptor to its responsive element.Biochemistry 41:2760-2768.2.Guo,Z.,et al.2002.Estradiol-induced nongenomic calcium signaling regu-lates genotropic signaling in macrophages.J.Biol.Chem.277:7044-7050.3.McCarthy,T.L.,et al.2003.Runx2integrates estrogen activity in osteo-blasts.J.Biol.Chem.278:43121-43129.4.Perissi,V.,et al.2004.A corepressor/coactivator exchange complex required for transcriptional activation by nuclear receptors and other regulated transcription factors.Cell 116:511-526.5.Omoto,Y.,et al.2005.Estrogen receptor αand imprinting of the neonatal mouse ventral prostate by A 102:1484-1489.6.Pedram,A.,et al.2007.A conserved mechanism for steroid receptor translocation to the plasma membrane.J.Biol.Chem.282:22278-22288.7.Klein,C.,et al.2010.Transcriptional profiling of equine endometrium during the time of maternal recognition of pregnancy.Biol.Reprod.83:102-113.ER α(MC-20):sc-542.Western blot analysis of human recombinant ER α.<ERα116K -80K -52K -35K -30K-ER α(MC-20):sc-542.Immunoperoxidase staining of formalin-fixed,paraffin-embedded human breast carcinoma tissue showing nuclear localization.。

心脏正常值

心脏正常值
心脏
(一)二维超声心动图 1.胸骨旁左室长轴切面 正常值:AAO内径:22.4-33.1mm AO窦内径24.0-32.3mm AV环径:18.2-22.1mm RVOT内径21.0-33.0mm(男)
23.0-32.0mm(女) LA前后径26.2-34.2mm MV环收缩末最大径 21.0-34.0mm RVAW厚度3.25-4.52mm RV舒张末前后径 18.9-24.0mm LV舒张末前后径44.8-47.6mm(男)
RV
舒张末内径 17.3-21.5mm
收缩末内径 15.8-19.1mm
IVS
舒张末厚度 6.47-9.50mm
收缩末厚度 9.87-14.5mm
收缩幅度
5.86-9.42
收缩速度
2.48-3.55cm/s
舒张速度
2.90-4.50cm/s
LV 舒张末内径
45.8-49.3mm(男)
43.7-46.4mm(女 )
正常值:E峰峰值:46-79cm/s(成年人) 50-80cm/s(儿童)
A峰峰值:34-50cm/s E/A值: 1.02-1.92 3.左室流出道、主动脉前向彩色多普勒及频谱多普勒 正常值:左室流出道血流峰值: 79-107cm/s 左室流出道血流峰值压差: 2.50-4.79mmHg 主动脉前向血流峰值: 110-134cm/s(成年人 )
二尖瓣环TDI显像 正常值:Ea>Aa Sa波>5cm/s
(五)心功能评估
1.左室收缩功能
正常值:LVESV:(24±10)ml/m2 LVEDV:(70±20) ml/m2
SV:60-120ml FS:34%±5%
EF:67%±8%
2.左室舒张功能

PMDA_ODA型聚酰亚胺制备工艺与聚集态结构的研究进展

PMDA_ODA型聚酰亚胺制备工艺与聚集态结构的研究进展

第23卷第2期高分子材料科学与工程V o l.23,N o .2 2007年3月POL Y M ER M A T ER I AL S SC IEN CE AND EN G I N EER I N GM ar .2007P MDA -ODA 型聚酰亚胺制备工艺与聚集态结构的研究进展Ξ翟 燕1,2,顾 宜1(1.高分子材料工程国家重点实验室,四川大学高分子科学与工程学院,四川成都610065;2.中北大学分校,山西太原030008)摘要:综述了P M DA 2ODA 型聚酰亚胺的制备工艺,并阐述了酰亚胺化工艺对聚酰亚胺聚集态结构的影响。

关键词:均苯四羧酸二酐;4,4’2二氨基二苯醚;聚酰亚胺;聚集态中图分类号:TQ 323.7 文献标识码:A 文章编号:100027555(2007)022******* 聚酰亚胺(P I )是主链上含有酰亚胺环的一类化学结构高度规整的刚性链聚合物[1]。

不同分子链结构的P I 材料(薄膜、纤维、粉末)呈现何种聚集态结构,成型工艺控制起着至关重要的作用。

P I 中最具有代表性的产品是杜邦公司在20世纪60年代初推出的Kap ton 薄膜(PM DA 2ODA 型),它具有优良的机械、电、热性能,被广泛应用于电工、微电子和机械化工等行业;又由于它具有良好的耐辐射性,在航空、航天等尖端技术领域也得到应用。

