抗阻训练的心血管调控机制及其在心脏康复中的应用复习进程
相关主题
- 1、下载文档前请自行甄别文档内容的完整性，平台不提供额外的编辑、内容补充、找答案等附加服务。
- 2、"仅部分预览"的文档,不可在线预览部分如存在完整性等问题,可反馈申请退款(可完整预览的文档不适用该条件!)。
- 3、如文档侵犯您的权益，请联系客服反馈,我们会尽快为您处理(人工客服工作时间：9:00-18:30)。
• There was no sign of myocardial ischemia during RTmax, but happened in DEmax. RTmax时心肌缺血少于DEmax
7
Why no ischemia during IE？
HR IE<DE
DBP IE>DE
Perfusion time IE>DE
• 电刺激等长收缩运动心血管反应与肌群质量的关系。中国 运动医学杂志 2000；19（4）：392-395
11
May cardiovascular response to IE be modified?
• Central command has more profound effect on EOPs.
• EOPs
– Enkephalin, ENK, LEK – β- endorphin,β-END – Dynaphin, DYN
10
Publications
• 等长收缩运动时血浆内啡肽对心血管反应的调节作用. 中 华物理医学杂志, 1998；20(2): 105-108
• 等长收缩运动时生理性肺毛细血管楔嵌压增高的机理。中 国运动医学杂志；2000; 19(3): 251-253
2.Regulation of cardiovascular responses to isometric exercise in coronary patRTnts. Aust New Zealand J Med 1991; 21(4):524
3.Mechanism of Pulmonary Wedge Pressure Elevation During Isometric Exercise in PatRTnts with Coronary Artery Disease. J Nanjing Med Coll 1993;7(2):1-10
CA perfusion IE>DE
Perfusion pressure IE>DE
ischemia IE<DE
hymodynamics IE<DE
MVO2 IE<DE
Publications
1.Effects of isometric exercise on hemodynamics and left ventricular systolic and diastolic function in normal subjects and patRTnts convalescent from myocardial infarction. Aust New Zealand J med 1990;20(3):325
• 血浆内啡肽对等长收缩运动心血管反应的中枢调节定位。 中华物理医学与康复杂志，2000;22(5):287-290
• 大脑行为对等长收缩运动时心血管反应的调控作用与机制 。中国康复医学杂志2000;15(3):140-143
• RT induced PWP elevation is neither a result of LV failure nor myocardial ischemia, but the increase of intra-thoracic pressure. 抗阻运动引起肺毛压增高的原因是胸内压增高， 而不是左心功能障碍。
抗阻训练的心血管调控机制及其 在心脏康复中的应用
心血管康复的内容 approach in cardiac rehab
Study structure
Central hemodynamics
Clinical observation
Mechanism
Brain Spine Muscle EOPs
Recording
4.冠心病人等长收缩运动时肺毛细血管嵌顿压升高的机理。中国康复医学 杂志 1993;8(1):1
5.分级等长收缩运动对正常人和心肌梗塞患者二尖瓣血流的影响。中国康 复医学杂志 1993;8(6):244-247
6.乏氏动作对冠心病人等长收缩运动心血管反应的影响。中国康复医学杂 志，1995;10(1):13-16
6
Cardiac responses to RT
• Increase of MVO2 was weaker during RT than DE. 抗阻运动时心肌耗氧量的增加低于动力性运动
• RT resulted in parallel increase of SBP and DBP which is decreased during DE. RT时SBP和DBP同时升高
• Brain behavior modification may adjust cardiovascular responwk.baidu.come to IE through mechanism of enhanced release of EOPs.
Publications
• 中枢内啡肽对等长收缩运动时心血管反应的调控意义。中 华物理医学与康复杂志，2000;22(4):211-214
Animal study
6
5
• 20 dogs
4
• IntraThoracic 3
TP PWP
Pressure is related 2
to PWP
1
(r=0.89)
0
5
Cardiac responses to RT
• Cardiac response to RT has its own rule and does not induce undue cardiovascular responses. 抗阻运动并不诱发不可预期的高心血管反应
7.影响正常人和心肌梗塞病人对等长收缩运动心血管反应的有关因素. 中 国康复医学杂志 1995;10(5):193-197
Why RT produce weaker cardiovascular response than DE?
