西内111
西内口诀
46 、房颤发生后易引起哪种合并症
体循环动脉栓塞
47 、二尖瓣狭窄早期大咯血的原因是
支气管静脉破裂
48 、风心病二尖瓣狭窄发生房颤后，常见的并发症是
动脉栓塞
49 、哪种心脏病，不宜使用血管扩张剂
心包填塞征
50 、二尖瓣狭窄合并房颤，心室率 120 次 / 分，首选治疗是
结肠癌
结肠癌，经常见，分为左半和右半。右半结肠管腔粗，血液循环最丰富。贫血消瘦常出现，腹部肿块为常见。左半结肠管腔细，产生梗阻最容易。便秘腹泻常交替，便血为主来求医。
直肠癌
直肠癌脓血便，大便性状有改变。里急后重较典型，大便变细为特征。
肝癌
肝癌早期难诊断，症状体征不明显。腹胀乏力体重减，右上腹痛肿块现。高低不平质地硬，增大迅速为特征。腹水消瘦加黄疸，肝癌完全可诊断。
10、心梗的并发症：
心梗并发五种症 动脉栓塞心室膨 乳头断裂心脏破 梗塞后期综合症
11、心梗与其他疾病的鉴别
痛哭流涕、肺腑之言
痛——心绞痛；流——主动脉瘤夹层分离；腑——急腹症；肺——急性肺动脉栓塞；言——急性心包炎。
12、主动脉瓣狭窄的表现： 难、痛、晕
13、二尖瓣狭窄
劳力性呼吸困难
25 、诊断急性肺水肿，最有特征意义的表现是
严重呼吸困难伴粉红色泡沫痰
26 、诊断右心功能不全时，最可靠的体征是
颈静脉怒张，肝颈静脉回流征阳性
27 、呼吸困难最常见于
左心功能不全
28 、哪种情况产生急性肺水肿时，宜用吗啡
急性心肌梗死伴持续性疼痛
29 、心源性哮喘与支气管哮喘主要不同点是
甲亢症，很特殊，眼睛大，脖子粗。烦热多汗夜失眠，情绪波动手振颤。脉搏增快心里慌，高压高来低压降。食欲亢进体重减，停经脱发常出现。
Omron K8AB-TH温度监测传感器启动指南说明书
WiringPrecautions for Safe Use1)The product is designed for indoor use only. Do not use the product outdoors•or in any of the following locations.E Places directly subject to heat radiated from heating equipment.E Places subject to splashing liquid or oil atmosphere.E Places subject to direct sunlight. Places subject to icing and condensation. E Places subject to dust or corrosive gas (in particular, sulfide gas andammonia gas).E Places subject to intense temperature change.E Places subject to vibration and large shocks.2)Use/store within the rated temperature and humidity ranges.Provide forcedcooling if required.3)Install K8AB in a correct direction.4)Be sure to wire properly with correct polarity of terminals.5)Wire the input and output terminals correctly.6)Use this product within the rated load and power supply.7)Be sure that the temperature sensor type and the input type set on K8AB arethe same.8)When extending the lead wires on a thermocouple, be sure to use compensating conductors suitable for the thermocouple type.9)When extending the lead wires on a platinum resistance thermometer, uselead wires with a low resistance (5 ƒ max. per line) and be sure that the resistance is the same for all three lead wires.10)Use the recommended solderless terminals.11)Do not wire the terminals which are not used.12)Make sure that the rated voltage is attained within 1 seconds13)Design system (control panel, etc) considering the 1 second of delay thatK8AB's output to be determined after power ON.14)Make sure that K8AB has 30 minutes or more to warm up after power ON.Turning ON the power before starting monitor to the correct temperature.15)Separate the high-voltage or large-current power lines from other lines, andavoid parallel or common wiring with the power lines when you are wiring to the terminals.16)Allow as much space as possible between K8AB and devices that generatepowerful high frequencies or surge.17)Do not use a microwave receiver near K8AB. Microwave interference mayaffect K8AB.18) A switch or circuit breaker should be provided close to this unit. The switch orcircuit breaker should be within easy reach of the operator, and must be marked as a disconnecting means for this unit.19)Do not use paint thinner or similar chemical to clean with. Use standard gradealcohol.20)Use tools when dismantling parts for disposal.21)Install the K8AB inside a cabinet.22) This is a class A product. In residential area‚ it may cause radio interference,in which case the user may be required to take adequate measures to reduce interference.SpecificationsPower supply voltage Operating frequency Operating valtage rangeOperating frequency rangePower consumption Inrus current Accuracy Relay outputAmbient temperatureAmbient humidity Strage temperature Strage humidity WeightDegree of protection Altitude Installation environmentExternal input Output currentMemory protectionAC100-240V type AC/DC24V type 50-60Hz85 to 110% of the rated voltage 95 to 105% of the rated frequency5VA max.(AC100-240V)4VA max.(AC24V)2W max.(DC24V)15A max.(AC100-240V)10A max.(AC/DC24V)2%FS3A,250VAC/30VDC(resisteve load)1A,250VAC/30VDC(inductive load)Mechanical life 10million times Electorical life 50,000times(N.O) 30,000times(N.C)1c contact -10 to 55•(Avoide freezing or condensation)RH 25 to 85%-25 to 65•RH 25 to 85%Approx.130g IP20Max 2,000mSetup category ‡U ,pollutionDegree 2(as per EN61010-1)Output current:approx.10mAContact input ON :1kƒ max., OFF:100kƒ min.No-contact input ON :residual voltage (NPN) 1.5Vmax.,OFF:leakage current 0.1mA min. EEPROM(non-volatile memory)(endurance:200,000 erase/write cycles)Dimensions (mm)Names of partsSetting rangeSensor input rangeSwitch OperationTime ChartMountingDismountingConnectionsTH11STH12SSolderless terminals Use the small screw driver when you operate the SWs or the Button.OMRON ELECTRONICS LLC.One Commerce Drive Schaumburg, IL 60173-5302 U.S.A Phone 1-847-843-7900 FAX 1-847-843-7787OMRON CANADA INC. 885 Milner Avenue Scarborough, Ontario M1B 5V8, Canada Phone 1-416-286-6465 Fax 1-416-286-6648OMRON EUROPE B.V.Wegalaan 67-69 2132 JD Hoofddorp The Netherlands Phone 31-23-56-81-300 FAX 31-23-56-81-388OMRON ELECTRONICS PTY. LTD. 71 Epping Road, North Ryde, Sydney, N.S.W 2113, Australia Phone 61-2-9878-6377 Fax 61-2-9878-6981OMRON ASIA PACIFIC PTE. LTD.No.438A Alexandra Road #05-05/08(Lobby 2), Alexandra Technopark, Singapore 119967Phone 65-6835-3011 FAX 65-6835-2711OMRON CORPORATIONShiokoji Horikawa, Shimogyo-ku, KYOTO, 600-8530 Japan Phone 81-75-344-7109 FAX 81-75-344-7149Alarm type:Hysteresis 2•(or ßF )Non-failsafe/Failsafe Latch:Holding the relay output until •g latch reset”SV protect mode: In this mode, the changes of SW or R_SWs are • • NOT available. When K8AB comes out from this • •mode, the changes are available.Behavior of LEDs Relay(Failsafe)Relay(Non-failsafe)Alarm, Other errs Source Relay(Latch)Alarm, Other errsSourceSV protect modeSourcePWR_LED Sv protect modeSourceALM_LEDAlarm Source Other errs Lower-limit alarm Upper-limit alarm SV* Other errs:Sensor burnout,Input range over,Setting range over, memory err.* If LATCH_RST or EXTERNAL INPUT has been ON more than 5s, K8AB-TH goes into or comes out from SV protect mode.SWErrors(ALM_LED:flash)Comes out of SV protect mode.A Reset the latch.B Confirm the wiring and parameter settings.C Reset the SOUCE.If K8AB return to normal state, the cause may be the • noise.If not, there is need to replace it.The state of latched output and the state of SV • protect mode are backed up by EEPROM.Trouble shootingFollowing (1)•(3) may occur.(1)Sensor burn out or Senor input range over. (2)Setting range over.(3)Inner error(devices,memories,etc.).O NOF FF O N F OF FF O N F OF F。
天津法租界
编号法租界时期中文路名法租界时期法文路名收回后路名现时路名现时路段一号路海大道Rue de Takou 大沽路大沽北路张自忠路至营口道二号路七月十四日路Rue du 14 Juillet 长春道长春道大沽北路至解放北路三号路石教士路Rue Chevrier 黑龙江路黑龙江路长春道至哈尔滨道四号路葛公使路Rue Paron Gros 滨江道滨江道大沽北路至张自忠路，现解放北路至张自忠路段已不存在。
五号路巴黎路Rue de Paris 吉林路吉林路张自忠路至营口道六号路杜麦路狄总领事路Rue Paul DoumerRue Dillon哈尔滨道丹东路哈尔滨道大沽北路至和平路，现已不存在。
大沽北路至张自忠路，现解放北路至张自忠路段已不存在。
七号路大法国路Rue de France 中正路解放北路张自忠路至营口道八号路巴斯德路水师营路Rue PasteurRue de L'Amirauté赤峰道赤峰道解放北路至和平路张自忠路至解放北路九号路古拔路Rue Courbet 松江路松江原张自忠路至滨江道，现已不存在。
路十号路领事馆路Rue du Consulat 承德道承德道张自忠路至解放北路十一号路宝总领事路Rue HenryBourgeois合江路合江路原张自忠路至赤峰道，现已不存在。
十二号路圣鲁易路Rue Saint Louis 营口道营口道张自忠路至南京路十三号路克莱蒙梭广场PlaceClemenceau承德道承德道解放北路至吉林路十四号路威尔顿路Rue de Verdun 承德道承德道吉林路至花园路十五号路柏公使河坝Quai AugusteBoppe张自忠路张自忠路大沽北路至解放北路十六号路十七号路十八号路十九号路大法国河坝Quai de France张自忠路张自忠路台儿庄路解放北路至营口道二十号路拉大夫路Rue Laville哈尔滨道哈尔滨道大沽北路至和平路二十一号路杜总领事路Rue du Chaylard罗斯福路和平路锦州道至营口道二十二号路泰伟路Rue Tréve 菜市街菜市街大沽北路至锦州道，现已不存在。
醇还原胺化反应催化剂研究进展
CHEMICAL INDUSTRY AND ENGINEERING PROGRESS 2018年第37卷第10期·3832·化 工 进展醇还原胺化反应催化剂研究进展余秦伟1，2，惠丰1，2，张前1，2，袁俊1，2，王为强1，2，赵锋伟1，2，杨建明1，2，吕剑1，2（1西安近代化学研究所，陕西 西安 710065；2氟氮化工资源高效开发与利用国家重点实验室，陕西 西安 710065）摘要：醇还原胺化反应是胺合成最有效、最有应用潜力的方法之一，而催化剂是还原胺化反应的关键。
本文详细阐述了Ru 、Ir 、Pd 、Cu 、非金属等均相催化剂和Co 、Ni 、Ru 、Pd 等非均相催化剂在醇还原胺化反应中的研究进展，介绍了不同催化体系的催化性能和反应规律、应用特点和局限性。
指出了均相催化体系的回收使用仍然是阻碍其应用的难题，研究重点应集中在高效、廉价催化体系开发、拓展应用范围和分离回收研究；非均相反应催化剂的专用性强，性能难以满足工业应用需求，加强微观结构及反应机理、高性能催化剂、高压体系中流场状态与过程研究以及提高活性、选择性和稳定性是未来的研究重点。
关键词：醇；还原胺化；催化剂；胺；合成中图分类号：O69 文献标志码：A 文章编号：1000–6613（2018）10–3832–11 DOI ：10.16085/j.issn.1000-6613.2017-2302Progress in the catalyst for reductive amination of alcoholYU Qinwei 1,2, HUI Feng 1,2, ZHANG Qian 1,2, YUAN Jun 1,2, WANG Weiqiang 1,2, ZHAO Fengwei 1,2,YANG Jianming 1,2, LÜ Jian 1,2（1Xi’an Modern Chemistry Research Institute, Xi’an 710065, Shaanxi, China; 2State Key Laboratory of Fluorine &Nitrogen Chemicals, Xi’an 710065, Shaanxi, China ）Abstract ：Reductive amination of alcohol is one of the most effective and promising methods for thesynthesis of amine, and the high-performance catalyst is the core. The progress in homogeneous catalysts of Ru, Ir, Pd, Cu, and nonmetallic ones and heterogeneous catalysts of Co, Ni, Ru, Pd, et al. for reductive amination of alcohols was outlined in detail respectively in this paper. The catalytic performance and reaction regularity of different catalytic systems were discussed together with their respective characteristics and limitations. Finally, it is pointed out that the application of homogeneous catalytic system is limited by the recovery problem. The research should focus on the development of efficient and cheap catalytic systems, the expansion of their applications and the recovery of the catalyst. The heterogeneous reaction catalysts are of high specificity and the performance is difficult to meet the need of industrial application. The studies on the microstructure and the reaction mechanism of high performance catalyst, flow field state and process of high pressure system should be strengthened to improve the activity, selectivity and stability of the catalysts. Key words: alcohol; reductive amination; catalyst; amine; synthesis应用催化研究。
科尼卡自动风卷器F - 科尼卡FC-1系统说明书
143KN980
'Specifications subject to change Without notice
Konica Division BERKEY MARKETING COMPANIES
Frame Speed . .. .. .. .. .. ..... ... 1.5 frames Байду номын сангаасer second
Shutter Speed Coupling Range ... . ... All shutter speeds 2 seconds to 1/1000 second.
