脑囊虫病影像诊断

Parenchymal
Ventricular(the third or fourth) Meningeal
Mixed
Subclinical stage
Active stage Degeneration and deathபைடு நூலகம்stage
Calcification stage
Mixed stage
Active stage
small excentric spotty shadow of the mural
cysticercus scolex low signals on T1WI,high signals onT2WI capsular liquid : higher than CSF on FLAIR unremarkable peripheral edema
most commonly in the whole body
cysticercosis(80％) most commonly in cerebral parasitic disease more prevalent in the country Pathogen: the cysticercus of armed tapeworm
necrosis and cystic degeneration
obvious nodular or ringlike enhancement
Supratentorial(temporal lobe)
Pathogen:MRSA,streptococcus,pneumococcus Periodization
Degeneration and death stage scolex disappearance enlarged capsular cavity remarkable peripheral edema “white/black target sign” “multi-ring sign” “delanminated sign”
Metastatic encephaloma Brain abscess
Children and youth
Classification
tuberculous meningitis tuberculoma tuberculous brain abscess
Tuberculous meningitis
CAg: activity of neurocysticercosis
IgG4:therapy effect IHA/ELISA:supplementary indicator
Medical treatment : active stage
albendazole+praziquantel Surgical operation : degeneration and death stage&granulomatous stage cysticercosis enucleation decompressive craniectomy ventriculoperitoneal shunt
acute encephalitis stage suppuration stage capsule formation stage
acute encephalitis stage
low signal on T1WI,high signal on T2WI suppuration and capsule formation stage the wall: complete,smooth,thin,homogeneous circular enhancement “dark belt” sign
Calcification stage lower signals on T1WI and T2WI peripheral edema disappearance
Mixed stage
Enhanced scanning:ringlike enhancement
Intracranial tuberculosis
By Head Group 2013-5-9
Female,48Y
Sudden onset;headache, nausea and vomit for
10 years; aggravation for 1 day CSF culture(－)
Cerebral Cysticercosis
hydrocephalus distention of subarachnoid spaces abnormal meningeal and cistern enhancement
Tuberculoma
space-occupying effect hydrocephalus calcification “target sign” ringlike or eggshell-like enhancement
Making a correct diagnosis should be based
on epidemiology, patient history, clinical manifestations, neuroimaging and laboratory examination,carring out a comprehensive analysis. Some non-typical makes misdiagnosed easily, rechecking MRI and laboratory examination can help the differential diagnosis.
Primary leision(lung,braest,melanoma)
Multiple Supratentorial(80％)，subtentorial(20％)
Cortex-medullary junctions
Isointensity on T1WI
Iso- or mildly hypointensity on T2WI “small nodule and big edema” sign