• Endogenous Opioids (EOPs) , a modulator of cardiac neuro-control, increase more significantly during RT than DE and thus adjust cardiac response to RT.
7
Why no ischemia during IE？
HR IE<DE
DBP IE>DE
Perfusion time IE>DE
• 电刺激等长收缩运动心血管反应与肌群质量的关系。中国 运动医学杂志 2000；19（4）：392-395
11
May cardiovascular response to IE be modified?
• Central command has more profound effect on EOPs.
• EOPs
– Enkephalin, ENK, LEK – β- endorphin,β-END – Dynaphin, DYN
10
Publications
• 等长收缩运动时血浆内啡肽对心血管反应的调节作用. 中 华物理医学杂志, 1998；20(2): 105-108
• 等长收缩运动时生理性肺毛细血管楔嵌压增高的机理。中 国运动医学杂志；2000; 19(3): 251-253
2.Regulation of cardiovascular responses to isometric exercise in coronary patRTnts. Aust New Zealand J Med 1991; 21(4):524
3.Mechanism of Pulmonary Wedge Pressure Elevation During Isometric Exercise in PatRTnts with Coronary Artery Disease. J Nanjing Med Coll 1993;7(2):1-10
CA perfusion IE>DE
Perfusion pressure IE>DE
ischemia IE<DE
hymodynamics IE<DE
MVO2 IE<DE
Publications
1.Effects of isometric exercise on hemodynamics and left ventricular systolic and diastolic function in normal subjects and patRTnts convalescent from myocardial infarction. Aust New Zealand J med 1990;20(3):325
• 血浆内啡肽对等长收缩运动心血管反应的中枢调节定位。 中华物理医学与康复杂志，2000;22(5):287-290
• 大脑行为对等长收缩运动时心血管反应的调控作用与机制 。中国康复医学杂志2000;15(3):140-143
• RT induced PWP elevation is neither a result of LV failure nor myocardial ischemia, but the increase of intra-thoracic pressure. 抗阻运动引起肺毛压增高的原因是胸内压增高， 而不是左心功能障碍。
抗阻训练的心血管调控机制及其 在心脏康复中的应用
心血管康复的内容 approach in cardiac rehab
Study structure
Central hemodynamics
Clinical observation
Mechanism
Brain Spine Muscle EOPs
Recording
4.冠心病人等长收缩运动时肺毛细血管嵌顿压升高的机理。中国康复医学 杂志 1993;8(1):1
5.分级等长收缩运动对正常人和心肌梗塞患者二尖瓣血流的影响。中国康 复医学杂志 1993;8(6):244-247
6.乏氏动作对冠心病人等长收缩运动心血管反应的影响。中国康复医学杂 志，1995;10(1):13-16
6
Cardiac responses to RT
• Increase of MVO2 was weaker during RT than DE. 抗阻运动时心肌耗氧量的增加低于动力性运动
• RT resulted in parallel increase of SBP and DBP which is decreased during DE. RT时SBP和DBP同时升高
• Brain behavior modification may adjust cardiovascular responwk.baidu.come to IE through mechanism of enhanced release of EOPs.
Publications
• 中枢内啡肽对等长收缩运动时心血管反应的调控意义。中 华物理医学与康复杂志，2000;22(4):211-214
Animal study
6
5
• 20 dogs
4
• IntraThoracic 3
TP PWP
Pressure is related 2
to PWP
1
(r=0.89)
0
5
Cardiac responses to RT
• Cardiac response to RT has its own rule and does not induce undue cardiovascular responses. 抗阻运动并不诱发不可预期的高心血管反应
7.影响正常人和心肌梗塞病人对等长收缩运动心血管反应的有关因素. 中 国康复医学杂志 1995;10(5):193-197
Why RT produce weaker cardiovascular response than DE?
• Endogenous Opioids (EOPs) , a modulator of cardiac neuro-control, increase more significantly during RT than DE and thus adjust cardiac response to RT.