Operation Indicator . .... . . . .. ... .. LED lights when film is being advanced.
Key Features
• Provides fully automatic 10ac1lng for the Konica FC-1 System. Drop the film cass~te into the Konlca FC-1, extend the leader and clo$8 the back. The Konlca Autowinder will automatically advance the film to frame NO.1.
End of Film Indicator . . ...... ........ LED remains on to indicate end of roll.
Film Loading Automatically advances film to frame NO.1 .
全国高速公路一览表
京银线（八达岭高速)
G110
1996
马甸
昌平西关
31
31
京哈线
G102
1994。08
北关
燕郊
12
12
12489
15310
京广路
G106
2001。06
玉泉营
榆垡
36
18
18
17857
20769
六环路
S002
2001.6
京沈路
黄村路
38
38
3683
5350
五环路
S001
2001。9
G110路
机场路
335
2
京福线(京沪高速）
G020
2000.12
河北北京界
吴桥宋门乡
148
148
5850
9322
丹拉线(京沈高速）
G025
1999。9
河北辽宁界
河北北京界
221
221
5422
7748
丹拉线
G025
1995.9
宣化小幔岭
宣化下八里
11
11
9931
16771
唐津支线（唐津高速)
G025支线
1999.11
沈桃连接线
S108
1988.09
沈阳市
沈阳市
5
4
1
15349
19916
沈桃连接线
S109
1990.08
沈阳市
沈阳市
5
5
13336
22852
吉林
382
382
20
同三线（长四高速)
G010
美国各州简称及区号
major cities: Chicago suburbs
773
major city: Chicago
815
major cities: Joliet, La Salle, Rockford
847
major cities: Chicago, Evanston, Waukegan
Indiana
印第安那
British Columbia and Northwest Territories
604
major city: Vancouver
BritishVirgin Islands
284
all locations
-C-
California
加利福尼亞
209
major cities: Fresno, Modesto
Grenada
473
all locations
Guam
671
all locations
GU
-H-
Hawaii
夏威夷
808
all locations
HI
-I-
Idaho
愛達荷
208
Idaho all locations
ID
Illinois
伊利諾
217
major cities: Champaign-Urbana, Springfield
-A-
Alabama
阿拉巴馬
205
major cities: Birmingham, Huntsville
AL
334
major cities: Montgomery, Mobile
256
major cities: Anniston, Gadsden, Huntsville, Florence
PROMAFOUR 自凝钙硅酸板产品说明书
Supersedes : 0 / 0 / 01.1. Product identifierTrade name:PROMAFOUR®Identification of the product:Self-supporting calcium silicate board.Type of product:Article.1.2. Relevant identified uses of the substance, mixture or article and uses advised against Use:High temperature insulation.1.3. Details of the supplier of the safety data sheetCompany identification:Promat UK LtdThe Sterling CentreEastern Road - BerkshireRG12 2TD Bracknell UNITED KINGDOMTel.: +44 (0) 1344 381400Fax: +44 (0) 1344 3814011.4. Emergency telephone numberEmergency phone nr:+44 (0) 1344 381 400Symptoms relating to use:For the installed product in its final application: no hazards known.During machining the product (drilling, cutting, sanding, etc.), airborne dust can bereleased.- Inhalation:As with most types of nuisance dust, excessive inhalation of dust may causeirritation of the bronchial tubes.The handling and machining of this product may lead to the release of quartzcontaining dust. The inhalation of dust containing quartz, in particular the fine (respirable) dust fraction, in high concentrations or over a prolonged period of timemay lead to lung disease (silicosis) and an increased risk of lung cancer.Long term hazards: see section 11.- Skin contact:Prolonged skin contact may lead to skin irritation for sensitive persons.- Eye contact:Eye contact with dust may lead to transient eye irritation or inflammation.- Ingestion:Not expected to present a significant ingestion hazard under anticipated conditionsof normal use.2.1. Classification of the substance or mixtureClassification EC 67/548 or EC 1999/45This product is classified as not hazardous.Hazard Class and Category Code Regulation EC 1272/2008 (CLP)This product is classified as not hazardous.2.2. Label elementsLabelling EC 67/548 or EC 1999/45Supersedes : 0 / 0 / 0No labelling required.Labelling Regulation EC 1272/2008 (CLP)No labelling required.2.3. Other hazardsNone under normal conditions.Components:This article is classified as not hazardous.This product is a manufactured article, not a substance nor a preparation.Substance name Contents CAS No EC No Annex No Ref REACH ClassificationWollastonite:13983-17-0237-772-5----------Not classified. (DSD/DPD)----------------------------------Not classified. (GHS)Cellulose fibers:65996-61-4265-995-8----------Not classified. (DSD/DPD)----------------------------------Undefined. (GHS)Quartz (SiO2):14808-60-7238-878-4----------Not classified. (DSD/DPD)----------------------------------Not classified. (GHS)Crystalline calcium silicate hydrate:1344-95-2215-710-8----------Not classified. (DSD/DPD)----------------------------------Undefined. (GHS)Vermiculite:1318-00-9---------------Not classified. (DSD/DPD)----------------------------------Not classified. (GHS)4.1. Description of first aid measuresFirst aid measures- Inhalation:Remove to fresh air and drink water.- Skin contact:Rinse the skin with water.- Eye contact:Do not rub the eye. Rinse the eye out with plenty of clean water for at least 15minutes. If eye irritation or inflammation persists, seek medical advice.- Ingestion:Drink water.4.2. Most important symptoms and effects, both acute and delayedSee sections 7 and 8.4.3. Indication of any immediate medical attention and special treatment neededNo data available.5.1. Extinguishing mediaSuitable extinguishing media:All extinguishing media can be used.5.2. Special hazards arising from the substance or mixtureSupersedes : 0 / 0 / 0Nothing to report.5.3. Advice for fire-fightersFlammable class:The product is non-combustible.Protection against fire:Do not enter fire area without proper protective equipment, including respiratoryprotection.6.1. Personal precautions, protective equipment and emergency proceduresPersonal precautions:Minimize generation of dust. Avoid breathing dusts. Avoid eye and skin contact.Use recommended respiratory protection.6.2. Environmental precautionsEnvironmental precautions:Prevent spread of dust.6.3. Methods and material for containment and cleaning upMethods and material for containment:Use closed containers to avoid dust release.Clean up methods:Shovel up small pieces. Dampen down any dust before putting into appropriateskips.6.4. Reference to other sectionsSee sections 7 and 8.7.1. Precautions for safe handlingPersonal protection:Dust, generated during machining and processing must be exhausted and theregulatory occupational exposure limits (workplace exposure limits in UK) for totaland respirable dust and respirable quartz dust must be respected.Use always respiratory protective equipment when exposures are likely or can beforeseen to exceed the Occupational Exposure Limits or Workplace ExposureLimits in UK (refer to local regulations).Collect dust with a vacuum cleaner or soak with water before sweeping up. Technical protective measures:Work in a well ventilated areaUse tools with appropriate dust exhaust equipment.7.2. Conditions for safe storage, including any incompatibilitiesStorage:Store in dry, covered and frost proof area.7.3. Specific end use(s)Fire protection in buildings. High temperature insulation.Supersedes : 0 / 0 / 08.1 Control parametersOccupational Exposure Limits:When machining boards (drilling, cutting, sanding, etc.), respect Occupational (UK: Workplace Exposure Limits)Exposure Limits (OEL) or Workplace Exposure Limits (WEL in the UK) for inhalableand respirable dust and for respirable quartz dust.Check the latest Occupational Exposure Limits (OEL) or Workplace ExposureLimits (WEL in the UK) for airborne contaminants that are applicable in yourcountry.Typical Occupational Exposure Limits or Workplace Exposure Limits in the UK (8hrs TWA) and Ireland on the date of issue of this document are:Control parameters for airborn:- Quartz dust (CAS number: 14808 - 60 - 7):contaminants - Respirable: 0.1 mg/m³(UK) - 0.05 mg/m³ (IE)- Particles not otherwise classified or regulated (nuisance dust)- Inhalable: 10 mg/m³.- Respirable: 4 mg/m³.8.2 Occupational exposure controls8.2.1 General protection controls- Industrial hygiene:Ensure vacuum dust exhaust with correct filter when using motorised machiningtools.8.2.2. Individual protection controls- Respiratory protection:Avoid breathing dusts.Use appropriate respiratory equipment when exposures are likely or can beforeseen to exceed the Occupational Exposure Limits or Workplace ExposureLimits for the UK (e.g. for exposures up to 10 times the OEL (WEL) use at least aP2 type duct mask. For higher exposure, use a P3 type mask).- Skin protection:Avoid contact with skin.Use working clothes and gloves to protect against mechanical injury and direct skincontact.- Eye protection:Avoid contact with eyes.Use safety glasses whenever tools are used and dusts are produced.- Ingestion:When using, do not eat, drink or smoke.• Appearance:Board (solid)• Physical state at 20 °C:Solid.• Colour:White-beige• Odour:None.• pH value:10 - 11• Flammability:Non flammable.• Density:ca. 950 kg/m3• Solubility in water:Insoluble.• Other properties:Information on other physical and chemical properties, as listed in the section 9.1 ofAnnex II of the Commission Regulation EU 453/2010 of 20 May 2010 is notavailable10.1. ReactivitySupersedes : 0 / 0 / 0Stability and reactivity:Stable under normal conditions.10.2. Chemical stabilityChemical stability:Stable under normal conditions of storage, handling and use.10.3. Possibility of hazardous reactionsHazardous reactions:None.Hazardous properties:None10.4. Conditions to avoidConditions to avoid:None known.10.5. Incompatible materialsMaterials to avoid:Strong acids.10.6. Hazardous decomposition productsNone known.11.1. Information on toxicological effectsToxicity information:No data available.Acute toxicity:No acute toxicity has been reported, apart from some exceptional cases oftransient eye irritation or inflammation, skin irritation or irritation of the mucosae (throat, bronchial tubes) by excessive exposure to dust.11.2 Additional information:• On product:The inhalation of quartz containing dust, in particular the fine dust fraction(respirable size), in high concentrations or over repeated or prolonged periods oftime can be hazardous to health and may lead to chronic lung disease and anincreased risk of lung cancer. This risk will be minimal if correct working practicesare observed and applied. (Refer to Section 8). However, for this product, withexposure assessments performed by accredited European laboratories usingreference workplace monitoring methods, any quartz levels in the respirable dustwere below the detection limit.According to the International Agency for Research on Cancer (IARC MonographVolume 100C - 2012) “Crystalline silica inhaled in the form of quartz or cristobaliteis carcinogenic to humans(Group 1).”12.1. ToxicityNo known effects.12.2. Persistence - degradabilitySupersedes : 0 / 0 / 0No data available.12.3. Bioaccumulative potentialNo data available.12.4. Mobility in soilNo data available.12.5. Results of PBT and vPvB assessmentNo data available.12.6. Other adverse effectsNo information available.Ecological effects information:No data available.13.1. Waste treatment methodsHandle as construction industry waste.13.2. GeneralProduct disposal:Dispose in a safe manner in accordance with local/national regulations. Packaging disposal:Dispose according to local legislation.EWC (European Waste Catalogue) -:170107N°.General information:Not classified as dangerous in the meaning of transport regulations.Symbol(s):None.R Phrase(s):None.S Phrase(s):None.Further information:None.DISCLAIMER OF LIABILITYThe information in this SDS was obtained from sources which we believe are reliable. However, the information is provided without any warranty, express or implied, regarding its correctness. The conditions or methods of handling, storage, use or disposal of the product are beyond our control and may be beyond our knowledge. For this and other reasons, we do not assume responsibility and expressly disclaim liability for loss, damage or expense arising out of or in any way connected with the handling, storage, use or disposal of the product. This SDS was prepared and is to be used only for this product. If the product is used as a component in another product, this SDS information may not be applicable.This data sheet and the information it contains is not intended to supersede any terms or conditions of sale and does notSupersedes : 0 / 0 / 0constitute a specification. Nothing contained herein is to be construed as a recommendation for use in violation of any patent or applicable laws or regulations.The contents and format of this SDS are in accordance with REGULATION (EC) No 1907/2006 OF THE EUROPEAN PARLIAMENT AND OF THE COUNCILEnd of document。