其突出的综合性能和广泛的应用领域引起很多学者的关注,并对PM DA 2ODA 型P I 做了大量深入研究。

这些研究结果在很大程度上也适用于其它类型的P I ,因此本文概述了PM DA 2ODA 型P I的制备工艺与聚集态结构研究现状,这将对P I的制备具有一定的理论指导意义。

1 P MDA -ODA 型聚酰亚胺的制备1.1 聚酰胺酸的合成将单体均苯四羧酸二酐(PM DA )与4,4’2二氨基二苯醚(ODA )在如N ,N 2二甲基甲酰胺(DM F )、N ,N 2二甲基乙酰胺(DM A C )、N 2甲基吡咯烷酮(NM P )等极性溶剂中缩聚得到聚酰胺酸(PAA )溶液。

Rexroth力士乐电磁阀手册

Rexroth力士乐电磁阀手册

= No code = No code = B08 = B10 = B12
= No code =V Attention! The compatibility of the seals and pressure fluid has to be taken into account!
1)
When connecting to an AC supply a DC solenoid must be used which is controlled via a rectifier (see table below). With an individual connection a large plug-in connector with built-in rectifier can be used (separate order, see page 3). DC solenoids used with an AC supply AC supply (permissible voltage tolerance ± 10%) Nominal voltage of the DC solenoid when used with an AC supply via rectifier 120 V – 60 Hz 110 V 205 V Order detail
Electric Drives and Controls
Hydraulics
Linear Motion and Assembly Technologies
Pneumatics
Service
4/3, 4/2 and 3/2-way directional valves with wet pin DC or AC solenoids
List of contents

汉语拼音《ieueer》课时教案

汉语拼音《ieueer》课时教案

11、ie ǖe er设计理念兴趣是最好的老师。

一年级的小学生好动、注意力无法持久,喜欢听故事、做游戏。

而传统的拼音教学是十分枯燥的,难以吸引的学生的注意力。

因此,本设计根据学生的特点,尝试以生动有趣的故事贯穿其中,猜谜、游戏穿插其间,使课堂充满生活情趣,可以取得事半功倍的效果。

教学目标1.复韵母ie üe,特殊韵母er和整体认读音节ye yue,能读准音,认清形,正确书写,会读它们的四声。

2.学会拼读带有复韵母ie üe的拼音,掌握ü上两点的省略规则。

3.能够看图说话,根据音节拼读词语和句子。

4.能够自己拼读儿歌,做到词语连读。

5.认识“家、飞、机、有、儿、河、入、校”8个生字,并能在一定的语境中使用。

教学重点学会复韵母ie üe,特殊韵母er和整体认读音节ye yue。

教学难点使学生能够区别ie üe和ye yue,掌握ü上两点的省略规则。

课前准备教师准备:教学插图、拼音卡片等课时安排两课时教学过程第一课时课时目标:1.学会复韵母ie üe和整体认读音节ye yue,能读准音,认清形,正确书写,会读它们的四声。

2.学会拼读带有复韵母ie üe er的拼音。

教学过程:一.复习检查,导入新课1.教师:孩子们,咱们在拼音王国里已经认识了不少拼音朋友,猜一猜他是谁(师说一扇小门等,生猜出n l j q x h sh)2.昨天刚认识的复韵母朋友想念你们了。

出示:ai ei ui ao ou iu 认读。

3.有几个整体认读音节家族的朋友好久不见了。

出示i和yi、ü和yu。

让学生读一读。

4.出示①让学生读一读。

②说说ü上两点省略规则③指名认读音节:nü lü和ju qu xu,复习ü上两点省略规则的儿歄。

(小ü见了j q x,去掉两点还念ü。

)④今天我们再学习2个复韵母和一个特殊韵母,谁有信心能学好!真棒!板书:11. ie üe er[设计意图]以充满人性化的语言亲情导入,从复习旧的知识开始,引导小学生在成功复习的基础上树立学习信心,十分得体,十分自然。

关于脑氧饱和度监测下控制性降压对老年高血压患者术后影响研究

关于脑氧饱和度监测下控制性降压对老年高血压患者术后影响研究

关于脑氧饱和度监测下控制性降压对老年高血压患者术后影响研究发布时间:2022-10-14T07:39:08.361Z 来源:《医师在线》2022年14期作者:殷丹丹通讯作者:谢云斌[导读] 目的:探讨脑氧饱和度(rSO2)监测下控制性降压对老年高血压患者术后谵妄(POD)的影响。