Epson项目机用户指南.pdf_1701921423.574341说明书
Quick SetupIMPORTANT: Before using this product, make sure you read these instructions and the safety instructions in the online User’s Guide .ContentsMake sure your projector box includes all of these parts:Connect the projectorChoose from the following connections. See the sections below or the online User’s Guide for details.ComputerUSB cable HDMI cableVGA cableHDMI portConnect one end of an HDMI cable to the projector’s HDMI port and the other end to an HDMI port on your computer.Computer portComputer port and theUSB portConnect the square end of a USB cable to the projector’s USB-B (square) port. Connect the flat end of the cable to any USB port on your computer.Windows Vista ® or later: After turning on the projector, follow the on-screen instructions to install the Epson ® USB Display software (EMP_UDSE.EXE ; only on first connection). If the software screen does not display automatically, open My Computer , Computer , or This PC , then double-click EPSON_PJ_UD .OS X 10.7.x or higher: After turning on the projector, the setup folder for USB Display appears in the Finder. Double-click USB Display Installer and follow the on-screen instructions to install the Epson USB Display software (only on first connection).Video deviceConnect one end of an HDMI cable to the projector’s HDMI port and the otherend to an HDMI port on your video device.Turn on your equipment1T urn on your computer or video source.2P lug in the projector. Thepower light on the projector turns blue.3P ress the power button on the projector or remote control. Theprojector beeps, the Status light flashes blue, and then stays on.Note: To shut down the projector, press the power button twice.4U se the arrow buttons on the remote control to highlight any of theoptions on the Home screen that appears, then press to select it.You can switch between projection sources and quickly access various adjustment options from this screen.5 T he default language of the menu system is English. To select anotherlanguage, press the Menu button on the projector or remote control.Select Extended and press . Select Language and press . Select your language and press . Press the Menu button to exit the menu system.Adjust the image1If you don’t see an image, press the Source Search button on the projector or remote control to select the image source.Note: If you still see a blank screen or have other display problems, see the troubleshooting tips.2To raise the image, press the foot release button and lift the front of the projector. Release the button to lock the foot in position.3To reduce or enlarge the image, press the E-Zoom buttons on the remotecontrol.4To sharpen the image, turn the focus ring.5I f your image looks like or , you may have placed the projector off to one side of the screen at an angle. Place the projector directly in front of the center of the screen, facing the screen squarely. If you can’t move the projector, select the Keystone setting on the Home screen or in the Settings menu to correct the image shape.6If your image looks like or, press theprojector to correct it.Using the remote controlInstall the batteries as shown (two AA batteries).Choose which source to displayon and offAccess projectormenusTurn off projectordisplayAccess the Home screenNavigate projector menusFor more information on using the remote control, see the online User’s Guide .Troubleshooting• If you see a No signal message after turning on the projector, make sure your computer or video device is properly connected.•I f you see a blank screen, check the following: • Make sure the power light on the projector is blue and notflashing.•P ress the power button to wake the projector from standby or sleep mode, if necessary.• If you see a blank screen when a computer is connected, check the following:• On some Windows ® laptops, you may need to hold down the Fn key and press F7 or the function key that lets you display on an external monitor. It may be labeled CRT/LCD or have an icon such as .On Windows 7 or later, hold down the Windows key and press P at the same time, then click Duplicate .• If you’re using a Mac laptop, open System Preferences and select Displays . Click the Arrangement tab and select the Mirror Displays checkbox.• If the projector does not respond to remote control commands, make sure the batteries in the remote control are installed correctly. Replace the batteries if necessary.RegistrationRegister today to get product updates and exclusive offers. Visit/support and search for your product, then click Registration to register your product.Where to get helpManualsFor more information about using the projector, you can view or download the online manuals from the Epson website, as described below.Internet supportVisit /support and search for your product to download software and utilities, view manuals, get FAQs and troubleshooting advice, or contact Epson.Telephone support servicesYou can also speak to a service technician by calling one of these numbers:Argentina(54 11) 5167-0300Guatemala*1-800-835-03580800-288-37766México01-800-087-1080Bolivia*800-100-116México City (52 55) 1323-2052Chile(56 2) 2484-3400Nicaragua*00-1-800-226-0368Colombia 018000-915235Panama*00-800-052-1376 Bogota (57 1) 592-2200Paraguay 009-800-521-0019Costa Rica 800-377-6627Peru 0800-10126Dominican Lima (51 1) 418-0210 Republic 1-888-760-0068Uruguay 00040-5210067Ecuador*1-800-000-044Venezuela(58 212) 240-1111El Salvador*800-6570* C ontact your local phone company to call this toll free number from a mobile phone.If your country does not appear on the list, contact your nearest Epson sales office. Toll or long distance charges may apply.Optional accessoriesFor a list of optional accessories, see the online User’s Guide .You can purchase other accessories from an Epson authorized reseller. To find the nearest reseller, contact Epson.NoticesSupplier’s Declaration of ConformityAccording to 47CFR, Part 2 and 15, Class B Personal Computers and Peripherals; and/or CPU Boards and Power Supplies used with Class B Personal Computers:We: Epson America, Inc.Located at: 3840 Kilroy Airport WayMS: 3-13Long Beach, CA 90806-2469Telephone:(562) 981-3840Declare under sole responsibility that the product identified herein, complies with47CFR Part 2 and 15 of the FCC rules as a Class B digital device. Each product marketed, is identical to the representative unit tested and found to be compliant with thestandards. Records maintained continue to reflect the equipment being produced can be expected to be within the variation accepted, due to quantity production and testing on a statistical basis as required by 47CFR §2.906. Operation is subject to the following two conditions: (1) this device may not cause harmful interference, and (2) this device must accept any interference received, including interference that may cause undesired operation.Trade Name: Epson Type of Product: LCD Projector Model: H975AMarketing Name: PowerLite E10+Options:Remote controller (model 2198635xx [x=0–9])Limited Warranty1. Limited Warranty for Epson ProductsEpson products are covered by warranty against defects in workmanship and materials when operated under normal use and handling conditions, as indicated in the product documentation, for the period specified in section “Limited Warranty Coverage”.Coverage begins as of the date of delivery to the purchaser by Epson or by an authorized Epson dealer, in the country of purchase.Epson also guarantees that the consumables (batteries, filters, lenses) included with the product, will perform according to the usage specifications as long as they are used prior to the expiration of their warranty period.2. Scope of WarrantyShould Epson receive notice of a defect during the warranty period, it may, at its discretion, repair or replace the defective product at no cost to the purchaser. In the event of a replacement, the replaced equipment will become property of Epson. The replaced item may be new or previously repaired to the Epson standard of quality, and will assume the remaining warranty period of the product that was originally purchased.This warranty does not include any compensation or damages whatsoever, resulting from the operating failures of the Epson product. In any event, Epson’s maximum liability toward the purchaser will be limited to the purchase price paid to Epson or to the authorized Epson dealer.Epson’s warranty does not cover any software not manufactured by Epson, even when delivered with an Epson product. The software may have a warranty provided by the manufacturer, as indicated in the documentation attached thereto.3. Limitations and ExclusionsThe warranty will be invalid in the following cases:3.1 When the product’s serial number has been removed or altered.3.2 W hen the product has been misused, has undergone unauthorized modifications,has been used or stored in ways that are not in compliance with the environmental specifications of the product.3.3 W hen the damage resulted from the use of refilled supplies or consumables.3.4 W hen damages resulted from the inappropriate transport of the equipment(packaging, installation, maintenance, transportation).3.5 W hen damages were caused by natural or intentional disasters (fire, floods, lightning,earthquakes, etc.), power surges, or interaction with other brand products. 3.6 W hen the product has been totally or partially disassembled, or has been repairedother than by an Epson Authorized Service Center.3.7 W hen substances have been spilled on the product.3.8 W hen the external plastic case has been scratched or abused.3.9 W hen damages resulted from inappropriate tests, installation, maintenance oradjustments.The warranty does not extend to accessories (lids, covers, trays) or replenishing supplies and consumables (batteries, lamps, filters, and lenses), which, due to their nature, must be regularly purchased through authorized dealers. 4. How to Obtain Warranty ServicePlease refer to the user documentation to ensure that the product settings have been properly set and to perform the diagnostic tests indicated therein. Furthermore, in order for the unit to operate optimally, always use original Epson supplies and consumables.To obtain warranty service, the purchaser may take the product to any Epson Authorized Service Center, along with proof of purchase, or call any of the Epson Technical Assistance Centers (see country list below).Upon calling an Epson Authorized Service Center, the purchaser will be asked to provide the product model and serial numbers, and may be asked to provide information on the location and the date of purchase (the warranty is only valid in the country of purchase).If there are no Authorized Service Centers in your area, call the Technical Assistance Center for service referral.5. Responsibilities of PurchaserThe purchaser is responsible for the safety of all confidential and proprietary information stored on the product, as well as for recovery backup files in case of damage. Any activity related with the re-installation or resetting of the software that was delivered with the equipment, will be billed to the purchaser at the rates in effect at the Epson Authorized Service Center. This also applies if the failure was the result of a defect in the purchaser’s proprietary software or of a computer “virus”.In order to obtain on-site service, the purchaser will be required to provide: access to the product; an appropriate working area; access to electrical facilities; access to theresources that may be necessary for the installation, repair or maintenance of the unit; a safety environment for Epson staff and their work tools.6. Limited Warranty Coverage Product: Projector Model: PowerLite E10+Coverage: Two (2) years Conditions: Service CenterNote:Lamp has a ninety (90) day warranty.EPSON and PowerLite are registered trademarks and EPSON Exceed Your Vision is a registered logomark of Seiko Epson Corporation.Windows is a registered trademark of Microsoft Corporation in the United States and/or other countries.Mac and OS X are trademarks of Apple Inc., registered in the U.S. and other countries.General Notice: Other product names used herein are for identification purposes only and may be trademarks of their respective owners. Epson disclaims any and all rights in those marks.This information is subject to change without notice.© 2020 Epson America, Inc., 12/20Printed in XXXXXXCPD-58734R1Technical Support ServiceEpson offers technical assistance through electronic and telephone services. Before calling Epson, refer to the documentation included with your product. If you do not find the solution to your problem, visit Epson’s website at: /support .Technical Assistance Centers Argentina (54 11) 5167-0300Guatemala*1-800-835-03580800-288-37766México01-800-087-1080Bolivia*800-100-116México City (52 55) 1323-2052Chile(56 2) 2484-3400Nicaragua*00-1-800-226-0368Colombia 018000-915235Panama*00-800-052-1376 Bogota (57 1) 592-2200Paraguay 009-800-521-0019Costa Rica 800-377-6627Peru 0800-10126Dominican Lima (51 1) 418-0210 Republic 1-888-760-0068Uruguay 00040-5210067Ecuador*1-800-000-044Venezuela(58 212) 240-1111El Salvador*800-6570* C ontact your local phone company to call this toll free number from a mobile phone.If your country does not appear on the list, contact your nearest Epson sales office. Toll or long distance charges may apply.You may receive help or consult information online for all Epson products. Go to Epson’s website at /support and search for your product, then click on Registration to register your product.Extended Warranty Support and ServiceIn some countries, Epson offers warranty extension services. You may purchase this service by calling the Technical Assistance Center.Epson Subsidiaries and Offices in Latin America Epson Argentina S.R.L.San Martín 334, Piso 4(C1004AAH), Buenos Aires ArgentinaTel: (54 11) 5167-0300Fax: (54 11) 5167-0333Epson Chile Concepción 322Providencia, Santiago, Chile Tel: (56 2) 2484-3400Fax: (56 2) 2484-3413Epson Colombia Ltda.Calle 100, No. 19-54Piso 7, Of. 704Bogotá, Colombia Tel: (57 1) 592-2200Epson Costa Rica S.A.102 Avenida EscazúTorre 1, Piso 4, Suite 401/402Escazú, San José, Costa Rica Tel: (506) 2588-7855Fax: (506) 2588-7888Epsodecua Cia. Ltda.Av. de los Shyris N36-120 y Suecia Edificio Allure Park, Piso PH Quito, EcuadorTel: (593 2) 395-5951Tel: (593 2) 602-3070Epson México, S.A. de C.V .Blvd. Manuel Ávila Camacho No. 389Col. Irrigación, Delg. Miguel Hidalgo C.P . 11500Ciudad de México, México Tel: (52 55) 1323-2000Epson Perú S.A.Av. Canaval y Moreyra 590San Isidro, Lima 27, PerúTel: (51 1) 418-0210Fax: (51 1) 418-0220Epson Venezuela S.R.L.Calle 4 con Calle 11-1La Urbina SurCaracas, Venezuela Tel: (58 212) 240-1111。
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A 70-YEAR-OLD MAN WITH EXTRAPYRAMIDAL SYMPTOMS, DEMENTIA AND HEMOSIDEROSIS Fabrice Chrétien, MD, PhD1,2; Jérôme Servan, MD3; Jacqueline Mikol, MD, PhD4;Michèle Trierweiller, MD3;Dominique Elghozi, MD3;Françoise Gray, MD, PhD41 INSERM EMI 0011, Université Paris XII, Créteil, France;2 Service d’Histologie—Hôpital Henri Mondor AP-HP, 94010 Créteil cedex, France;3 Service de Neurologie, Hôpital René Dubos, Pontoise, France;4 Service Central d’Anatomie et de Cytologie Pathologiques, Hôpital Lariboisière AP-HP, Paris, France.CLINICAL HISTORYA 70-year-old man presented with an 8-year history of cognitive decline. Initial presenting signs included mild memory loss, behavioral disturbances, and transient disorientation. He had frontal release signs, including severe confusion, hallucinations and bilateral grasping, pyramidal signs, facial dyskinesia and dysmetria. Initial “mini mental status” (MMS) was quoted as 25/30. Laboratory investigations showed undetectable ceruloplasmin in serum, increased ferritin (1777 µg/L—nl 30–300) and a slight decrease of serum iron (11.8 µmol/L–nl 12–30) and copper (0.06 mg/L—nl 0.8–1.4). Fundus oculi was normal; there was neither Kayser–Fleischer ring nor retinal abnormality. There were no signs of diabetes mellitus. A magnetic resonance image (MRI) revealed bilateral increased signal intensities of deep nuclei on T1 weighted that were more marked on T2 weighted sequences. The abnormalities involved predominantly the putamen, caudate nucleus, pulvinar, and dentate nucleus (Figure 1A,B). There was no contrast enhancement. A liver biopsy showed marked iron deposition but no cir-rhosis. Aspirated bone marrow was normal except for massive iron deposition. The patient’s condition progressively worsened. Six years after initial presentation, he was bedridden with obvious dementia (M M S less than 5/30) and mutism. He died from aspiration bronchopneumonia, 8 years after the onset of neurological signs. There was neither a family history of similar disease nor consanguinity between the close parents. T wo brothers had diabe-tes mellitus, progressive mental deteriora-tion, movement disorders and cerebellarataxia, together with aceruloplasminemiawhereas serum ferritin and serum iron wereincreased. In the descendants, two of thethree children presented with a low bloodlevel of ceruloplasmin compatible with aheterozygous form of the disease.GROSS PATHOLOGYVisceral examination showed diffusehemosiderosis of the heart, liver and lung.Gross examination of the brain (1170 g)showed predominant involvement of thebasal ganglia with rust-brown discolorationand cavitation of the striatum, posteriorthalamus and dentate nuclei (Figure 2A,B)as well as mild cortico-subcortical atrophy.MICROSCOPIC PATHOLOGYProminent changes were observed inthe striatum, posterior thalamus and den-tate nucleus. Lesions were symmetrical andbilateral. In the necrotic foci, there wassevere neuronal loss with fibrosis associatedwith marked astrocytosis and macrophageactivation. Astrocytosis included gemisto-cytic astrocytes and Alzheimer’s type IIglia. M acrophages expressing CD68 andHLA-DR were numerous and some ofthem had the morphological characteristicsof Opalski cells (Figure 3A,B). Iron depos-its better identified on Perls stain wereabundant in the necrotic foci but weremore diffuse, and also found in the cerebraland cerebellar cortex (Figure 3C,D) and toa lesser extent in the white matter. Theywere observed in the cytoplasm of mac-rophages and astrocytes but were alsopresent in neurons or free in the neuropil.Axonal swellings better demonstratedby Bodian silver impregnation or beta-Amyloid Protein Precursor immunostain-ing, were numerous within and around thenecrotic foci. They frequently containediron deposits. There was severe involve-ment of the cerebellar cortex with irondeposition, loss of Purkinje cells and pro-liferation of Bergmann glia. The whitematter, globus pallidus and substantianigra did not show significant changesexcept slight iron deposits (please see theonline version of this case for additionalimages /divisions/neuropath/bpath/cases/case118.html).CORRESPONDENCE: Reader Submitted Case ReportsEdited by Dr Ronald L. HamiltonFigure 1.A BFigure 2.A BFigure 3.A B C DDIAGNOSIS AND DISCUSSION Diagnosis. Hereditary ceruloplasmin deficiency with hemosiderosis. DISCUSSIONCeruloplasmin deficiency is a newly recognized autosomal recessive disorder,first described in Japan in 1987 (4), linked to a mutation of the ceruloplasmin gene located on chromosome 3. This case is the first French case (6). Ceruloplasmin is an abundant serum glycoprotein, a copper-binding oxidase that carries more than 95% of the plasma copper (3). It is synthe-sized in hepatocytes and can oxidize several substrates and especially plays the role of a ferroxidase. In the few reported cases of aceruloplasminemia, patients presented with non-specific symptoms, that is, association of intellectual deterioration, extrapyramidal signs and, inconstantly, cerebellar ataxia. Associated diabetes melli-tus and pigmentary degeneration of the retina are frequent. Biological tests show complete lack of ceruloplasmin in keeping with a homozygous mutation of the ceru-loplasmin gene. All the patients had increased serum ferritin concentration and serum iron level. Cerebrospinal fluid exam-ination also showed significant increase of iron concentration (3).There are only few neuropathological studies (2, 5). The pathological changes involve predominantly the basal ganglia, particularly the striatum and posterior thalamus, and the dentate nucleus; the cerebral cortex is less affected. They include