殷丹丹通讯作者:谢云斌常州市第二人民医院麻醉科单位邮编:213100常州市第一人民医院麻醉科单位邮编:213000摘要:目的:探讨脑氧饱和度(rSO2)监测下控制性降压对老年高血压患者术后谵妄(POD)的影响。

方法:择期全麻下行鼻泪道手术的老年高血压患者110例,男53例,女57例,年龄65~85岁,ASAⅡ或Ⅲ级,高血压Ⅰ或Ⅱ级,采用随机数字表法分为两组:rSO2监测组(S组)和对照组(C组),每组55例。

术后1、2、3d采用谵妄评定方法中文修订版(CAM-CR量表)对患者进行POD评估。

结果与T0时比较,T1—T2时两组MAP明显下降(P<0.05),C组rSO2明显下降(P<0.05),T3—T4时逐渐回升至术前水平。

T1—T2时S组rSO2明显高于C组(P<0.05),术中rSO2最低值明显高于C组(P<0.05),rSO2较基础值下降的最大百分比明显低于C组(P<0.05)。

术后1dS组POD发生率明显低于C组(P<0.05)。

结论:rSO2监测下控制性降压能减少老年高血压患者鼻泪道手术后谵妄的发生,提高围术期安全性。

关键词:脑氧饱和度;控制性降压;老年;谵妄 1引言鼻内镜操作对视野清晰度要求较高,通常要在术中进行控制性降压以减少鼻腔内出血,但对于老年高血压患者而言,慢性高血压对脑血管自主调节能力产生损害,盲目降压很可能造成脑组织血氧供应不足,从而产生神经损害。

局部脑氧饱和度监测仪可无创、实时地监测脑组织氧供需状态,研究表明术中rSO2降低与患者术后谵妄相关,通过维持rSO2在一定范围可减少术后认知障碍和相关神经损伤的发生。

相关主题
  1. 1、下载文档前请自行甄别文档内容的完整性,平台不提供额外的编辑、内容补充、找答案等附加服务。
  2. 2、"仅部分预览"的文档,不可在线预览部分如存在完整性等问题,可反馈申请退款(可完整预览的文档不适用该条件!)。
  3. 3、如文档侵犯您的权益,请联系客服反馈,我们会尽快为您处理(人工客服工作时间:9:00-18:30)。