necrosis with massive iron deposi-tion, marked neuronal loss and gliosis. The neuropathological findings in our case are comparable to those in the previously reported pathological cases (2) except for the presence of Opalski cells and Alzhe-imer type II astrocytes, which were not found in the latter.The main differential diagnosis includes three congenital entities sharing common clinical, biological and pathological features.Wilson disease (hepatolenticular degen-eration) is an autosomal recessive disorder of copper excretion with variable pheno-typic expression. It is related to mutations of the Wilson gene on chromosome 13 (more than 190 mutations). Its product is ATP7B, a cathion transporting P-type ATPase with six copper-binding domains,located in mitochondria. Hypoceruleoplas-minemia is considered a characteristic fea-ture of Wilson’s disease; however, it is onlythe consequence of copper deficiency anddoes not have any physiopathological role.Clinically, the disease include cirrhosis,progressive rigidity, tremor, dysarthria,dysphagia and, if untreated, dementia. Aring of brown pigmentation in the cornea(Kaiser Fleischer ring) is a pathognomonicsign. The clinical presentation is quite sim-ilar with aceruloplasminemia except fordiabetes mellitus. In contrast, pathologicalexamination in Wilson disease does notshow iron deposition in tissues but exces-sive copper deposition in the liver (excep-tionally associated with cirrhosis) brainand kidneys. At neuropathological exami-nation, the topography of the lesions issuperimposable to that in aceruloplasmine-mia and includes necrosis, gliosis withAlzheimer’s type II astrocytes and micro-glial activation with Opalski cells. Thesecells have been originally described in Wil-son disease and are considered characteris-tic of the condition. They are large cellswith small nuclei and no obvious cytoplas-mic processes. They have a finely granularor foamy cytoplasm (1). In Wilson disease,they contain copper deposits.Pantothenate kinate-associated neuro-degeneration (PKAN) (Hallervorden-Spatz syndrome) is an autosomal recessivedisease linked to a mutation in the pan-tothenate kinase gene (PANK2). PKAN isclearly an iron deposition disease related toa reduction of the cysteine dioxigenaseactivity. The disease begins usually aroundthe age of 15 years and presents with gaitdisorders, rigidity of the lower limbs, dys-tonia, motor slowing, dysarthria, mentaldeterioration and a high incidence ofchoreoathetosis. Most patients die by theage of 35 years. In patients with longersurvival, the disease usually presents as anextrapyramidal movement disorder associ-ated with dementia. MRI, is usually diag-nostic and shows “eye of the tiger sign” thatis considered as a specific feature. Themicroscopic changes are roughly similar tothose in aceruloplasminemia includingiron deposition and numerous axonalspheroids, but are limited to the globuspallidus and pars reticularis of the substan-tia nigra. In these cases, the dentatenucleus, striatum, and thalamus are unaf-fected. There is no systemic extra-cerebraliron deposition.Idiopathic hemochromatosis is anuncommon HLA-related genetic defect inthe regulation of gastrointestinal ironabsorption leading to excessive systemiciron deposition, particularly in the liver.The latter induces cirrhosis, which appearsaround 40 years of age. The cirrhosis isresponsible for secondary neurologicalinvolvement, that is, hepatic encephalopa-thy. In the brain, iron deposition can onlybe found in areas in which the blood-brainbarrier is absent (e.g. choroids plexus, areapostrema).REFERENCES1. Gray F, Poirier J, De Girolami U (eds) (2003)Escourolle and Poirier’s Manual of Neuropathology,4th edn. Elsevier: Boston.2. Kaneko K, Yoshida K, Arima K, Ohara S, MiyajimaH, Kato T, Ohta M, Ikeda SI (2002) Astrocytic defor-mity and globular structures are characteristic ofthe brains of patients with aceruloplasminemia. JNeuropathol Exp Neurol 61:1069–1077.3. M iyajima H, Fujimoto M, Kohno S, Kaneko E,Gitlin JD (1998) CSF abnormalities in patients withaceruloplasminemia. Neurology 51:1188–1190.4. M iyajima H, Nishimura Y, M izoguchi K, Saka-moto M, Shimizu T, Honda N (1987) Familial apo-ceruloplasmin deficiency with blepharospasmand retinal degeneration. Neurology 37:761–767.5. M orita H, Ikeda SI, Yamamoto K, M orita S,Yoshida K, Nomoto S, Kato M, Yanagisawa N(1995) Hereditary ceruloplasmin deficiency withhemosiderosis: a clinicopathological study of aJapanese family. J Neurol Neurosurg Psychiatry61:506–509.6. Servan J, Elghozi D, Gaynot S, Duclos H (1998)Hémosidérose cérébrale liée à un déficit hérédi-taire en céruléoplasmine—Etude clinique d’uncas familial. Rev Neurol 154:158–162.。
详细版国际象棋开局百科
国际象棋开局百科全书2021版，研究开局的必备利器。
涵盖了4900多种开局的详细解释，还包括380万多盘的数据库。
下面我给你介绍全书，欢迎阅读。
[ECO]--------C类开局C00 法兰西防御，1 e4 e6C01 法兰西防御，兑换变例，1 e4 e6 2 d4 d5 3 exd5 exd5 4 Nc3 Nf6 5 Bg5C02 法兰西防御，推进变例，1 e4 e6 2 d4 d5 3 e5C03 法兰西防御，塔拉什防御，1 e4 e6 2 d4 d5 3 Nd2，C04 法兰西防御，塔拉什防御，吉马德主变，1 e4 e6 2 d4 d5 3 Nd2 Nc6 4 Ngf3 Nf6C05 法兰西防御，塔拉什防御，1 e4 e6 2 d4 d5 3 Nd2 Nf6C06 法兰西防御，塔拉什防御，1 e4 e6 2 d4 d5 3 Nd2 Nf6 4 e5 Nfd7 5 Bd3 c5 6 c3 Nc6 7 Ne2 cxd4 8 cxd4C07 法兰西防御，塔拉什防御，1 e4 e6 2 d4 d5 3 Nd2 c5C08 法兰西防御，塔拉什防御，开放变例，1 e4 e6 2 d4 d5 3 Nd2 c5 4 exd5C09 法兰西防御，塔拉什防御，开放变例主变，1 e4 e6 2 d4 d5 3 Nd2 c5 4 exd5 exd5 5 Ngf3 Nc6C10 法兰西防御，保尔森变例，1 e4 e6 2 d4 d5 3 Nc3C11 法兰西防御，1 e4 e6 2 d4 d5 3 Nc3 Nf6C12 法兰西防御，迈克卡西昂变例，1 e4 e6 2 d4 d5 3 Nc3 Nf6 4 Bg5 Bb4C13 法兰西防御，古典变例，1 e4 e6 2 d4 d5 3 Nc3 Nf6 4 Bg5 Be7C14 法兰西防御，古典变例，1 e4 e6 2 d4 d5 3 Nc3 Nf6 4 Bg5 Be7 5 e5 Nfd7 6 Bxe7 Qxe7C15 法兰西防御，温纳维尔尼姆佐维奇变例，1 e4 e6 2 d4 d5 3 Nc3 Bb4C16 法兰西防御，温纳维尔尼姆佐维奇变例，推进变例,1 e4 e6 2 d4 d5 3 Nc3 Bb4 4 e5C17 法兰西防御，温纳维尔尼姆佐维奇变例，推进变例,1 e4 e6 2 d4 d5 3 Nc3 Bb4 4 e5 c5C18 法兰西防御，温纳维尔尼姆佐维奇变例，推进变例,1 e4 e6 2 d4 d5 3 Nc3 Bb4 4 e5 c5 5 a3 Bxc3+ 6 bxc3C19 法兰西防御，温纳维尔尼姆佐维奇变例，推进变例,1 e4 e6 2 d4 d5 3 Nc3 Bb4 4 e5 c5 5 a3 Bxc3+ 6 bxc3 Ne7C20 王兵开局,1 e4 e5C21 中心开局，丹麦弃兵，1 e4 e5 2 d4 exd4C22 中心开局，1 e4 e5 2 d4 exd4 3 Qxd4 Nc6C23 飞象开局，1 e4 e5 2 Bc4C24 飞象开局，柏林防御，1 e4 e5 2 Bc4 Nf6C25 维也纳开局，1 e4 e5 2 Nc3C26 维也纳开局，佛克比尔变例，1 e4 e5 2 Nc3 Nf6C27 维也纳开局，1 e4 e5 2 Nc3 Nf6 3 Bc4 Nxe4C28 维也纳开局，1 e4 e5 2 Nc3 Nf6 3 Bc4 Nc6C29 维也纳弃兵局，考夫曼变例，1 e4 e5 2 Nc3 Nf6 3 f4C30 拒绝王翼弃兵局，1 e4 e5 2 f4C31 拒绝王翼弃兵局，佛克比尔反弃兵，1 e4 e5 2 f4 d5C32 拒绝王翼弃兵局，佛克比尔反弃兵，1 e4 e5 2 f4 d5 3 exd5 e4 4 d3 Nf6C33 承受王翼弃兵局，1 e4 e5 2 f4 exf4C34 承受王翼弃兵局，1 e4 e5 2 f4 exf4 3 Nf3C35 承受王翼弃兵局，科宁汉姆防御，1 e4 e5 2 f4 exf4 3 Nf3 Be7C36 承受王翼弃兵局，阿巴西亚防御古典防御，现代防御，1 e4 e5 2 f4 exf4 3 Nf3 d5C37 承受王翼弃兵局，科沃爱德弃兵，1 e4 e5 2 f4 exf4 3 Nf3 g5 4 Nc3C38 承受王翼弃兵局，1 e4 e5 2 f4 exf4 3 Nf3 g5 4 Bc4 Bg7C39 承受王翼弃兵局，1 e4 e5 2 f4 exf4 3 Nf3 g5 4 h4C40 王翼马开局，1 e4 e5 2 Nf3C41 菲立道尔防御，1 e4 e5 2 Nf3 d6C42 俄罗斯防御彼得罗夫防御，1 e4 e5 2 Nf3 Nf6C43 俄罗斯防御彼得罗夫防御，现代攻击斯坦尼茨攻击，1 e4 e5 2 Nf3 Nf6 3 d4 exd4 4 e5 Ne4 5 Qxd4C44 庞齐阿尼开局，1 e4 e5 2 Nf3 Nc6C45 苏格兰开局，1 e4 e5 2 Nf3 Nc6 3 d4 exd4 4 Nxd4C46 三马开局，1 e4 e5 2 Nf3 Nc6 3 Nc3C47 四马开局，苏格兰变例，1 e4 e5 2 Nf3 Nc6 3 Nc3 Nf6C48 四马开局，西班牙变例，1 e4 e5 2 Nf3 Nc6 3 Nc3 Nf6 4 Bb5C49 四马开局，1 e4 e5 2 Nf3 Nc6 3 Nc3 Nf6 4 Bb5 Bb4C50 意大利开局钢琴开局，1 e4 e5 2 Nf3 Nc6 3 Bc4 Bc5C51 意大利开局，伊文思弃兵，1 e4 e5 2 Nf3 Nc6 3 Bc4 Bc5 4 b4C52 意大利开局，伊文思弃兵，1 e4 e5 2 Nf3 Nc6 3 Bc4 Bc5 4 b4 Bxb4 5 c3 Ba5C53 意大利开局，1 e4 e5 2 Nf3 Nc6 3 Bc4 Bc5 4 c3C54 意大利开局，1 e4 e5 2 Nf3 Nc6 3 Bc4 Bc5 4 c3 Nf6 5 d4 exd4 6 cxd4C55 双马防御，1 e4 e5 2 Nf3 Nc6 3 Bc4 Nf6C56 双马防御，1 e4 e5 2 Nf3 Nc6 3 Bc4 Nf6 4 d4 exd4 5 O-O Nxe4C57 双马防御，1 e4 e5 2 Nf3 Nc6 3 Bc4 Nf6 4 Ng5C58 双马防御，1 e4 e5 2 Nf3 Nc6 3 Bc4 Nf6 4 Ng5 d5 5 exd5 Na5C59 双马防御，1 e4 e5 2 Nf3 Nc6 3 Bc4 Nf6 4 Ng5 d5 5 exd5 Na5 6 Bb5+ c6 7 dxc6 bxc6 8 Be2 h6C60 西班牙开局，1 e4 e5 2 Nf3 Nc6 3 Bb5C61 西班牙开局，别尔德防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 Nd4C62 西班牙开局，斯坦尼茨防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 d6C63 西班牙开局，雅尼什弃兵，1 e4 e5 2 Nf3 Nc6 3 Bb5 f5C64 西班牙开局，古典体系，1 e4 e5 2 Nf3 Nc6 3 Bb5 Bc5C65 西班牙开局，柏林防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 Nf6C66 西班牙开局，柏林防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 Nf6 4 O-O d6C67 西班牙开局，柏林防御，开放变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 Nf6 4 O-O Nxe4C68 西班牙开局，兑换变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Bxc6C69 西班牙开局，兑换变例，格里戈利奇变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Bxc6 dc 5 O-O f6 6 d4C70 西班牙开局，现代斯坦尼茨防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4C71 西班牙开局，现代斯坦尼茨防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 d6C72 西班牙开局，现代斯坦尼茨防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 d6 5 O-OC73 西班牙开局，现代斯坦尼茨防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 d6 5 Bxc6+ bxc6 6 d4C74 西班牙开局，现代斯坦尼茨防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 d6 5 c3C75 西班牙开局，现代斯坦尼茨防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 d6 5 c3 Bd7C76 西班牙开局，现代斯坦尼茨防御，侧翼象防御布龙斯坦防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 d6 5 c3 Bd7 6 d4 g6C77 西班牙开局，摩菲防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6C78 西班牙开局，摩菲防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-OC79 西班牙开局，延迟斯坦尼茨防御俄罗斯防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O d6C80 西班牙开局，开放变例塔拉什防御、苏格兰变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Nxe4C81 西班牙开局，开放变例，霍维尔攻击，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Nxe4 6 d4 b5 7 Bb3 d5 8 dxe5 Be6C82 西班牙开局，开放变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Nxe4 6 d4 b5 7 Bb3 d5 8 dxe5 Be6 9 c3C83 西班牙开局，开放变例，古典体系，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4Nf6 5 O-O Nxe4 6 d4 b5 7 Bb3 d5 8 dxe5 Be6C84 西班牙开局，封闭体系，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7C85 西班牙开局，兑换变例双重延迟，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Bxc6 dxc6C86 西班牙开局，沃罗尔攻击，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Qe2C87 西班牙开局，封闭体系，阿维尔巴赫变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 d6C88 西班牙开局，封闭体系，反马歇尔弃兵变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3C89 西班牙开局，封闭体系，马歇尔弃兵，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d5C90 西班牙开局，封闭体系，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6C91 西班牙开局，封闭体系，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6 9 d4C92 西班牙开局，封闭体系，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6 9 h3C93 西班牙开局，封闭体系，斯梅斯洛夫防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6 9 h3 h6C94 西班牙开局，封闭体系，布列热尔防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6 9 h3 Nb8C95 西班牙开局，封闭体系，布列热尔防御，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6 9 h3 Nb8 10 d4C96 西班牙开局，封闭体系，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6 9 h3 Na5 10 Bc2C97 西班牙开局，封闭体系，奇戈林变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6 9 h3 Na5 10 Bc2 c5 11 d4 Qc7C98 西班牙开局，封闭体系，奇戈林变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6 9 h3 Na5 10 Bc2 c5 11 d4 Qc7 12 Nbd2 Nc6C99 西班牙开局，封闭体系，奇戈林变例，1 e4 e5 2 Nf3 Nc6 3 Bb5 a6 4 Ba4 Nf6 5 O-O Be7 6 Re1 b5 7 Bb3 O-O 8 c3 d6 9 h3 Na5 10 Bc2 c5 11 d4 Qc7 12 Nbd2 cxd4 13 cxd4。
pes2011 妖人 最佳阵容终极版 全红 均值破百 入手教程
好了，临时想起来的就这些了，有不明白的，想要存档的，想批评我的都可以发我的E邮箱
wolfegg@
我叫丹丹，嘿嘿。
最近在更新妖人的名单，由于妖人20年或者10多年之后可能重现，所以名单的属性我也做了无作弊器修改，目前踢到了2040赛季，财产90000W...也就是9亿美元。不相信我可以发给你我的存档，编辑栏里面绝对一个人都没有...
解决了这两大难题，组成这么强大的阵容也不是多难的事情了，最后再提点儿经营球队的建议把，由于有了教练系统，咱们可以少踢了不少球，我也只是对手厉害的时候踢踢，弱队手的确没意思。我想说的是，教练不用雇佣多好的300W的那个就行，而其他都顾最好的，500W的，然后青年队和俱乐部都可以降到LV1（一级），花钱不讨好的组织。
备注 队里还有几个潜力股我就不晒数据了，目前数据不太好看。要注意的是门将的属性，门将总评破95的是凤毛麟角，这样就不错了。另外我玩儿的英文版的，汉化也没汉化名字，所以我就直接打上了，怕自己翻译的不准。
OK,数据晒完了，是不是很心动啊，心动没问题，想要存档没问题，给我发邮件。
wolfegg@
了解了这种困难，我们就开始想办法，怎么能低价续约呢？
我就想了，，不行，丫不新了，没准还因为得了最佳足球先生还巨有名，新个P。
想到这，我灵机一动，丫不新，你给他变新不就行了！
办法是：开除(英文版是RELEASE)
第二个难题，球队，但是由于续签妖人后要的工资太TM高了，到最后都濒临破产...直到我发现了一个方法...先别急，咱先看看为啥他们的价格会疯长。
PES2011在买卖球员方面和现实对接的很像，20岁以前签约新人普遍价格低的让人发指，因为你丫年轻，就是便宜，但是一旦丫成长起来了就麻烦了，举例比如一个球员起薪80W(万美元),20岁总评到了95左右，那年薪必然涨到800W左右，前锋更贵。好，再续约，又过了几年到了巅峰年龄25左右，总评到了105，那完蛋，工资必然超过2000W，不用说，队里有两三个这么狠的肯定经营的惨淡，濒临破产。这是可以理解的，毕竟人家黄金年龄嘛。
韩语日常用语(中文发音)
韩语1.你好: 啊你啊塞哟 -2.多多关照: 擦儿不大卡米大 -3.谢谢: 卡目沙米大 -4.对不起: 罪送哈米大 -5.见到你很高兴: 满拉索盼嘎不是米大 -7.再见,走好(客人对主任说的话): 安宁习, 给色哟 -8.我爱你: 萨郎黑哟 -9.喜欢:做啊黑哟 -11.我吃饱了: 别不儿罗哟 -12.肚子饿了: 过怕哟 -14.晚安: 安宁习, 租目塞哟 -19.我叫...: 错能... -20.我是中国人: 错能, 总谷沙拉米米打 -词语: -1.知道: 啊拉嗦 -2.开始: 洗嫁 -5.真是的: 啊西 -6.你先说: on这巴爹哟 -7.他们: kei du(第三声) -8.没有(不是): 啊你哦 -9.你是谁?: 怕你衣死尬? -10.你疯了: u捉索 -11.但是: 肯爹 -12.奇怪: 衣索念 -13.什么: 爹? -14.是: 爹 -韩语中文读法这是什么: (一个森波西你噶)您有时间吗? : (西赶你是你噶)好吃: 马西达你: 弄喜欢你: 出挖嘿- 1 - / 13爱你: 撒浪嘿傻瓜: 怕不 (加感叹词: 怕不呀)有趣: 退米一搜你说什么?: 木孙素里啊?你说慌!/骗人!: 阔几满!无赖/没教养: 撒嘎几臭混蛋: 望杂个几 (智恩老爱这么叫英宰...汗!)想死吗?: 出过列?你疯了吗?: 弄皮差搜?!不行!: 安对不要/不: 西罗吃吧: 摸果操(骂人D): 西吧儿 (非常D脏, 不要随便骂~哈哈) 亲爱的: 差嘎啊~怎么回事?: 温泥里呀?怎么了?: 为以类? (或者, 为古类?)怎么/怎么办: 哦提开知道了: 啊拉 (啊拉搜哟)起来!: 以罗那 (智恩老是爱说: 以罗那挖哟!)出来!: 纳挖快点: 摆里说说看/说吧!: 马类吧为什么?: 无为?是的: 也 (也可以说 "DAE")生日: 三以日祝贺: 出卡嘿哟真的: 虫么儿 (也可以说"亲加")等待: KI大里过呀可爱: KI哟达漂亮: 以扑达可是/但是: 哈几满说实话: 素几KI那够了!: 西库罗结束: 古那S- 2 - / 13再一次: 汉波满很想你: 不过西破 (加感叹次可以是 "就" 或者 "搜")没事吧/不要紧吧?: 捆察那哟? (可以回答: 捆察那=我没事!) 过分!: 诺满达! (真过分：亲加诺满达!)跟我来: 脱罗挖你死定了!: 出过以西!走!!: 卡!快走!!: 摆里卡!走啦~: 卡扎!我走了!: 那儿看达!爸爸：啊爸（几）妈妈：哦妈（泥）哥哥（女生叫的）：OPPA哥哥（男生叫的）：hiang奶奶：哈拉不你爷爷：哈拉不几姐姐（女生叫的）：哦你姐姐（男生叫的）：努那大叔：阿则西小姐：啊加西姨妈：姨末阿姨：阿吉妈朋友：亲古舅舅：桑丘你：诺我们：吾利谁：怒古（塞哦是说话的语气词）怒古塞哦我：那 (是我，那呀；我走了：那儿看达)喜欢你: 丘外嘿我爱你：沙郎EI（这个音更近）亲爱的：差嘎啊~非常(很)：侬木侬木(非常喜欢你：侬木侬木丘外嘿)是；内/也不是：啊你呀- 3 - / 13不行：安对不知道：木拉不要/不喜欢：西罗不要走：卡几马走!：卡!快走!!：摆里卡!走啦~：卡扎!我走了!：那儿看达!起来!：以罗那出来!：纳挖进来!：脱罗挖上车：踏你好：阿尼阿塞约：可以有很多用法，打招呼可以，分手再见也可以谢谢：卡撒哈米大/古吗诶哦/古吗扑四米大不好意思：捶送哈米大对不起：米呀内（米呀哈米大）没事/没关系：捆擦那有恭喜：粗卡EI祝贺: 出卡嘿哟拜托：扑他哈米大可爱: KI哟达漂亮: 以扑达傻瓜: 怕不 (加感叹词: 怕不呀)王八蛋：望撒个几无赖/没教养: 撒嘎几有趣: 退米一搜真神奇：兴噶达疯了：米错索什么：摸或者摸噶（加多一个为什麽说为er）为什么?: 无为?怎么了?: 为古类怎么回事?: 温泥里呀?怎么/怎么办: 哦提开- 4 - / 13你说什么?: 木孙素里啊?真的: 虫么儿 (也可以说"亲加")知道：阿拉索，知道吗也是用阿拉索，只不过最後那个索就是重音,还要很强的语气快点: 摆里又：度但是：哈几满可是：恳对说说看/说吧!: 马类吧好吃: 马西达吃吧: 摸果韩语发音1、 (安宁哈塞哟)：见到韩国朋友说一句"您好"中文：你好！拼音：an(1) ning(2) ha(1) sai(4) yo(1)2、 (满拉所盼嘎不是米大)：见到你很高兴3、 (擦儿不它卡米大)：多多关照4、 (卡目沙哈米大)：谢谢谢谢: 嘎么撒哈么呢达 !够吗撕么呢达中文：谢谢！拼音：gao(1) mab(1) sim(1) ni(4) da(2)谢谢: （嘎么撒哈么呢达!）或者：够吗撕么呢达谢谢：卡撒哈米大/古吗诶哦/古吗扑四米大5、 ( 罪送哈米大) 或（米安哈么呢达）：对不起对不起：米呀内（米呀哈米大）6、 (安宁习卡色哟)：再见，走好，在客人离开的时候主人对客人说的话中文：再见！（送客人时用）拼音：an(1) ning(2) hi(1) gai(2) sai(4) yo7、 (安宁习给色哟 )：再见，对主人说的话中文：再见！（从客人家离开时用）拼音：an(1) ning(2) hi(1) gai(1)sai(4) yo- 5 - / 138、 (萨朗黑哟) ：我爱你，在韩剧中经常可以听到的。
西内重复试题
西医内科学第一单元传染病细目一：细菌性痢疾3.诊断。
A1型题2002年试题45.典型细菌性痢疾的粪便呈：（D）试题难度: ＊＊A.稀水样便B.米泔水样便C.鲜血便D.粘液脓血便E.灰白色便2003年试题45.典型细菌性痢疾的粪便呈：（D）试题难度: ＊＊A.稀水样D.米泔水样C.鲜血便D.粘液脓血便E.灰白色便细目二：霍乱4．实验室诊断A2型题2002年试题103.某患者由印尼入境后2天，频繁腹泻，无腹痛及里急后重，伴有呕吐，最重要的检查是：（D）试题难度: ＊A.血常规B.尿常规C.电解质D.泻、吐物悬滴检查E.以上均非2003年试题103.一患者由印尼入境后2天，频繁腹泻，无腹痛及里急后重，伴有呕吐。
下列哪项检查是最重要的：（D）试题难度: ＊A.血常规B.尿常规C.电解质D.泻、吐物悬滴检查E.以上均非细目三：伤寒2.实验室检查A1型题2002年试题46.血培养伤寒杆菌阳性率最高的时间是：（B）试题难度: ＊A.第1周B.第2周C.第3周D.第4周E.第5周2003年试题46.伤寒菌血液培养，阳性率最高的时间是：（B）(基层卫生技术人员传染性疾病防治知识全员培训P125)试题难度: ＊A.第1周B.第2周C.第3周D.第4周E.第5周细目四：病毒性肝炎2.临床表现及实验室检查。
A2型题2002年试题104.患者，男，20岁，近2周自觉乏力，食欲不振，厌油，腹胀。
检查：巩膜无黄染，肝肋缘下2cm，有压痛，丙氨酸转氨酶升高。
应首先考虑的是：（A）试题难度: ＊A.急性肝炎B.慢性肝炎C.重型肝炎D.瘀胆型肝炎E.肝炎肝硬化2003年试题104.患者，男，20岁。
近2周自觉乏力，食欲不振，厌油，腹胀。
查体：巩膜无黄染，肝肋缘下2cm，有压痛。
丙氨酸转氨酶升高。
应首先考虑的是：（A）试题难度: ＊A.急性肝炎B.慢性肝炎C.重型肝炎D.淤胆型肝炎E.肝炎肝硬化第二单元呼吸系统疾病细目一：支气管炎1.诊断与鉴别诊断。
磁性物理中的LLG方程的求解与讨论.docx
山西师范大学本科毕业论文磁性物理中的LLG方程的求解与讨论姓名院系专业班级学号指导教师答辩日期成绩郭勤皇物理与信息工程学院物理学135201041354010443白宇浩磁性物理中的LLG方程的求解与讨论内容摘要磁性物质是材料科学研究中的重点课题，研究磁性物质中磁矩随时间的演化行为不仅在基础物理上具有较强的理论意义，而且也是实际磁性器件设计和使用中的一个重要问题。
Landeiu-Lifshitz-Gibert (LLG)方程是描述铁磁物质的磁矩在交变磁场屮随时间变化的基本方程，也是磁学领域中最重要的一个数理方程。
本文叙述了LLG方程的理论背景与发展状况，并以磁隧道结传感器为例，介绍了LLG方程的具体求解过程；另外, 我们对理论计算结果也进行了较为详细的讨论。
木论文的研究结果有利于我们对LLG方程的进一步理解和认识，也有利于我们讨论磁性物质的磁化反转过程。