101 ERLEMPLOYMENT RELATIONSEssay Topic:Is workplace conflict inevitable?Student Number: 257-4267For the past two decades, there has been a dramatic change in the Australian workplace relations; in particular, constant changes to the employment regulations between employers and employees have taken place over the years. These changes have brought about a number of different views and strategies on how different companies should be run with regard to employee relations throughout many industries (Nankervis and Lewing, 2004). In Rasmussen and Lamm (2002), the author contrasted two main approaches to workplace relations within organisations; unitarist and pluralist, these approaches reflect the different employee relations and human resource management strategies being applied. A unitarist believes organisations should have an integrated group, a simple authority and a common set of goals and values shared between employees and management, and regard unions as unnecessary as conflicts can be eliminated internally. On the other hand, a pluralist sees conflict as rational and inevitable, resulting from industrial and organisational factors, along with differing managerial and employee groups. Trade Unions are well accepted in this approach and conflict is contained via collective bargaining, conciliation and arbitration (Rasmussen and Lamm, 2002) and (Nankervis and Lewing, 2004).The purposes of this paper is to critically discuss and evaluate the practise of employment relations within organisations, and provide materials to support the argument that conflict is inevitable in the employment relationship. In reaching this conclusive statement, we will begin by looking at the definition of conflict. Following this we will analyse the pros and cons of conflict and the factors that may influence it in our working environment.Sing (1998) describes conflict as the clashes of ideas, attitudes and goals between individuals and groups. He further suggested that this is usually present in the workplace whenever people from diverse groups work together, Ramsey (2003) also supports this claim and believes it is a result of misunderstanding and agreements.There are many situations in which conflict can arise in employment relations: conflict can occur when the stakeholders’ objectives are different. Stakeholders are groups of people who has an interest in business activities, these can include: owners, customers, employees, suppliers and government bodies (Mctaggart and Knotes, 2005). Each type of stakeholder is likely to have a set of goals they want to achieve. For example, shareholders want regular and high returns and will attempt to ensure managers undertake decisions to maximise stock prices (Jones and Netter, 2002); employees want jobs and secure employment (Smith, 2004). Both these stakeholders have different objectives and maybe difficult to fulfil. Conflicts can exist between many different groups of stakeholders during decision making, since they have varied goals (Sing 1998). For instance, there are times where issues between owners and managers can occur; the management team may take advantage of their position by seeking their own interests rather than those of the owners, this can involve paying themselves a large paycheck or organising time to suit their own needs and only achieving satisfactory levels of profits rather than high levels of profits (Vaknin, n.d.).Another situation where conflict can occur is between the employees and the owners or managers of a business, this usually takes place when there are disagreements on such things as the levels of pay, working condition and changing practices (Mctaggart & Knotes 2005; Managing and Resolving Conflict n.d.; Coan 2004). This is because workers generally want more than what owners are prepared to pay. However, money does not always provide happiness to the employees, they want to be valued and appreciated within their organisation. Being recognised and having a sense of purpose and importance means a lot to people (Haas, 2001). Conversely, managers prefer to maximise profits by minimising labour costs either by a way of lowering wages or laying-off workers, and to increase the intensity of work effort by the employee in the firm.In addition to the employment conflicts mentioned above, there are other issues relating to conflict within the working environment, discrimination. This is one of the largest problem facing our world today and occurs everyday not just in the workplace but everywhere. There are many types of discrimination, Time For Equality At Work (2003) gave extensive examples of workplace discrimination: when workers are rejected or granted positions of employment because of their appearance or when a competent woman manager is paid less than a male colleague with equal productivity, these are regarded as discrimination. There are many forms of discrimination, it can range from differences in sexual preferences or religion to where a woman having to undergo a pregnancy test inorder to be considered for a job.Conflict in the workforce is quite common and often not dealt with openly within organisations; this may be due to the fact that it is hidden and not easily seen by management. However, in saying this, management sometimes fail to act to resolve conflict even when it is visible. One explanation of this is that management really only cares about the operations of the business and its success, not the driving force behind the business which is its people (Haas, 2001).The facts provided above, clearly suggests that these types conflict in the workplace pose a serious problem for the whole environment within organisations. It is therefore vital to deal with the problem quickly and most effectively, once discovered. Failing to do so can lead employees to have no motivation to work and as a result, the company may experience decreased productivity and a lower level of revenue and profit (Fiore, 2004). In a research article developed by Jordan (2005), an stonishing result involving Irish workplaces showed employees failing to turn up to work as a result of stress, this outcome s trongly supports Foire’s claim.Often conflicts within organisations can be resolved internally as suggested by the unitarist approach. However, sometimes serious personality clashes can occur; this can affect teamwork building between employer and employee and can lead to less productivity and effectiveness. Upon this, if workers constantly are being pressured to work harder and produce more, tension will rise (Ramsey, 2003).There is a need in most workplaces to improve the communication skills between the employer and the employee. If this kind of work cooperation is being ignored, the consequences can be expensive for the organisation. Unresolved stress and tension in a workplace can lead to more conflicts (Haas, 2001).Although negative results are likely to occur from conflict there are still many positive consequences that can emerge. Conflict can be seen as a productive force, by allowing individual members organisations to increase their knowledge and skills, by sharing and contributing ideas to the organisation and its members. This view of conflict provides people with feedback for any potential problems. By adapting to this view of conflict and recognising that each conflicting situation provides opportunity to improve, there would be a more positive approach to conflict and so rather than trying to eliminate it, the task of managing conflict would take its place and would result in better developed people and a stronger organisation (Bacal, 2004).In contrast to Bacal’s suggestion that conflict management leads to greater productivity, solving and managing conflicts can be expensive and time consuming for companies. Every year, thousands of dollars are spent on legal fees to resolve disputes with employees, clients and business partners (Reade, 2004). However, there are other alternatives to cut costs when handling conflicts and that is through the use of integrated conflict management systems (Lewis & Sacks, 2001). Conflict Management Systems deals with paying attention to conflict signals, ignoring it can worsen the situation. It teaches how to deal with various situations such as: If an issue arises, deal directly withthat person and to be open to suggestions by others, which is often not always followed by management because they do not like their authority to be questioned.Another way of resolving conflict problem in the workplace is through collective bargaining. It involves determining conditions of work and terms of employment through negotiations between employees and employers representatives, such as trade unions. The representative body has more strength and influence and can negotiate for its members since, if many individuals with collective interests, come together as a group, they have a much bigger influence towards the employers. The primary goal for the unions is to protect the economic conditions for the employees, and to advance their wages. Without such a bargaining process, employers and managers would be able to set wages and conditions without taking into account employees’ interests. The result of collective bargaining is a collective agreement. These agreements are usually written and are signed by the parties and will be binding. Agreements include pay, work conditions, fringe benefits and also redundancy, dismissal and promotion procedures. The advantages are that it may encourage worker to work more effectively as they think that their opinions might be accepted. Also it helps to improve the relationship between owners and employees because they do not have to argue independently. The representative body can help them to solve the problems together ( Silva, 1996).To conclude, conflict is inevitable and an essential ingredient for the management process, it cannot be avoided and ignored. It may impact organisations and its members in a negative way, but this negative effect can help build positive ones, and as a result, may create more growth within organisations. In an article by Pilgram (2005), the author also supports this statement and added that, if dealt with effectively it will lead to greater productivity among employees, hence growth. Conflict is therefore not regarded as evil. It is a natural process and provides the opportunity to explore and resolve differences in a constructive manner. Furthermore, it enables managers to create a more efficient workforce, bring in new ideas and constructive suggestions to the business and therefore stimulates business growth.ReferencesFiore, T., (2004). Resolving Workplace Conflict: 4 Ways to a Win-Win Solution, Attard Communications Inc. Retrieved 30 August, 2005, from /manage/resolve.htm Bacal, R., (2004). Organisational Conflict – The Good, the Bad and the Ugly, The Journal for Quality and Participation. Retrieved 30 August, 2005, from /p/articles/mi_qa3616/is_200407/ai_n9425843Coan, G., (2004). Managing Workplace Conflicts. Retrieved 30 August, 2005, from/guestconflict/manworkplaceconflict.htmHart, R., (2004). The Mind-Body Connection: Workplace Conflict, Stress & the Risk of Injury, The Authority On Occupational Safety, Health and Loss Prevention. Retrieved 30 August, 2005, from/articles/12168Haas, T.D., (2001), ‘Conflict? What an Opportunity’, Charter, vol. 72, no. 8, pp. 50. Retrieved, 30 August, 2005, from ProQuest 5000.Jones, S.L., & Netter, J.M., (2002). Efficient Capital Markets. Retrieved 30 August, 2005, from /library/Enc/EfficientCapitalMarkets.htmlJordan, A., (2005). Report Stresses Need For Happy Workers, Irish Echo Newspaper Corp. Retrieved 30 August, 2005, from /newspaper/ story.cfm?id=16928Lewis, A., & Sacks, M., (2001), ‘Turning disputes to corporate advantage Arguments between staff can result in costly litigation. However, Michael Sacks and Andy Lewis believe a resolution systemcan turn conflict into a potentially creative force’, Financial Times, Iss. Nov 26 , p. 02. Retrieved,30 August, 2005, from ABI/INFORM Global.McTaggart, J.M., & Knotes, P.W., (2005), The Governing Corporate Objective: Shareholders Versus Stakeholders. Retrieved 30 August, 2005, from /ideas_pdf/id_930601_kontes.pdfNankervis, A., & Lewing J. (2004). Employment Relations, 4th Edition Publish: Thompson.Pilgram, S., (2005). An Essential Ingredient For Growth, Pertinent Information Ltd.Retrieved 30 August, 2005, from /articles/communication/spilgrim4.aspRamsey, R.D., (2003), ‘Peacekeeping in the Workplace: How to Handle Personality Clashes Among Employees’, Supervision, vol. 64, no. 5, pp. 14. Retrieved, 30 August, 2005, from ABI/INFORM Global.Rasmussen, E., & Lamm E. (2002). An Introduction to Employment Relations in New Zealand.Auckland: Pearson Education.Reade, Q., (2004). Workplace Conflict is Time-Consuming Problem for Business, Personnel Today.Retrieved 30 August, 2005, from /Articles/2004/09/30/25840/Workplace+conflict+is+time-consuming+problem+for+business.htmSilva, S.D., (1996). Collective Bargaining Negotiations, International Labor Organisation.Retrieved 30 August, 2005, from /public/ english/dialogue/actemp/ papers/1998/srscbarg.htm Sing, H., (1998), ‘The Management of Conflict at Workplace’, New Strait Times (Malaysia), Access no.73431101842. Retrieved 30 August, 2005 from Business Source Premier.Smith, D., (2004). Values vs Values: The Organisational Split, Informit Network. Retrieved 30 August, 2005, from /articles/article.asp?p=170966&rl=1Time for Quality at Work (2003). Retrieved 30 August, 2005, from /dyn/declaris/ DECLARATIONWEB.DOWNLOAD_BLOB?Var_DocumentID=1558Vaknin, S., (n.d). Making Your Workers Your Partners.Retrieved 30 August, 2005, from/nm038.html。

相关文档
最新文档