【关键词】Landau-L i fsh itz-G i bert方程磁性传感器磁矩反转垂直磁各向异性磁阻设备Solution and Discussion of the LLG equation in the field of themagnetic physicsAbstractThe research of the magnetic material is the basic issue in the field of the materials science. The magnetization reversal process in the ferromagnetic materials is very meaningful in the investigation of the theoretical physics. In addition, it can also be useful for the desig n of the related magn etic devices. The Landau-Lifshitz-Gibert(LLG) equation is an basic equation in the field of the magnetism which is used to describe the time evolution of the magnetization under the condition of the alternating electromagnetic field. In this paper, we have introduced the background and the development of the LLG equation. Furthermore, taking the magnetic tunnel junction for example, we introduce the method of solving the LLG equation, at the same time, we also make a detailed discussion about the calculated results・Our investigations are useful to understand the LLG equation. Additionally, it will also be useful to discuss the magnetization reversal process in the ferromagnetic materials.[Key Words] Landau-Lifshitz-Gibert equation Magnetic sensors spin valves,magnetoresistive devices perpendicularmagneticanisotro一、弓I言 (1)二、............................. LLG方程的具体求解过程4（一） ................................. 、单轴铁磁晶体处于交变磁场中的LLG方程4（二） ........................................................ 、LLG方程的求解5三、........................................ 对LLG方程进彳亍讨论................................... 错误!未定义书签。
内蒙古六盟四十九旗
内蒙古六盟四十九旗漠南蒙古藩封科尔沁六旗，亲王四人，郡王四人，贝勒二人，贝子一人，镇国公二人，辅国公六人；扎赉特一旗，贝勒一人；杜尔伯特一旗，贝子一人；郭尔罗斯二旗，镇国公一人，辅国公一人，一等台吉一人：四部十旗为一会盟于哲里穆。
敖罕一旗，郡王一人，贝子一人，辅国公一人；奈曼一旗，郡王一人；翁牛特二旗，郡王一人，贝勒一人，贝子一人，镇国公一人；巴林二旗，郡王二人，贝子二人，镇国公一人；扎鲁特二旗，贝勒二人，镇国公一人；喀尔喀左翼一旗，贝子一人；阿禄科尔沁一旗，贝勒一人；克西克腾一旗，一等台吉一人：八部十一旗为一会盟于召乌达。
喀喇沁三旗，郡王一人，贝子一人，镇国公一人，辅国公二人，一等塔布囊一人；土默特二旗，贝勒一人，贝子一人，附喀尔喀贝勒一人：二部五旗为一会盟于卓索图。
乌珠穆秦二旗，亲王一人，郡王一人，贝勒一人，镇国公一人，辅国公一人；阿霸垓二旗，郡王一人，贝子一人，辅国公一人；蒿齐忒二旗，郡王二人；苏尼特二旗，郡王二人，贝子一人，辅国公一人；阿霸哈纳尔二旗，贝勒一人，贝子一人：五部十旗为一会盟于锡林。
四子部落一旗，郡王一人；喀尔喀右翼一旗，贝勒一人，贝子一人，辅国公一人；毛明安一旗，贝勒一人，一等台吉一人；乌喇特三旗，镇国公一人，辅国公一人：四部六旗为一会盟于乌兰察布。
鄂尔多斯七旗，郡王一人，贝勒二人，贝子三人，辅国公一人，一等台吉（各）一人，自为一会盟于伊克召。
每会设盟长一人，副盟长一人。
惟归化城土默特会盟，不设盟长，均集于本城，听简命大臣裁定。
漠北蒙古藩封喀尔喀后路土谢图汗二十旗，汗一人，亲王二人，郡王一人，贝勒二人，辅国公七人，一等台吉八人，为一会盟于汗阿林。
东路车臣汗二十三旗，汗一人，亲王一人，郡王一人，贝勒、贝子各二人，镇国公三人，辅国公三人，一等台吉十四人，为一会盟于克鲁伦巴尔河屯。
,西路扎萨克图汗十七旗，汗一人，贝勒一人，镇国公三人，辅国公六人，一等台吉九人，为一会盟于扎克毕赖塞钦毕都里也诺尔。
高斯投影分带.ppt
高斯-克吕格投影
坐标轴向西 平移500公 里后的坐标
原点
纵坐标（ ）
投影带的中央子午线
赤道
′
横坐标（ ）
公里
°
8
高斯-克吕格投影
• 由于高斯-克吕格投影每一个投影带的坐标 都是对本带坐标原点的相对值，所以各带 的坐标完全相同，使用时只需变一个带号 即可。
9
中央经线 为111°° 和117°或 六度分带 的19和20 带及三度 分带的37 和39带的 地图中的 同一位置 的坐标是 相同的。
11
3
高斯-克吕格投影
横切 圆柱
地球
4
六度分带 中每个带 的中央经
度
三度分带中 每个带的中
央经度
高斯投影分带
六度分 带的带
号
三度分带 的带号
5
高斯-克吕格投影
(广东省内的分带)
赤道带编号 带编号 Nhomakorabea°° ° ° °
6
高斯-克吕格投影
• 在高斯坐标系中，为了避免横坐标Y有负值， 将其起算原点向西移动500公里，即对横坐 标Y值按代数法加上500000米。此外，在 计算出来的和数前面加上带号，以便识别 该点位于何带。例如位于19带之某一点， 其横坐标值为Y=-126568.24米，根据上面 的规定，改变的(通用的)横坐标值 Y=45373431.76米。
1
高斯投影分带
• 我国的经度范围西起 73度东至135度，可 分成六度带十一带或三度带二十二带。六 度带可用于中小比例尺（1：25000以下） 测图，三度带可用于大比例尺（如 1： 10000和大于1：10000比例尺）测图。广 东省的经度范围是109°39′至117°12′，六 度带有两个带，带号分别是19和20，三度 带有3个，带号分别是37、38、39。分带图 如下：
自己总结的大航海时代4 补给点全览之白金版
法尔维尔 北65西39 （住宿、水手）
那维克 北69东18 （修理、水手）
季克西 北72东129
狄克逊 北73东80
科尔夫 北60东165
海克拉 北63西18（住宿） 邱吉尔 北58西93
圣塔巴巴拉 北34西120 （住宿）
速亚群岛 北38西28
鳕角 北41西70
北海道 北42东141 （住宿、修理）
夏威夷 北22西159 关岛 北15东145
喀劳 南8西78 大溪地 南14西147 里约热内卢 南23西 43（住宿） 卡里比布 南22东14
雷巴屈 南16东122（住宿）
法尔巴拉朔 南38西73 （住宿） 蒙特维多 南38西60ห้องสมุดไป่ตู้
帕士南 南31东115
偌母北68西165衣奴维克北69西136水手住宿修理圣塔巴巴拉北34西120住宿邱吉尔北58西93鳕角北41西70法尔维尔北65西39住宿水手海克拉北63西18住宿那维克北69东18修理水手狄克逊北73东80季克西北72东129科尔夫北60东165北海道北42东141住宿修理夏威夷北22西159速亚群岛北38西28关岛北15东145网上的邱吉尔地名坐标不对应该是
汪加奴 南39东174
网上的邱吉尔地名坐标不对，应该是：北58西93
北京同仁医院东、西区乘车路线
北京同仁医院东、西区乘车路线北京同仁医院东、西区乘车路线同仁医院东西两区院内部都设有地面停车场,但数量不多,两区分别有60余车位,收费标准:白天(7:00-21:00)小型车2.0元/小时，大型车4元/小时，白天(21:00-7:00)小型车0.5元/小时，大型车1元/小时.东区北边另有一个小型停车场，但由于工地施工，目前只有15个车位。
北京同仁医院东区、西区地处北京市繁华闹市区，日门诊量达7000人次，停车位极度紧缺，院领导及北京市政府正在积极采取措施；为了您方便就医，尽量不驾车，乘坐公交、地铁、出租均可到达。
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1.慢性肺源性心脏病急性加重期的处理原则答（一）控制感染原则上选用窄谱抗生素为主（二）氧疗保持呼吸道通畅，纠正缺氧和二氧化碳潴留（三）控制心力衰竭利尿剂血管扩张药、强心剂（四）控制心律失常（五）纠正呼吸衰竭合理氧疗呼吸兴奋剂建立人工气道和机械通气（六）降低肺动脉高压（七）抗凝治疗（八）加强护理工作。
2.危重哮喘的处理原则（1）保持呼吸道通畅，吸氧（2）拟肾上腺激素药物的应用（3）氨茶碱静脉滴注。
（4）大量补液，稀释痰液(5)琥珀酸氢化可的松静脉滴注。
（6）保持电解质及酸碱平衡，酸中毒者注意使用碱性药物进行纠正。
（7）积极控制肺部感染对于控制哮喘发作具有重要意义。
(8)病情严重或者患者已出现意识障碍，应尽早考虑机械通气治疗，以挽救患者生命。
3.肺炎的抗菌药物：首选青霉素G。
青霉素过敏者可选用红霉素或环丙沙星、氧氟沙星（喹诺酮类药物并发感染性休克的处理原则(1)一般治疗：保暖、平卧、吸氧生命体征及出入液量检测等(2) 补充血容量（3）纠正酸中毒（4）在不足血容量基础上使用血管活性药物多巴胺、间羟胺等（5）控制感染：大剂量青霉素静(6)糖皮质激素抗毒、抗休克等(7)纠正水、电解质紊乱，防治心肾功能不全等4.肺结核的化学治疗原则：早期、适量、联合、规律、全程及疗效考核指标：痰液细菌检查是考核疗效的主要指标。
痰菌转阴说明病灶内细菌大量减少或完全灭绝，已不再是传染源；若痰菌转阳，则提示病变复发，治疗失败。
另外X线检查是判断病情转归的重要依据，但疗效判定需结合痰菌检查和临床表现。
5.NYHA心功能的分级：1级：体力活动不受限制。
一般活动不引起乏力、心悸、呼吸困难或心绞痛。
2级：体力活动轻度受限。
休息无症状，日常活动即可引起乏力、心悸、呼吸困难或心绞痛。
3级：体力活动明显受限。
休息无症状，少微活动(小于日常活动）即可引起乏力、心悸、呼吸困难或心绞痛。
4级：不能从事任何体力活动，休息时即可引起乏力、心悸、呼吸困难或心绞痛。
体力活动后加重。
6.原发性高血压病与嗜铬细胞留、原发性醛固酮增多症的鉴别原发性高血压病与嗜铬细胞留、原发性醛固酮增多症四者都有血压增高，但原发性高血压是有单纯性血压升高。
嗜铬细胞留：高血压可持续性或阵发性并伴有头痛、心悸、恶心、多汗、四肢冰冷和麻木感、视力减退、上腹部或胸骨后疼痛等症状。
血和尿的检查有儿茶酚胺及其代谢产物出现。
原发性醛固酮增多症：轻度和中度高血压，多尿以夜尿为甚，有口渴、尿比重下降、碱性尿、蛋白尿并伴有发作性肌无力或瘫痪、肌痛、手足麻木。
实验室检查提示血和尿中醛固酮升高，PRA降低。
7.高血压病的药物治疗1）利尿剂：可作为轻中度无并发症高血压患者的首选药物，尤其适合老年性高血压或并发心力衰竭的患者，有痛风的禁用。
2）Β受体阻滞剂；适用轻、中度高血压，尤其对心率较快的中青年高血压患者,或伴心绞痛或心肌梗塞后,以及伴室上性快速性心律失常者.3）血管紧张素转换酶抑制剂(ACEI):适用于轻,中度或严重高血压,尤适用于伴左心室肥厚,心力衰竭,糖尿病并有微量蛋白尿,肾脏损害并有蛋白尿等患者.可用于伴有慢性阻塞性肺疾病或哮喘\周围血管疾病或雷诺现象,抑郁症,以及1型糖尿病患者.4）AT-Ⅱ受体拮抗剂(ARB):主要用于ACEI治疗后不能耐受发生干咳的患者,尤其适用于伴高尿酸血症或痛风的患者. 5）钙通道阻滞剂:适用于各型高血压,特别是老年收缩期高血压,并发糖耐量减低,心绞痛患者.6）α1受体阻滞剂:一般用于轻,中度高血压,尤其适用伴高脂血症或前列腺肥大的患者.反正高血压的治疗应采取个体化的原则,综合考虑患者的实际情况决定治疗方案.8急性左心衰的临床表现及处理原则(1)临床表现：急性左心衰竭发病急骤，主要表现为急性肺水肿或心源性休克。
病人突发严重呼吸困难、端坐呼吸、烦躁不安、面色苍白、皮肤湿冷、大汗淋漓，并频繁咳嗽，咳粉红色泡沫样痰。
较重者可因为缺氧而致神志模糊。
发病开始可有一过性血压升高，病情如不缓解，血压可持续下降至休克，听诊时两肺底布满湿性罗音和广泛哮鸣音，心尖部舒张期奔马律，肺动脉瓣第二心音亢进。
(主要突发严重呼吸困难,咳大量粉红色泡沫样痰).（2）处理原则：．（1）患者取坐位或半卧位，以减少回心血量(2)．高流量鼻导管或面罩吸氧，增加供氧(3)．应用镇静剂(4)．快速利尿，可用呋塞米或利尿酸钠（5）．应用血管扩张剂（6）．应用强心剂,增加心肌收缩力.（7）．可使用茶碱类药物（8）．其它措施如应用糖皮质激素（9）．两肺湿啰音和哮鸣音监测和各项生命指标监测及血压、尿量监测（10）．进行心电监护、心电图检查和电解质监测9典型心绞痛的胸痛特点心绞痛以发作性胸痛为主要临床表现疼痛特点为（1）诱因：发作常由体力劳动、情绪激动、饱食、寒冷刺激、吸烟、心动过速、休克等增加心肌耗氧的因素所诱发，疼痛发生在诱因的当时，以清晨为多。
（2）性质：呈压榨性或紧缩性，可伴烧灼感。
因伴濒死感、恐惧感。
使患者出现强迫体位，直至症状缓解。
（3）部位：位于胸骨体上中段之后，可波及心前区，约手掌范围大小，边界不清，常放射至左肩、左臂内侧直达环指吃侧和小指，或至颈部或下颌部。
（4）持续时间：疼痛出现后常逐渐加重，多在3-5分钟逐渐消失。
多数患者持续时间《15分钟。
（5）缓解方式:祛除诱因即可缓解，舌下含服硝酸甘油多在数分钟内完全缓解。
可数日或数周发作一次，也可一日内多次发作。
发作频率与诱因出现的频率有关。
10急性心肌梗死的治疗原则:保护和维持心功能,挽救濒死的心肌,防止梗死面积扩大,缩小心肌缺血范围,及时处理严重心律失常,心力衰竭,休克和各种并发症,防止猝死.11.急性心肌梗死的诊断:根据典型的临床表现(胸痛多发生于早晨,呈压榨性疼痛,多再胸骨体上、中段之后,常伴有放射痛向左肩,左臂方向,疼痛持续的时间长,含硝酸甘油不能缓解.心电图出现特征性改变(典型的坏死波)是诊断心肌梗死的可靠依据,血沉增快,血清心肌酶测定,CK-MB增高,LDM增高AST增高.13．消化性溃疡的并发症：并发溃疡出血、穿孔、幽门梗阻和癌变等14．消化性溃疡的药物治疗及抗HP的方案药物治疗：抗HP药物治疗。
用抑制胃酸药物。
保护胃黏膜药物。
胃肠动力药物。
抗HP药物治疗：应用三联疗法是胶体铋或质子泵抑制剂为基础再加2种抗菌药的三联疗法，疗程为7日。
15．急性胰腺炎的病因及治疗原则(1)急性胰腺炎的病因是：胆石症和胆道疾病、胰管梗阻、大量饮酒与暴饮暴食、、手术与创伤内分泌及代谢障碍、感染、药物等(2)治疗原则1)饮食控制2)胃肠减压3)静脉输液，及时补足血容量4)镇痛5)抗菌药物6)营养支持7)预防和治疗肠道衰竭8)抑制胰液分泌9)抑制胰酶药 10) H2受体拮抗药和PPI抑制胃酸分泌间接抑制胰液分泌11)抗炎症因子药物12)鼻胆管引流或内镜下括约肌切开术(EST)对感染性胰腺坏死、胰腺脓肿：应手术治16．肝硬化失代偿期的临床表现（一）肝功能减退的临床表现（1）全身症状和体征：一般状况较差，疲倦。
乏力、精神不振，营养状况较差消瘦，呈肝病面容，皮肤干枯粗糙，夜盲、水肿、舌炎、口角炎等（2）消化系统症状：食欲减退最为常见的症状，上腹饱胀，伴恶心呕吐，上述症状的出现与胃肠道淤血水肿、消化吸收功能紊乱和肠道菌群失调等因素有关。
（3）出血倾向和贫血由于肝合成凝血因子减少，脾功能亢进和毛细血管脆性增加，导致凝血功能障碍，常出现鼻出血、牙龈出血、皮肤紫癜，女性常有月经过多；由于营养不良，肠道吸收障碍、胃肠道失血和脾功能亢进等因素，病人可有不同程度的贫血。
（4）内分泌失调1）雌激素增多，雄激素和糖皮质激素减少：男性病人常性欲减退，睾丸萎缩，毛发脱落及乳房发育；女性病人可有月经失调，闭经不孕等，部分病人出现蜘蛛痣、肝掌。
2）醛固酮和抗利尿激素增多：尿少、水肿。
（二）门静脉高压症（1）脾大、侧支循环的建立和开放、腹水。
17.肝硬化腹水的治疗原则限制水、钠的摄入，应用利尿剂主要使用螺内脂和呋塞米，提高血浆胶体渗透压可定期少量、多次静脉输注鲜血和白蛋白，大量腹水以上治疗无效的可以放腹水疗法、腹水浓缩回输术或颈静脉肝内门－体分流术。
18上消化道大出血时如何估计出血量？每日出血量5-10ml以上大便隐血试验呈阳性，每日出血量50-100ml可出现黑便。
胃内积血250-300ml可引起呕血，一次出血400-500ml可引起头昏、心慌、乏力，大于10000ml可出现休克症状。
19.上消化道大出血的治疗原则治理原则（1）.积极控制出血; （2）.治疗原发病; （3）.必要时输血及手术治疗。
1. 一般处理:保持安静休息,控制饮食,留置胃管并严密观察病情变化。
2. 输液、输血防治休克:3. 止血剂的应用4. 控制胃内pH保护胃粘膜:5. 胃内降温及局部药物的应用:6. 内镜下局部止血:7. 血管造影及介入治疗止血:8. 门脉高压食管胃底静脉曲张破裂出血者,可应用降低门脉压力的药物(垂体后叶素、心得安等) ,也可应用三腔二囊管压迫止血。
9. 手术止血:20、慢性肾小球肾炎的临床表现答：急性肾炎迁延不愈超过病程1年。
有急性肾炎病史临床症状缓解1-2年，而病理变化仍缓慢进展。
无急性肾炎病史，一开始就表现为慢性肾炎临床表现 :早期可有乏力、疲倦、腰痛、纳差。
水肿可有可无，有的可无明显症状，以蛋白尿、血尿、高血压和水肿为基本的临床表现（1）水肿 (2)高血压持续中等度以上160-180/90-110mmHg (3)、肾功能不全肾小球率过滤下降，内生肌酐清除率下降 (4)、全身症状头晕、乏力、食欲不振、精神差等 (5)、尿检查异常尿蛋白1-3g/24小时，偶有大量蛋白尿，尿比重在1.020以下。
21、慢性肾衰时高钾血症的处理原则答：原则：高钾血症时除了限制钾摄入外，应采用利尿、导泻加速钾的排泄，血钾>6．5mmol/L时需紧急处理，静脉注射10%葡萄糖酸钙20ml，或滴注碳酸氢钠以拮抗钾，也可用胰岛素、葡萄糖按1u：3-5g的比例静脉滴注。
上述方法暂时将血钾转入细胞内，仅维持6-8小时，应准备透析治疗。
当尿毒症患者并发感染、酸中毒或长期服保钾利尿剂、输含钾多的库存血、或严重少尿时均可致高钾血症。
其临床表现是心律失常、甚至心跳骤停，以及四肢肌肉无力、手足感觉异常等22、再障的治疗方案一、支持治疗（一）保护措施：预防感染；避免出血；杜绝接触各类危险因素；必要的心理护理。
（二）对症治疗（1）贫血，可输注去白细胞的浓缩红细胞血（2）出血常规止血药（3）感染，积极给予抗感染治疗药物（4）护肝治疗二、针对发病机制的治疗（一）免疫抑制治疗（1）免疫抑制剂（2）免疫调节剂（二）(1)促造血治疗雄激素，以刺激骨髓造血干细胞分行增值，并促进肾脏产生促红细胞生成素 (2)造血生长因子（三）造血肝细胞移植对40岁以下、无感染及其他并发症可考虑移植。